RhoA is dispensable for skin development, but crucial for contraction and directed migration of keratinocytes - Test2 - elhamkhani - TriplyDB

RhoA is dispensable for skin development, but crucial for contraction and directed migration of keratinocytes

RhoA is dispensable for skin development, but crucial for contraction and directed migration of keratinocytes

http://www.wikidata.org/entity/Q34611649

about

Rho GTPase signalling in cell migration Palatogenesis and cutaneous repair: A two-headed coin Rgnef (p190RhoGEF) knockout inhibits RhoA activity, focal adhesion establishment, and cell motility downstream of integrins The small GTPase RhoA is required for proper locomotor circuit assembly Differential effects of phosphatase inhibitors on the calcium homeostasis and migration of HaCaT keratinocytes Rho-A prenylation and signaling link epithelial homeostasis to intestinal inflammation.Gene targeting RhoA reveals its essential role in coordinating mitochondrial function and thymocyte development Mouse macrophages completely lacking Rho subfamily GTPases (RhoA, RhoB, and RhoC) have severe lamellipodial retraction defects, but robust chemotactic navigation and altered motility.Plakophilin 2 affects cell migration by modulating focal adhesion dynamics and integrin protein expression.RhoA of the Rho family small GTPases is essential for B lymphocyte development.Desmoglein 3 acting as an upstream regulator of Rho GTPases, Rac-1/Cdc42 in the regulation of actin organisation and dynamics.Interferon regulatory factor 6 regulates keratinocyte migration.The effect of secretory factors of adipose-derived stem cells on human keratinocytes.Keratin 5-Cre-driven excision of nonmuscle myosin IIA in early embryo trophectoderm leads to placenta defects and embryonic lethality.RhoA GTPase is dispensable for actomyosin regulation but is essential for mitosis in primary mouse embryonic fibroblasts Loss of RhoA in neural progenitor cells causes the disruption of adherens junctions and hyperproliferation.Rhophilin-1 is a key regulator of the podocyte cytoskeleton and is essential for glomerular filtration.Digital imaging analysis to assess scar phenotype.Microneedle fractional radiofrequency increases epidermal hyaluronan and reverses age-related epidermal dysfunction.Suppression of the migration and invasion is mediated by triptolide in B16F10 mouse melanoma cells through the NF-kappaB-dependent pathway.Podocyte-specific loss of Cdc42 leads to congenital nephropathy Whole genome microarray data of chronic wound debridement prior to application of dermal skin substitutes.Rho GTPases in embryonic development Mimicking normal tissue architecture and perturbation in cancer with engineered micro-epidermis Triptolide inhibits ovarian cancer cell invasion by repression of matrix metalloproteinase 7 and 19 and upregulation of E-cadherin A comparison of epithelial-to-mesenchymal transition and re-epithelialization The GEF Bcr activates RhoA/MAL signaling to promote keratinocyte differentiation via desmoglein-1 RhoA GTPase controls cytokinesis and programmed necrosis of hematopoietic progenitors Cell type-specific signaling function of RhoA GTPase: lessons from mouse gene targeting.Cdc42 inhibits ERK-mediated collagenase-1 (MMP-1) expression in collagen-activated human keratinocytes.Identification of common non-coding variants at 1p22 that are functional for non-syndromic orofacial clefting.Rho GTPases in animal cell cytokinesis: an occupation by the one percent.Matrix hyaluronan-activated CD44 signaling promotes keratinocyte activities and improves abnormal epidermal functions.Integrin-mediated adhesion and mechano-sensing in cutaneous wound healing.Blocking RhoA/ROCK inhibits the pathogenesis of pemphigus vulgaris by suppressing oxidative stress and apoptosis through TAK1/NOD2-mediated NF-κB pathway.Dynamics of focal adhesions and reorganization of F-actin in VEGF-stimulated NSCs under varying differentiation states.RhoA controls retinoid signaling by ROCK dependent regulation of retinol metabolism.Rho GTPases and cancer.A GAL4-inducible transgenic tool kit for the in vivo modulation of Rho GTPase activity in zebrafish Cytoskeletal Stability in the Auditory Organ In Vivo: RhoA Is Dispensable for Wound Healing but Essential for Hair Cell Development.

P2860

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P2860

RhoA is dispensable for skin development, but crucial for contraction and directed migration of keratinocytes

http://www.wikidata.org/entity/Q34611649

description

2011 nî lūn-bûn

@nan

2011 թուականի Յունուարին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի հունվարին հրատարակված գիտական հոդված

@hy

2011年の論文

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2011年論文

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2011年論文

@zh-hant

2011年論文

@zh-hk

2011年論文

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2011年論文

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2011年论文

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name

RhoA is dispensable for skin d ...... ted migration of keratinocytes

@ast

RhoA is dispensable for skin d ...... ted migration of keratinocytes

@en

RhoA is dispensable for skin d ...... ted migration of keratinocytes

@nl

type

label

RhoA is dispensable for skin d ...... ted migration of keratinocytes

@ast

RhoA is dispensable for skin d ...... ted migration of keratinocytes

@en

RhoA is dispensable for skin d ...... ted migration of keratinocytes

@nl

prefLabel

RhoA is dispensable for skin d ...... ted migration of keratinocytes

@ast

RhoA is dispensable for skin d ...... ted migration of keratinocytes

@en

RhoA is dispensable for skin d ...... ted migration of keratinocytes

@nl

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MBC.E09-10-0859

P1433

Molecular Biology of the Cell

P1476

RhoA is dispensable for skin d ...... ted migration of keratinocytes

@en

P2093

Alexandra M Ochsenbein

http://www.w3.org/2001/XMLSchema#string

Astrid Basse

http://www.w3.org/2001/XMLSchema#string

Ben Jackson

http://www.w3.org/2001/XMLSchema#string

Esben Pedersen

http://www.w3.org/2001/XMLSchema#string

Gudula Schmidt

http://www.w3.org/2001/XMLSchema#string

Jolanda van Hengel

http://www.w3.org/2001/XMLSchema#string

Karine Peyrollier

http://www.w3.org/2001/XMLSchema#string

Markus Ladwein

http://www.w3.org/2001/XMLSchema#string

Richard Karlsson

http://www.w3.org/2001/XMLSchema#string

Tine Lefever

http://www.w3.org/2001/XMLSchema#string

P2860

Rho GTPases have diverse effects on the organization of the actin filament system

RhoC is dispensable for embryogenesis and tumor initiation but essential for metastasis.

The Rho-mDia1 pathway regulates cell polarity and focal adhesion turnover in migrating cells through mobilizing Apc and c-Src

GTP-binding proteins of the Rho/Rac family: regulation, effectors and functions in vivo

Cell migration: integrating signals from front to back

Regulation of cell polarity and protrusion formation by targeting RhoA for degradation

Why three Rho proteins? RhoA, RhoB, RhoC, and cell motility

Actin-depolymerizing factor and cofilin-1 play overlapping roles in promoting rapid F-actin depolymerization in mammalian nonmuscle cells.

Cdc42 controls progenitor cell differentiation and beta-catenin turnover in skin

RhoB is dispensable for mouse development, but it modifies susceptibility to tumor formation as well as cell adhesion and growth factor signaling in transformed cells

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593-605

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scholarly article

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10.1091/MBC.E09-10-0859

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5

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22

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Cord Brakebusch

Klemens Rottner

Fabio Quondamatteo

P577

2011-01-05T00:00:00Z

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49731768

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21209320

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3046057

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