Senescence and apoptosis: dueling or complementary cell fates? - Test2 - elhamkhani - TriplyDB

Senescence and apoptosis: dueling or complementary cell fates?

Senescence and apoptosis: dueling or complementary cell fates?

http://www.wikidata.org/entity/Q34619554

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Hallmarks of progeroid syndromes: lessons from mice and reprogrammed cells Cellular senescence in aging and age-related disease: from mechanisms to therapy Cellular Response upon Stress: p57 Contribution to the Final Outcome DNA damage response (DDR) and senescence: shuttled inflamma-miRNAs on the stage of inflamm-aging Small molecule compounds that induce cellular senescence The cell biology of aging Aging of mice is associated with p16(Ink4a)- and β-galactosidase-positive macrophage accumulation that can be induced in young mice by senescent cells Age, gender and UV-exposition related effects on gene expression in in vivo aged short term cultivated human dermal fibroblasts Oncogenic senescence: a multi-functional perspective.Exosomes maintain cellular homeostasis by excreting harmful DNA from cells.Potency of Human Cardiosphere-Derived Cells from Patients with Ischemic Heart Disease Is Associated with Robust Vascular Supportive Ability.Clearance of senescent cells by ABT263 rejuvenates aged hematopoietic stem cells in mice Exercise promotes motor functional recovery in rats with corticospinal tract injury: anti-apoptosis mechanism Acquisition of estrogen independence induces TOB1-related mechanisms supporting breast cancer cell proliferation.Programmed cell death in aging.Induction of Cell Death in Growing Human T-Cells and Cell Survival in Resting Cells in Response to the Human T-Cell Leukemia Virus Type 1 Tax.The tumor suppressor ING1b is a novel corepressor for the androgen receptor and induces cellular senescence in prostate cancer cells.Depletion of γ-glutamylcyclotransferase inhibits breast cancer cell growth via cellular senescence induction mediated by CDK inhibitor upregulation.SIRT6 Depletion Suppresses Tumor Growth by Promoting Cellular Senescence Induced by DNA Damage in HCC.Sustained accumulation of prelamin A and depletion of lamin A/C both cause oxidative stress and mitochondrial dysfunction but induce different cell fates RAC3 more than a nuclear receptor coactivator: a key inhibitor of senescence that is downregulated in aging.Modes of Cell Death Induced by Photodynamic Therapy Using Zinc Phthalocyanine in Lung Cancer Cells Grown as a Monolayer and Three-Dimensional Multicellular Spheroids.Context-dependent effects of cellular senescence in cancer development.HBP1-mediated Regulation of p21 Protein through the Mdm2/p53 and TCF4/EZH2 Pathways and Its Impact on Cell Senescence and Tumorigenesis.Activation of p53 in Down Syndrome and in the Ts65Dn Mouse Brain is Associated with a Pro-Apoptotic Phenotype.Ionizing Radiation-Induced Endothelial Cell Senescence and Cardiovascular Diseases.Changes in the Transcriptome of Human Astrocytes Accompanying Oxidative Stress-Induced Senescence.Cristacarpin promotes ER stress-mediated ROS generation leading to premature senescence by activation of p21(waf-1).Apoptotic transition of senescent cells accompanied with mitochondrial hyper-function.Surfactin inducing mitochondria-dependent ROS to activate MAPKs, NF-κB and inflammasomes in macrophages for adjuvant activity.DNA damage induced by Strontium-90 exposure at low concentrations in mesenchymal stromal cells: the functional consequences EZH2 inhibition re-sensitizes multidrug resistant B-cell lymphomas to etoposide mediated apoptosis.The role of telomeres and vitamin D in cellular aging and age-related diseases.Resistance to RHD virus in wild Australian rabbits: Comparison of susceptible and resistant individuals using a genomewide approach.Molecular interplays in hepatic stellate cells: apoptosis, senescence, and phenotype reversion as cellular connections that modulate liver fibrosis.Murine mesenchymal cells that express elevated levels of the CDK inhibitor p16(Ink4a) in vivo are not necessarily senescent.Fetuin-A (alpha 2HS glycoprotein) modulates growth, motility, invasion, and senescence in high-grade astrocytomas.Secreted factors from equine mesenchymal stromal cells diminish the effects of TGF-β1 on equine dermal fibroblasts and alter the phenotype of dermal fibroblasts isolated from cutaneous fibroproliferative wounds.Fate of microglia during HIV-1 infection: From activation to senescence?Akt inhibition improves irinotecan treatment and prevents cell emergence by switching the senescence response to apoptosis.

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P2860

Senescence and apoptosis: dueling or complementary cell fates?

http://www.wikidata.org/entity/Q34619554

description

2014 nî lūn-bûn

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2014 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի հոտեմբերին հրատարակված գիտական հոդված

@hy

2014年の論文

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2014年論文

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2014年論文

@zh-hant

2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Senescence and apoptosis: dueling or complementary cell fates?

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Senescence and apoptosis: dueling or complementary cell fates?

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Senescence and apoptosis: dueling or complementary cell fates?

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type

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Senescence and apoptosis: dueling or complementary cell fates?

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Senescence and apoptosis: dueling or complementary cell fates?

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Senescence and apoptosis: dueling or complementary cell fates?

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Senescence and apoptosis: dueling or complementary cell fates?

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Senescence and apoptosis: dueling or complementary cell fates?

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Senescence and apoptosis: dueling or complementary cell fates?

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Senescence and apoptosis: dueling or complementary cell fates?

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Bennett G Childs

http://www.w3.org/2001/XMLSchema#string

Darren J Baker

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James L Kirkland

http://www.w3.org/2001/XMLSchema#string

Jan M van Deursen

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Judith Campisi

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P2860

Premature senescence involving p53 and p16 is activated in response to constitutive MEK/MAPK mitogenic signaling

An APAF-1.cytochrome c multimeric complex is a functional apoptosome that activates procaspase-9

Regulation of endothelium-derived nitric oxide production by the protein kinase Akt

Telomere shortening triggers senescence of human cells through a pathway involving ATM, p53, and p21(CIP1), but not p16(INK4a)

Prevention of apoptosis by Bcl-2: release of cytochrome c from mitochondria blocked

Functional connection between p53 and caspase-2 is essential for apoptosis induced by DNA damage

Extension of life-span by introduction of telomerase into normal human cells

Chk1 and p21 cooperate to prevent apoptosis during DNA replication fork stress

Cellular senescence controls fibrosis in wound healing

Senescence of activated stellate cells limits liver fibrosis

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10.15252/EMBR.201439245

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11

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15

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