High trans-ethnic replicability of GWAS results implies common causal variants.
about
Uncovering the Genetic Architectures of Quantitative TraitsPancreatic Cancer GeneticsGenetic and nongenetic risk factors for childhood cancerTrans-ethnic genome-wide association studies: advantages and challenges of mapping in diverse populations.267 Spanish Exomes Reveal Population-Specific Differences in Disease-Related Genetic VariationLeveraging Functional-Annotation Data in Trans-ethnic Fine-Mapping Studies.Properties of human disease genes and the role of genes linked to Mendelian disorders in complex disease aetiology.The impact of population demography and selection on the genetic architecture of complex traits.A population genetic signal of polygenic adaptation.Principal component analysis characterizes shared pathogenetics from genome-wide association studies.Identifying causal variants at loci with multiple signals of associationTransethnic replication of association of CTG18.1 repeat expansion of TCF4 gene with Fuchs' corneal dystrophy in Chinese implies common causal variantMeta-analysis of genome-wide association studies identifies two loci associated with circulating osteoprotegerin levelsThe National Longitudinal Study of Adolescent to Adult Health (Add Health) sibling pairs genome-wide data.Fine-mapping the HOXB region detects common variants tagging a rare coding allele: evidence for synthetic association in prostate cancer.Meta-analysis of genome-wide association studies of adult height in East Asians identifies 17 novel lociPolygenic risk for externalizing disorders: Gene-by-development and gene-by-environment effects in adolescents and young adultsGeographical, environmental and pathophysiological influences on the human blood transcriptome.Genome-wide meta-analysis identifies multiple novel associations and ethnic heterogeneity of psoriasis susceptibility.Cross-population joint analysis of eQTLs: fine mapping and functional annotation.High-density genotyping of immune loci in Koreans and Europeans identifies eight new rheumatoid arthritis risk loci.Whole genome sequences are required to fully resolve the linkage disequilibrium structure of human populationsChildhood cancer: an emerging public health issue in China.The Fourth Law of Behavior Genetics.Identification of novel candidate variants including COL6A6 polymorphisms in early-onset atopic dermatitis using whole-exome sequencingBMD Loci Contribute to Ethnic and Developmental Differences in Skeletal Fragility across Populations: Assessment of Evolutionary Selection Pressures.Complex disease and phenotype mapping in the domestic dog.Evaluation of type 2 diabetes genetic risk variants in Chinese adults: findings from 93,000 individuals from the China Kadoorie Biobank.Deciphering the genetic architecture of low-penetrance susceptibility to colorectal cancer.Studying the Genetics of Complex Disease With Ancestry-Specific Human Phenotype Networks: The Case of Type 2 Diabetes in East Asian Populations.Evaluating the transferability of 15 European-derived fasting plasma glucose SNPs in Mexican children and adolescents.Combining Multiple Hypothesis Testing with Machine Learning Increases the Statistical Power of Genome-wide Association Studies.Comprehensive functional annotation of 77 prostate cancer risk loci.Genetics of rheumatoid arthritis in Asia--present and future.10 Years of GWAS Discovery: Biology, Function, and Translation.GWAS identifies population-specific new regulatory variants in FUT6 associated with plasma B12 concentrations in Indians.The Genetic Architecture of Coronary Artery Disease: Current Knowledge and Future Opportunities.Trans-ethnic meta-analysis of genome-wide association studies for Hirschsprung disease.Genomic analysis of the blood attributed to Louis XVI (1754-1793), king of France.Trans-ethnic meta-regression of genome-wide association studies accounting for ancestry increases power for discovery and improves fine-mapping resolution.
P2860
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P2860
High trans-ethnic replicability of GWAS results implies common causal variants.
description
2013 nî lūn-bûn
@nan
2013 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
name
High trans-ethnic replicability of GWAS results implies common causal variants.
@ast
High trans-ethnic replicability of GWAS results implies common causal variants.
@en
High trans-ethnic replicability of GWAS results implies common causal variants.
@nl
type
label
High trans-ethnic replicability of GWAS results implies common causal variants.
@ast
High trans-ethnic replicability of GWAS results implies common causal variants.
@en
High trans-ethnic replicability of GWAS results implies common causal variants.
@nl
prefLabel
High trans-ethnic replicability of GWAS results implies common causal variants.
@ast
High trans-ethnic replicability of GWAS results implies common causal variants.
@en
High trans-ethnic replicability of GWAS results implies common causal variants.
@nl
P2860
P1433
P1476
High trans-ethnic replicability of GWAS results implies common causal variants
@en
P2093
Arcadi Navarro
P2860
P304
P356
10.1371/JOURNAL.PGEN.1003566
P577
2013-06-13T00:00:00Z