about
p19(ARF) deficiency reduces macrophage and vascular smooth muscle cell apoptosis and aggravates atherosclerosishMEF2C gene encodes skeletal muscle- and brain-specific transcription factorsSorting nexin 6 enhances lamin a synthesis and incorporation into the nuclear envelopeNestin(+) cells direct inflammatory cell migration in atherosclerosis.Nitric oxide-induced downregulation of Cdk2 activity and cyclin A gene transcription in vascular smooth muscle cellsNuclear envelope lamin-A couples actin dynamics with immunological synapse architecture and T cell activation.PI3K p110γ deletion attenuates murine atherosclerosis by reducing macrophage proliferation but not polarization or apoptosis in lesions.Synthesis, transport and incorporation into the nuclear envelope of A-type lamins and inner nuclear membrane proteins.The promoter activity of human Mfn2 depends on Sp1 in vascular smooth muscle cells.Inhibition of cellular proliferation by drug targeting of cyclin-dependent kinases.Identification of mitochondrial dysfunction in Hutchinson-Gilford progeria syndrome through use of stable isotope labeling with amino acids in cell culture.In vivo inhibition of c-MYC in myeloid cells impairs tumor-associated macrophage maturation and pro-tumoral activities.Clonal hematopoiesis associated with TET2 deficiency accelerates atherosclerosis development in miceInhibiting cyclin-dependent kinase/cyclin activity for the treatment of cancer and cardiovascular disease.Polμ deficiency increases resistance to oxidative damage and delays liver aging.Insulin receptor substrate-1 expression is increased in circulating leukocytes of patients with acute coronary syndromeControl of vascular cell proliferation and migration by cyclin-dependent kinase signalling: new perspectives and therapeutic potential.Novel phosphate-activated macrophages prevent ectopic calcification by increasing extracellular ATP and pyrophosphate.Myogenin expression, cell cycle withdrawal, and phenotypic differentiation are temporally separable events that precede cell fusion upon myogenesis.Regulation of Gax homeobox gene transcription by a combination of positive factors including myocyte-specific enhancer factor 2Telomere biology and cardiovascular disease.A new bipartite DNA-binding domain: cooperative interaction between the cut repeat and homeo domain of the cut homeo proteins.Murine models to investigate the influence of diabetic metabolism on the development of atherosclerosis and restenosis.Fast regulation of AP-1 activity through interaction of lamin A/C, ERK1/2, and c-Fos at the nuclear envelopeCardiac electrical defects in progeroid mice and Hutchinson-Gilford progeria syndrome patients with nuclear lamina alterations.Age-dependent defective TGF-beta1 signaling in patients undergoing coronary artery bypass grafting.Control of cell proliferation in atherosclerosis: insights from animal models and human studies.Role of A-type lamins in signaling, transcription, and chromatin organization.Aryl hydrocarbon receptor contributes to the MEK/ERK-dependent maintenance of the immature state of human dendritic cells.Targeting γ-secretases protect against angiotensin II-induced cardiac hypertrophy.Alternatively activated macrophages exhibit an anticalcifying activity dependent on extracellular ATP/pyrophosphate metabolism.Short Telomere Load, Telomere Length, and Subclinical Atherosclerosis: The PESA Study.A-type lamins and cardiovascular disease in premature aging syndromes.Tumor suppressor p27(Kip1) undergoes endolysosomal degradation through its interaction with sorting nexin 6.Plasma insulin levels predict the development of atherosclerosis when IRS2 deficiency is combined with severe hypercholesterolemia in apolipoprotein E-null mice.Oil Red O and Hematoxylin and Eosin Staining for Quantification of Atherosclerosis Burden in Mouse Aorta and Aortic Root.Role of E2F and ERK1/2 in STI571-mediated smooth muscle cell growth arrest and cyclin A transcriptional repression.A Novel Systems-Biology Algorithm for the Analysis of Coordinated Protein Responses Using Quantitative ProteomicsERK1/2 MAP kinases promote cell cycle entry by rapid, kinase-independent disruption of retinoblastoma-lamin A complexes.Ink4/Arf locus restores glucose tolerance and insulin sensitivity by reducing hepatic steatosis and inflammation in mice with impaired IRS2-dependent signalling.
P50
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P50
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P1053
I-6440-2014
P106
P21
P214
309149294240680521181
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P3829
P496
0000-0002-0125-7209
P734
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P7859
lccn-no2017061633