Tsg101 is essential for cell growth, proliferation, and cell survival of embryonic and adult tissues.
about
The human endosomal sorting complex required for transport (ESCRT-I) and its role in HIV-1 buddingRegulation of Tsg101 expression by the steadiness box: a role of Tsg101-associated ligaseTSG101 interaction with HRS mediates endosomal trafficking and receptor down-regulationThe penta-EF-hand protein ALG-2 interacts directly with the ESCRT-I component TSG101, and Ca2+-dependently co-localizes to aberrant endosomes with dominant-negative AAA ATPase SKD1/Vps4BThe role of the endosomal sorting complexes required for transport (ESCRT) in tumorigenesisMolecular targets for tumour progression in gastrointestinal stromal tumours.Endosomal-sorting complexes required for transport (ESCRT) pathway-dependent endosomal traffic regulates the localization of active Src at focal adhesions.A genomic-based approach combining in vivo selection in mice to identify a novel virulence gene in Leishmania.Common and distinct genetic properties of ESCRT-II components in DrosophilaGenetic interactions between the Drosophila tumor suppressor gene ept and the stat92E transcription factor.A mutation in dVps28 reveals a link between a subunit of the endosomal sorting complex required for transport-I complex and the actin cytoskeleton in Drosophila.Cell cycle arrest and cell death are controlled by p53-dependent and p53-independent mechanisms in Tsg101-deficient cells.A knockout of the Tsg101 gene leads to decreased expression of ErbB receptor tyrosine kinases and induction of autophagy prior to cell death.De-regulation of JNK and JAK/STAT signaling in ESCRT-II mutant tissues cooperatively contributes to neoplastic tumorigenesis.ESCRT proteins: Double-edged regulators of cellular signaling.Human MDM2 isoforms translated differentially on constitutive versus p53-regulated transcripts have distinct functions in the p53/MDM2 and TSG101/MDM2 feedback control loops.Role of the TSG101 gene in Epstein-Barr virus late gene transcription.Defective downregulation of receptor tyrosine kinases in cancerTSG101 Silencing Suppresses Hepatocellular Carcinoma Cell Growth by Inducing Cell Cycle Arrest and Autophagic Cell Death.TSG101, a tumor susceptibility gene, bidirectionally modulates cell invasion through regulating MMP-9 mRNA expression.Estrogen receptor-alpha expression in the mammary epithelium is required for ductal and alveolar morphogenesis in mice.Nipah Virus C Protein Recruits Tsg101 to Promote the Efficient Release of Virus in an ESCRT-Dependent PathwayESCRTs are everywhere.Oncogenic breakdowns in endocytic adaptor proteins.Cripto-1 ablation disrupts alveolar development in the mouse mammary gland through a progesterone receptor-mediated pathway.Role of mammary epithelial and stromal P450 enzymes in the clearance and metabolic activation of 7,12-dimethylbenz(a)anthracene in mice.Proteomic analysis of the balance between survival and cell death responses in cisplatin-mediated ototoxicity.TSGΔ154-1054 splice variant increases TSG101 oncogenicity by inhibiting its E3-ligase-mediated proteasomal degradation.Endosomal sorting complex required for transport proteins in cancer pathogenesis, vesicular transport, and non-endosomal functions.Identification of TSG101 functional domains and p21 loci required for TSG101-mediated p21 gene regulationAt the crossroads of polarity, proliferation and apoptosis: the use of Drosophila to unravel the multifaceted role of endocytosis in tumor suppression.Developmental and cellular functions of the ESCRT machinery in pluricellular organisms.ESCRT & Co.Expression of TSG101 protein and LSF transcription factor in HPV-positive cervical cancer cells.Shades of meaning: the pigment-type switching system as a tool for discovery.Functional ESCRT machinery is required for constitutive recycling of claudin-1 and maintenance of polarity in vertebrate epithelial cellsExtracellular Vesicles: Unique Intercellular Delivery Vehicles.An upstream regulatory element confers orientation-independent enhancement of the TSG101 promoter activity in transformed cells.Oligodendroglial deletion of ESCRT-I component TSG101 causes spongiform encephalopathy.Up-regulation of tumor susceptibility gene 101 protein in ovarian carcinomas revealed by proteomics analyses.
P2860
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P2860
Tsg101 is essential for cell growth, proliferation, and cell survival of embryonic and adult tissues.
description
2003 nî lūn-bûn
@nan
2003 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
Tsg101 is essential for cell g ...... f embryonic and adult tissues.
@ast
Tsg101 is essential for cell g ...... f embryonic and adult tissues.
@en
Tsg101 is essential for cell g ...... f embryonic and adult tissues.
@nl
type
label
Tsg101 is essential for cell g ...... f embryonic and adult tissues.
@ast
Tsg101 is essential for cell g ...... f embryonic and adult tissues.
@en
Tsg101 is essential for cell g ...... f embryonic and adult tissues.
@nl
prefLabel
Tsg101 is essential for cell g ...... f embryonic and adult tissues.
@ast
Tsg101 is essential for cell g ...... f embryonic and adult tissues.
@en
Tsg101 is essential for cell g ...... f embryonic and adult tissues.
@nl
P2093
P2860
P1476
Tsg101 is essential for cell g ...... f embryonic and adult tissues.
@en
P2093
Aleata A Triplett
Andrea Krempler
Edmund B Rucker III
Gregory Riedlinger
Kay-Uwe Wagner
KyungRan Park
MaLinda D Henry
Yongyue Qi
P2860
P304
P356
10.1128/MCB.23.1.150-162.2003
P407
P577
2003-01-01T00:00:00Z