Sex- and brain region-specific acceleration of β-amyloidogenesis following behavioral stress in a mouse model of Alzheimer's disease.
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Behavioral stress fails to accelerate the onset and progression of plaque pathology in the brain of a mouse model of Alzheimer's diseaseAccelerated aging-related transcriptome changes in the female prefrontal cortexMechanisms underlying insulin deficiency-induced acceleration of β-amyloidosis in a mouse model of Alzheimer's diseasePosttraumatic stress disorder-like induction elevates β-amyloid levels, which directly activates corticotropin-releasing factor neurons to exacerbate stress responses.Limited effects of an eIF2αS51A allele on neurological impairments in the 5xFAD mouse model of Alzheimer's disease.Gene expression levels assessed by CA1 pyramidal neuron and regional hippocampal dissections in Alzheimer's diseasePERK mediates eIF2α phosphorylation responsible for BACE1 elevation, CREB dysfunction and neurodegeneration in a mouse model of Alzheimer's disease.Temporal gene profiling of the 5XFAD transgenic mouse model highlights the importance of microglial activation in Alzheimer's disease.Deletion of the eIF2α Kinase GCN2 fails to rescue the memory decline associated with Alzheimer's disease.Increased fragmentation of sleep-wake cycles in the 5XFAD mouse model of Alzheimer's diseaseUpregulation of select rab GTPases in cholinergic basal forebrain neurons in mild cognitive impairment and Alzheimer's disease.Male or Female? Brains are IntersexA combination Alzheimer's therapy targeting BACE1 and neprilysin in 5XFAD transgenic mice.Chronic stress exacerbates tau pathology, neurodegeneration, and cognitive performance through a corticotropin-releasing factor receptor-dependent mechanism in a transgenic mouse model of tauopathy7,8-dihydroxyflavone, a small-molecule TrkB agonist, reverses memory deficits and BACE1 elevation in a mouse model of Alzheimer's disease.TrkB reduction exacerbates Alzheimer's disease-like signaling aberrations and memory deficits without affecting β-amyloidosis in 5XFAD mice.The influence of chronic stress on dementia-related diagnostic change in older adultsChronic Noise Exposure Acts Cumulatively to Exacerbate Alzheimer's Disease-Like Amyloid-β Pathology and Neuroinflammation in the Rat HippocampusBACE1 is at the crossroad of a toxic vicious cycle involving cellular stress and β-amyloid production in Alzheimer's diseaseSex and gonadal hormones in mouse models of Alzheimer's disease: what is relevant to the human condition?Mechanisms that lessen benefits of β-secretase reduction in a mouse model of Alzheimer's disease.Glutamatergic transmission aberration: a major cause of behavioral deficits in a murine model of Down's syndrome.Roles of eIF2α kinases in the pathogenesis of Alzheimer's disease.EFAD transgenic mice as a human APOE relevant preclinical model of Alzheimer's disease.Modulation of neuroinflammation and pathology in the 5XFAD mouse model of Alzheimer's disease using a biased and selective beta-1 adrenergic receptor partial agonist.Diesel engine exhaust accelerates plaque formation in a mouse model of Alzheimer's disease.Practical considerations for choosing a mouse model of Alzheimer's disease.The Lesion Analysis of Cholinergic Neurons in 5XFAD Mouse Model in the Three-Dimensional Level of Whole Brain.BACE1 RNA interference improves spatial memory and attenuates Aβ burden in a streptozotocin-induced tau hyperphosphorylated rat model.Restraint stress in rats alters gene transcription and protein translation in the hippocampus.The Synergistic Roles of the Chronic Prenatal and Offspring Stress Exposures in Impairing Offspring Learning and Memory.A Multifunctional Biocompatible Drug Candidate is Highly Effective in Delaying Pathological Signs of Alzheimer's Disease in 5XFAD Mice.SENP1 and SENP2 regulate SUMOylation of amyloid precursor protein.The relationship between stress and Alzheimer's disease.Tau Phosphorylation in Female Neurodegeneration: Role of Estrogens, Progesterone, and Prolactin.Chronic stress as a risk factor for Alzheimer's disease: Roles of microglia-mediated synaptic remodeling, inflammation, and oxidative stress.Amyloid pathology fingerprint differentiates post-traumatic stress disorder and traumatic brain injury.Article Withdrawal Statement
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P2860
Sex- and brain region-specific acceleration of β-amyloidogenesis following behavioral stress in a mouse model of Alzheimer's disease.
description
2010 nî lūn-bûn
@nan
2010 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Sex- and brain region-specific ...... model of Alzheimer's disease.
@ast
Sex- and brain region-specific ...... model of Alzheimer's disease.
@en
Sex- and brain region-specific ...... model of Alzheimer's disease.
@nl
type
label
Sex- and brain region-specific ...... model of Alzheimer's disease.
@ast
Sex- and brain region-specific ...... model of Alzheimer's disease.
@en
Sex- and brain region-specific ...... model of Alzheimer's disease.
@nl
prefLabel
Sex- and brain region-specific ...... model of Alzheimer's disease.
@ast
Sex- and brain region-specific ...... model of Alzheimer's disease.
@en
Sex- and brain region-specific ...... model of Alzheimer's disease.
@nl
P2093
P2860
P356
P1433
P1476
Sex- and brain region-specific ...... e model of Alzheimer's disease
@en
P2093
Latha Devi
Masuo Ohno
Melissa J Alldred
P2860
P2888
P356
10.1186/1756-6606-3-34
P577
2010-11-08T00:00:00Z
P5875
P6179
1000576994