Neuronal MeCP2 is expressed at near histone-octamer levels and globally alters the chromatin state. - Test2 - elhamkhani - TriplyDB

Neuronal MeCP2 is expressed at near histone-octamer levels and globally alters the chromatin state.

Neuronal MeCP2 is expressed at near histone-octamer levels and globally alters the chromatin state.

http://www.wikidata.org/entity/Q35114553

about

Regulation of the nucleosome repeat length in vivo by the DNA sequence, protein concentrations and long-range interactions The epigenetic landscape of addiction Proximity of H2A.Z containing nucleosome to the transcription start site influences gene expression levels in the mammalian liver and brain Neonatal MeCP2 is important for the organization of sex differences in vasopressin expression Restricting retrotransposons: a review Modifiers and Readers of DNA Modifications and Their Impact on Genome Structure, Expression, and Stability in Disease MeCP2 and the enigmatic organization of brain chromatin. Implications for depression and cocaine addiction Rett Syndrome: Crossing the Threshold to Clinical Translation MECP2 disorders: from the clinic to mice and back MeCP2 post-translational modifications: a mechanism to control its involvement in synaptic plasticity and homeostasis?Epigenetics and psychostimulant addiction DNA modifications: function and applications in normal and disease States Astrocytes conspire with neurons during progression of neurological disease Cytosine modifications in neurodevelopment and diseases DNA methylation in mammals BDNF deregulation in Rett syndrome Epigenetics and drug addiction: a focus on MeCP2 and on histone acetylation Role of mecp2 in experience-dependent epigenetic programming MeCP2 Affects Skeletal Muscle Growth and Morphology through Non Cell-Autonomous Mechanisms Interactive actions of Bdnf methylation and cell metabolism for building neural resilience under the influence of diet Rett syndrome - biological pathways leading from MECP2 to disorder phenotypes A review of Rett syndrome (RTT) with induced pluripotent stem cells The Use of Induced Pluripotent Stem Cell Technology to Advance Autism Research and Treatment Rett syndrome: disruption of epigenetic control of postnatal neurological functions Orphan CpG islands identify numerous conserved promoters in the mammalian genome Histone methylation: a dynamic mark in health, disease and inheritance Association of modified cytosines and the methylated DNA-binding protein MeCP2 with distinctive structural domains of lampbrush chromatin Rett syndrome mutations abolish the interaction of MeCP2 with the NCoR/SMRT co-repressor Misregulation of Alternative Splicing in a Mouse Model of Rett Syndrome A brain-derived MeCP2 complex supports a role for MeCP2 in RNA processing Epigenetic suppression of neuroligin 1 underlies amyloid-induced memory deficiency Efficient and versatile CRISPR engineering of human neurons in culture to model neurological disorders Ex vivo treatment with a novel synthetic aminoglycoside NB54 in primary fibroblasts from Rett syndrome patients suppresses MECP2 nonsense mutations SUMOylation of MeCP2 is essential for transcriptional repression and hippocampal synapse development Methyl-CpG binding protein 2 (MeCP2) localizes at the centrosome and is required for proper mitotic spindle organization The Rett syndrome protein MeCP2 regulates synaptic scaling A critical and cell-autonomous role for MeCP2 in synaptic scaling up Dysfunction in GABA signalling mediates autism-like stereotypies and Rett syndrome phenotypes Unexpected cellular players in Rett syndrome pathology MECP2 regulates cortical plasticity underlying a learned behaviour in adult female mice

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Neuronal MeCP2 is expressed at near histone-octamer levels and globally alters the chromatin state.

http://www.wikidata.org/entity/Q35114553

description

2010 nî lūn-bûn

@nan

2010 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի փետրվարին հրատարակված գիտական հոդված

@hy

2010年の論文

@ja

2010年論文

@yue

2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

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name

Neuronal MeCP2 is expressed at ...... ly alters the chromatin state.

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Neuronal MeCP2 is expressed at ...... ly alters the chromatin state.

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Neuronal MeCP2 is expressed at ...... ly alters the chromatin state.

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Neuronal MeCP2 is expressed at ...... ly alters the chromatin state.

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Neuronal MeCP2 is expressed at ...... ly alters the chromatin state.

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J.MOLCEL.2010.01.030

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@en

P2093

Daniel J Turner

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Keith D James

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Peter J Skene

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Shaun Webb

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P2860

Rett syndrome is caused by mutations in X-linked MECP2, encoding methyl-CpG-binding protein 2

Transcriptional repression by the methyl-CpG-binding protein MeCP2 involves a histone deacetylase complex

Expression of MeCP2 in postmitotic neurons rescues Rett syndrome in mice

Amount and distribution of 5-methylcytosine in human DNA from different types of tissues of cells

MeCP2, a key contributor to neurological disease, activates and represses transcription

Integrated epigenomic analyses of neuronal MeCP2 reveal a role for long-range interaction with active genes

Chromatin compaction by human MeCP2. Assembly of novel secondary chromatin structures in the absence of DNA methylation

Deletions of a differentially methylated CpG island at the SNRPN gene define a putative imprinting control region

Purification of CpG islands using a methylated DNA binding column

Purification, sequence, and cellular localization of a novel chromosomal protein that binds to methylated DNA

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457-468

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4

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37

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Adrian Peter Bird

Robert S Illingworth

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2010-02-01T00:00:00Z

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41621252

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20188665

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4338610

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