Homologous recombination prevents methylation-induced toxicity in Escherichia coli.
about
A new role for translation initiation factor 2 in maintaining genome integrityBleomycin sensitivity in Escherichia coli is medium-dependentO6-Methylguanine DNA lesions induce an intra-S-phase arrest from which cells exit into apoptosis governed by early and late multi-pathway signaling network activation.Identification and characterization of human apurinic/apyrimidinic endonuclease-1 inhibitorsDNA Mismatch Repair.Characterization of the Neisseria meningitidis Helicase RecG.Mismatch repair-dependent processing of methylation damage gives rise to persistent single-stranded gaps in newly replicated DNA.DNA Polymerases ImuC and DinB Are Involved in DNA Alkylation Damage Tolerance in Pseudomonas aeruginosa and Pseudomonas putidaDNA mismatch repair-induced double-strand breaks.Methylating agents and DNA repair responses: Methylated bases and sources of strand breaks.Interplay of DNA repair pathways controls methylation damage toxicity in Saccharomyces cerevisiaeO6-methylguanine-induced cell death involves exonuclease 1 as well as DNA mismatch recognition in vivo.Characterization of the mycobacterial NER system reveals novel functions of the uvrD1 helicaseArylphosphonium salts interact with DNA to modulate cytotoxicity.RAD51D protects against MLH1-dependent cytotoxic responses to O(6)-methylguanine.Reverse Gyrase Functions in Genome Integrity Maintenance by Protecting DNA Breaks In Vivo.Genetic interactions of DNA repair pathways in the pathogen Neisseria meningitidis.Suppression of repeat-mediated gross mitochondrial genome rearrangements by RecA in the moss Physcomitrella patens.Single-molecule imaging reveals multiple pathways for the recruitment of translesion polymerases after DNA damage.Inactivation of genes involved in base excision repair of Corynebacterium glutamicum and survival of the mutants in presence of various mutagens.Tiling array study of MNNG treated Escherichia coli reveals a widespread transcriptional response.RuvABC is required to resolve holliday junctions that accumulate following replication on damaged templates in Escherichia coli.
P2860
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P2860
Homologous recombination prevents methylation-induced toxicity in Escherichia coli.
description
2006 nî lūn-bûn
@nan
2006 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
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name
Homologous recombination prevents methylation-induced toxicity in Escherichia coli.
@ast
Homologous recombination prevents methylation-induced toxicity in Escherichia coli.
@en
Homologous recombination prevents methylation-induced toxicity in Escherichia coli.
@nl
type
label
Homologous recombination prevents methylation-induced toxicity in Escherichia coli.
@ast
Homologous recombination prevents methylation-induced toxicity in Escherichia coli.
@en
Homologous recombination prevents methylation-induced toxicity in Escherichia coli.
@nl
prefLabel
Homologous recombination prevents methylation-induced toxicity in Escherichia coli.
@ast
Homologous recombination prevents methylation-induced toxicity in Escherichia coli.
@en
Homologous recombination prevents methylation-induced toxicity in Escherichia coli.
@nl
P2093
P2860
P356
P1476
Homologous recombination prevents methylation-induced toxicity in Escherichia coli.
@en
P2093
Anetta Nowosielska
Bevin P Engelward
M G Marinus
Stephen A Smith
P2860
P304
P356
10.1093/NAR/GKL222
P407
P577
2006-05-02T00:00:00Z