Polμ deficiency increases resistance to oxidative damage and delays liver aging. - Test2 - elhamkhani - TriplyDB

Polμ deficiency increases resistance to oxidative damage and delays liver aging.

Polμ deficiency increases resistance to oxidative damage and delays liver aging.

http://www.wikidata.org/entity/Q35137565

about

Essential role for polymerase specialization in cellular nonhomologous end joining.Increased neuronal death and disturbed axonal growth in the Polμ-deficient mouse embryonic retina.Vitamin transporters in mice brain with aging.

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P2860

Polμ deficiency increases resistance to oxidative damage and delays liver aging.

http://www.wikidata.org/entity/Q35137565

description

2014 nî lūn-bûn

@nan

2014 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի ապրիլին հրատարակված գիտական հոդված

@hy

2014年の論文

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2014年論文

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2014年論文

@zh-hant

2014年論文

@zh-hk

2014年論文

@zh-mo

2014年論文

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2014年论文

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name

Polμ deficiency increases resistance to oxidative damage and delays liver aging.

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Polμ deficiency increases resistance to oxidative damage and delays liver aging.

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type

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Polμ deficiency increases resistance to oxidative damage and delays liver aging.

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Polμ deficiency increases resistance to oxidative damage and delays liver aging.

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Polμ deficiency increases resistance to oxidative damage and delays liver aging.

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Polμ deficiency increases resistance to oxidative damage and delays liver aging.

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JOURNAL.PONE.0093074

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Polμ deficiency increases resistance to oxidative damage and delays liver aging.

@en

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Antonio Bernad

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Beatriz Escudero

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Candelas Carreiro

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Carmen Albo

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Daniel Lucas

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Encarnación Fuster

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Enrique Samper

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Jeff W Bacher

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Luis Blanco

http://www.w3.org/2001/XMLSchema#string

Manuel A González

http://www.w3.org/2001/XMLSchema#string

P2860

Altered hematopoiesis in mice lacking DNA polymerase mu is due to inefficient double-strand break repair

Delayed and accelerated aging share common longevity assurance mechanisms

Deletion of Ku70, Ku80, or both causes early aging without substantially increased cancer

Does reduced IGF-1R signaling in Igf1r+/- mice alter aging?

Increased learning and brain long-term potentiation in aged mice lacking DNA polymerase μ

A gradient of template dependence defines distinct biological roles for family X polymerases in nonhomologous end joining.

Genome maintenance mechanisms for preventing cancer

Immunoglobulin kappa light chain gene rearrangement is impaired in mice deficient for DNA polymerase mu

Wld(S) reduces paraquat-induced cytotoxicity via SIRT1 in non-neuronal cells by attenuating the depletion of NAD

Polymerase mu is up-regulated during the T cell-dependent immune response and its deficiency alters developmental dynamics of spleen centroblasts

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e93074

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scholarly article

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10.1371/JOURNAL.PONE.0093074

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Margarita Fernández

Isabel Fabregat

Aránzazu Sánchez

Vicente Andrés

José Rivera-Torres

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2014-04-01T00:00:00Z

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