Sensitivity to hepatotoxicity due to epigallocatechin gallate is affected by genetic background in diversity outbred mice - Test2 - elhamkhani - TriplyDB

Sensitivity to hepatotoxicity due to epigallocatechin gallate is affected by genetic background in diversity outbred mice

Sensitivity to hepatotoxicity due to epigallocatechin gallate is affected by genetic background in diversity outbred mice

http://www.wikidata.org/entity/Q35071460

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Circulating mitochondrial biomarkers for drug-induced liver injury Emerging Phytochemicals for the Prevention and Treatment of Head and Neck Cancer Collaborative Cross and Diversity Outbred data resources in the Mouse Phenome Database The safety of green tea extract supplementation in postmenopausal women at risk for breast cancer: results of the Minnesota Green Tea Trial.The anthocyanin cyanidin-3-O-β-glucoside modulates murine glutathione homeostasis in a manner dependent on genetic background Diversity Outbred Mice at 21: Maintaining Allelic Variation in the Face of Selection.Hepatotoxicity of green tea: an update.A comprehensive overview of hepatoprotective natural compounds: mechanism of action and clinical perspectives.The Promise of New Technologies to Reduce, Refine, or Replace Animal Use while Reducing Risks of Drug Induced Liver Injury in Pharmaceutical Development.Nutritional Epigenetics and the Prevention of Hepatocellular Carcinoma with Bioactive Food Constituents.Herbal and Dietary Supplement-Induced Liver Injury.Mapping Complex Traits in a Diversity Outbred F1 Mouse Population Identifies Germline Modifiers of Metastasis in Human Prostate Cancer.Epigallocatechin-3-gallate treatment to promote neuroprotection and functional recovery after nervous system injury Editor's Highlight: Collaborative Cross Mouse Population Enables Refinements to Characterization of the Variability in Toxicokinetics of Trichloroethylene and Provides Genetic Evidence for the Role of PPAR Pathway in Its Oxidative Metabolism.Mouse population-based evaluation of urinary protein and miRNA biomarker performance associated with cisplatin renal injury.A Synopsis of the "Influence of Epigenetics, Genetics, and Immunology" Session Part A at the 35th Annual Society of Toxicologic Pathology Symposium.Incorporating population-level genetic variability within laboratory models in toxicology: From the individual to the population.Inter-individual variation in health and disease associated with pulmonary infectious agents.New Rodent Population Models May Inform Human Health Risk Assessment and Identification of Genetic Susceptibility to Environmental Exposures.Selenium regulation of selenoprotein enzyme activity and transcripts in a pilot study with Founder strains from the Collaborative Cross.Population-based dose-response analysis of liver transcriptional response to trichloroethylene in mouse.Epigallocatechin-3-gallate treatment reduces thermal hyperalgesia after spinal cord injury by down-regulating RhoA expression in mice.Inter-individual variation in adaptations to endurance and resistance exercise training: genetic approaches towards understanding a complex phenotype.Silicosis and Silica-Induced Autoimmunity in the Diversity Outbred Mouse.

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P2860

Sensitivity to hepatotoxicity due to epigallocatechin gallate is affected by genetic background in diversity outbred mice

http://www.wikidata.org/entity/Q35071460

description

2014 nî lūn-bûn

@nan

2014 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Sensitivity to hepatotoxicity ...... ound in diversity outbred mice

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Sensitivity to hepatotoxicity ...... ound in diversity outbred mice

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type

label

Sensitivity to hepatotoxicity ...... ound in diversity outbred mice

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Sensitivity to hepatotoxicity ...... ound in diversity outbred mice

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prefLabel

Sensitivity to hepatotoxicity ...... ound in diversity outbred mice

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Sensitivity to hepatotoxicity ...... ound in diversity outbred mice

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P356

J.FCT.2014.11.008

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Food and Chemical Toxicology

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Sensitivity to hepatotoxicity ...... ound in diversity outbred mice

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P2093

Daniel M Gatti

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Gary A Churchill

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J Scott Eaddy

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Nanye Long

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Paul B Watkins

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Qiang Shi

http://www.w3.org/2001/XMLSchema#string

Rachel J Church

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Shawn Ballard

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Thomas J Urban

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Victor Navarro

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P2860

A mouse diversity panel approach reveals the potential for clinical kidney injury due to DB289 not predicted by classical rodent models

Hydroxycut hepatotoxicity: a case series and review of liver toxicity from herbal weight loss supplements

Genetic variation in IL28B predicts hepatitis C treatment-induced viral clearance

In situ detection of fragmented DNA (TUNEL assay) fails to discriminate among apoptosis, necrosis, and autolytic cell death: a cautionary note

PINK1-phosphorylated mitofusin 2 is a Parkin receptor for culling damaged mitochondria

Mitofusins Mfn1 and Mfn2 coordinately regulate mitochondrial fusion and are essential for embryonic development

In situ detection of fragmented dna (tunel assay) fails to discriminate among apoptosis, necrosis, and autolytic cell death: A cautionary note

Mitofusin-2 maintains mitochondrial structure and contributes to stress-induced permeability transition in cardiac myocytes.

The polymorphism architecture of mouse genetic resources elucidated using genome-wide resequencing data: implications for QTL discovery and systems genetics.

Hepatotoxicity of high oral dose (-)-epigallocatechin-3-gallate in mice.

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19-26

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scholarly article

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10.1016/J.FCT.2014.11.008

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76

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David W. Threadgill

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2014-11-28T00:00:00Z

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269177190

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