TL1, a novel tumor necrosis factor-like cytokine, induces apoptosis in endothelial cells. Involvement of activation of stress protein kinases (stress-activated protein kinase and p38 mitogen-activated protein kinase) and caspase-3-like protease.
about
The TNF-family cytokine TL1A: from lymphocyte costimulator to disease co-conspiratorStress-responsive JNK mitogen-activated protein kinase mediates aspirin-induced suppression of B16 melanoma cellular proliferationVascular endothelial growth inhibitor (VEGI), an endogenous negative regulator of angiogenesisOverexpression of phospholipid hydroperoxide glutathione peroxidase modulates acetyl-CoA, 1-O-alkyl-2-lyso-sn-glycero-3-phosphocholine acetyltransferase activityLITAF and TNFSF15, two downstream targets of AMPK, exert inhibitory effects on tumor growthCross-platform expression profiling demonstrates that SV40 small tumor antigen activates Notch, Hedgehog, and Wnt signaling in human cellsVEGI, a new member of the TNF family activates nuclear factor-kappa B and c-Jun N-terminal kinase and modulates cell growth.mda-7 (IL-24) Mediates selective apoptosis in human melanoma cells by inducing the coordinated overexpression of the GADD family of genes by means of p38 MAPK.Hematopoietic factor erythropoietin fosters neuroprotection through novel signal transduction cascades.Two functionally distinct isoforms of TL1A (TNFSF15) generated by differential ectodomain sheddingFunctional genetic variants of TNFSF15 and their association with gastric adenocarcinoma: a case-control studyIsoform of vascular endothelial cell growth inhibitor (VEGI72-251) increases interleukin-2 production by activation of T lymphocytes.p38 mitogen-activated protein kinase mediates the Fas-induced mitochondrial death pathway in CD8+ T cells.Vascular endothelial growth inhibitor (VEGI; TNFSF15) inhibits bone marrow-derived endothelial progenitor cell incorporation into Lewis lung carcinoma tumors.Suppression or induction of apoptosis by opposing pathways downstream from calcium-activated calcineurinPolymorphisms in PRKCDBP, a Transcriptional Target of TNF-α, Are Associated With Inflammatory Bowel Disease in Korean.TNFSF15 Modulates Neovascularization and InflammationDriving cellular plasticity and survival through the signal transduction pathways of metabotropic glutamate receptors.Cigarette smoke-induced CXCR3 receptor up-regulation mediates endothelial apoptosis.Preparation and characterization of a novel chimeric protein VEGI-CTT in Escherichia coli.Inhibition of endothelial progenitor cell differentiation by VEGI.TNFSF15 suppresses VEGF production in endothelial cells by stimulating miR-29b expression via activation of JNK-GATA3 signals.Advancement in the research on vascular endothelial growth inhibitor (VEGI).Functional CB1 cannabinoid receptors in human vascular endothelial cells.Review: Novel Insights Into Tumor Necrosis Factor Receptor, Death Receptor 3, and Progranulin Pathways in Arthritis and Bone Remodeling.Decoy receptor 3 (DcR3) induces osteoclast formation from monocyte/macrophage lineage precursor cells.TGF-β1 induces endothelial cell apoptosis by shifting VEGF activation of p38(MAPK) from the prosurvival p38β to proapoptotic p38α.Characterization of cis-regulatory elements of the vascular endothelial growth inhibitor gene promoter.VEGI-armed oncolytic adenovirus inhibits tumor neovascularization and directly induces mitochondria-mediated cancer cell apoptosis.Association of TNFSF15 with Crohn's disease in Koreans.Systemic and local expression levels of TNF-like ligand 1A and its decoy receptor 3 are increased in primary biliary cirrhosis.Topical dimethyl sulfoxide inhibits corneal neovascularization and stimulates corneal repair in rabbits following acid burn.Fas ligand-induced murine pulmonary inflammation is reduced by a stable decoy receptor 3 analogue.Expression of vascular endothelial growth factor receptors and their ligands in rat uterus during the postpartum involution period.Counterbalance: modulation of VEGF/VEGFR activities by TNFSF15
P2860
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P2860
TL1, a novel tumor necrosis factor-like cytokine, induces apoptosis in endothelial cells. Involvement of activation of stress protein kinases (stress-activated protein kinase and p38 mitogen-activated protein kinase) and caspase-3-like protease.
description
1999 nî lūn-bûn
@nan
1999 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի հունվարին հրատարակված գիտական հոդված
@hy
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
name
TL1, a novel tumor necrosis fa ...... ) and caspase-3-like protease.
@ast
TL1, a novel tumor necrosis fa ...... ) and caspase-3-like protease.
@en
TL1, a novel tumor necrosis fa ...... ) and caspase-3-like protease.
@nl
type
label
TL1, a novel tumor necrosis fa ...... ) and caspase-3-like protease.
@ast
TL1, a novel tumor necrosis fa ...... ) and caspase-3-like protease.
@en
TL1, a novel tumor necrosis fa ...... ) and caspase-3-like protease.
@nl
prefLabel
TL1, a novel tumor necrosis fa ...... ) and caspase-3-like protease.
@ast
TL1, a novel tumor necrosis fa ...... ) and caspase-3-like protease.
@en
TL1, a novel tumor necrosis fa ...... ) and caspase-3-like protease.
@nl
P2093
P2860
P356
P1476
TL1, a novel tumor necrosis fa ...... ) and caspase-3-like protease.
@en
P2093
A M Romanic
G Z Feuerstein
P2860
P304
P356
10.1074/JBC.274.3.1479
P407
P577
1999-01-01T00:00:00Z