about
Current State of Animal (Mouse) Modeling in Melanoma ResearchTargeting the Checkpoint to Kill Cancer CellsLKB1 loss in melanoma disrupts directional migration toward extracellular matrix cues.EZH2 couples pancreatic regeneration to neoplastic progression.Expression of p16(INK4a) prevents cancer and promotes aging in lymphocytes.Clinical and Histopathological Characteristics between Familial and Sporadic Melanoma in Barcelona, Spain.Genetics of melanomaLKB1/STK11 inactivation leads to expansion of a prometastatic tumor subpopulation in melanomaCtBP1 is expressed in melanoma and represses the transcription of p16INK4a and Brca1.Animal models of melanoma: a somatic cell gene delivery mouse model allows rapid evaluation of genes implicated in human melanoma.Modelling melanoma in mice.Narrowing the knowledge gaps for melanomaA tan in a test tube - in vitro models for investigating ultraviolet radiation-induced damage in skin.Recent advances in cutaneous melanoma: towards a molecular model and targeted treatment.The role of polo-like kinase 1 in carcinogenesis: cause or consequence?Insights into wild-type and mutant p53 functions provided by genetically engineered mice.Mechanisms of tumour vascularization in cutaneous malignant melanoma: clinical implications.An oncogenic Ezh2 mutation induces tumors through global redistribution of histone 3 lysine 27 trimethylation.Mutation-specific RAS oncogenicity explains NRAS codon 61 selection in melanoma.Stromal Senescence By Prolonged CDK4/6 Inhibition Potentiates Tumor Growth.Establishment and characterization of a primary and a metastatic melanoma cell line from Grey horses.Mouse melanoma models and cell lines.Treatment of melanoma with selected inhibitors of signaling kinases effectively reduces proliferation and induces expression of cell cycle inhibitors.Genetic Ablation of p16(Ink4a) Does Not Protect Against Cellular Senescence in Mouse Models of COPD/Emphysema.RAF inhibitor LY3009120 sensitizes RAS or BRAF mutant cancer to CDK4/6 inhibition by abemaciclib via superior inhibition of phospho-RB and suppression of cyclin D1.Pax3( GFP ) , a new reporter for the melanocyte lineage, highlights novel aspects of PAX3 expression in the skin.A genetically engineered mouse model with inducible GFP expression in melanocytes.Targeting the cyclin dependent kinase and retinoblastoma axis overcomes standard of care resistance in BRAF V600E -mutant melanoma.Spatiotemporal Labeling of Melanocytes in Mice.
P2860
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P2860
description
2010 nî lūn-bûn
@nan
2010 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Somatic p16
@nl
Somatic p16(INK4a) loss accelerates melanomagenesis
@ast
Somatic p16(INK4a) loss accelerates melanomagenesis
@en
type
label
Somatic p16
@nl
Somatic p16(INK4a) loss accelerates melanomagenesis
@ast
Somatic p16(INK4a) loss accelerates melanomagenesis
@en
prefLabel
Somatic p16
@nl
Somatic p16(INK4a) loss accelerates melanomagenesis
@ast
Somatic p16(INK4a) loss accelerates melanomagenesis
@en
P2093
P2860
P356
P1433
P1476
Somatic p16(INK4a) loss accelerates melanomagenesis
@en
P2093
G I Rozenberg
J Krishnamurthy
K B Monahan
M K Bradford
R A Depinho
S M Johnson
P2860
P2888
P304
P356
10.1038/ONC.2010.314
P407
P577
2010-08-09T00:00:00Z
P5875
P6179
1027925199