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Modulation of DNA damage and repair pathways by human tumour viruses

Modulation of DNA damage and repair pathways by human tumour viruses

http://www.wikidata.org/entity/Q26825783

about

Activation of DNA Damage Response Induced by the Kaposi's Sarcoma-Associated Herpes Virus Exosome Biogenesis, Regulation, and Function in Viral Infection DNA damage response (DDR) and senescence: shuttled inflamma-miRNAs on the stage of inflamm-aging The Adenovirus E4orf4 Protein Provides a Novel Mechanism for Inhibition of the DNA Damage Response Impact of the DNA Damage Response on Human Papillomavirus Chromatin.KSHV encoded LANA recruits Nucleosome Assembly Protein NAP1L1 for regulating viral DNA replication and transcription Kaposi's Sarcoma-Associated Herpesvirus MicroRNAs Target GADD45B To Protect Infected Cells from Cell Cycle Arrest and Apoptosis DNA damage response is hijacked by human papillomaviruses to complete their life cycle Human T-cell lymphotropic virus type 1 and its oncogenesis.The Rb binding domain of HPV31 E7 is required to maintain high levels of DNA repair factors in infected cells.A Role for the Host DNA Damage Response in Hepatitis B Virus cccDNA Formation-and Beyond?CD155, an onco-immunologic molecule in human tumors.Take your PIKK: tumour viruses and DNA damage response pathways.Human Papillomavirus and the DNA Damage Response: Exploiting Host Repair Pathways for Viral Replication.Deficiency in DNA damage response, a new characteristic of cells infected with latent HIV-1.Viral Interplay with the Host Sumoylation System.Virome characterisation from Guthrie cards in children who later developed acute lymphoblastic leukaemia.Induction of DNA damages upon Marek's disease virus infection: implication in viral replication and pathogenesis.Correction: Hollingworth, R.; Grand, R.J. Modulation of DNA Damage and Repair Pathways by Human Tumour Viruses. Viruses 2015, 7, 2542-2591.Epstein-Barr Virus Hijacks DNA Damage Response Transducers to Orchestrate Its Life Cycle.Hepatitis B Virus and DNA Damage Response: Interactions and Consequences for the Infection.How Human Papillomavirus Replication and Immune Evasion Strategies Take Advantage of the Host DNA Damage Repair Machinery.Role of HBx in hepatitis B virus persistence and its therapeutic implications.Serotype-specific restriction of wild-type adenoviruses by the cellular Mre11-Rad50-Nbs1 complex.TP53 Arg72Pro polymorphism is associated with increased overall survival but not response to therapy in Portuguese/Caucasian patients with advanced cervical cancer.

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P2860

Modulation of DNA damage and repair pathways by human tumour viruses

http://www.wikidata.org/entity/Q26825783

description

2015 nî lūn-bûn

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2015 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2015 թվականի մայիսին հրատարակված գիտական հոդված

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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name

Modulation of DNA damage and repair pathways by human tumour viruses

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Modulation of DNA damage and repair pathways by human tumour viruses

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Modulation of DNA damage and repair pathways by human tumour viruses

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type

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Modulation of DNA damage and repair pathways by human tumour viruses

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Modulation of DNA damage and repair pathways by human tumour viruses

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Modulation of DNA damage and repair pathways by human tumour viruses

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Modulation of DNA damage and repair pathways by human tumour viruses

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Modulation of DNA damage and repair pathways by human tumour viruses

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Modulation of DNA damage and repair pathways by human tumour viruses

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Modulation of DNA damage and repair pathways by human tumour viruses

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Robert Hollingworth

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Roger Grand

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Creative Commons Attribution

P2860

The mechanism of double-strand DNA break repair by the nonhomologous DNA end-joining pathway

Papillomavirus genome structure, expression, and post-transcriptional regulation

HERC2 coordinates ubiquitin-dependent assembly of DNA repair factors on damaged chromosomes

BLM-DNA2-RPA-MRN and EXO1-BLM-RPA-MRN constitute two DNA end resection machineries for human DNA break repair

Mechanism of 5'-directed excision in human mismatch repair

XLF interacts with the XRCC4-DNA ligase IV complex to promote DNA nonhomologous end-joining

The RIDDLE syndrome protein mediates a ubiquitin-dependent signaling cascade at sites of DNA damage

The DNA damage sensors ataxia-telangiectasia mutated kinase and checkpoint kinase 2 are required for hepatitis C virus RNA replication

Activity of DNA ligase IV stimulated by complex formation with XRCC4 protein in mammalian cells

Ku recruits the XRCC4-ligase IV complex to DNA ends

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