Emergence of fibroblasts with a proinflammatory epigenetically altered phenotype in severe hypoxic pulmonary hypertension - Test2 - elhamkhani - TriplyDB

Emergence of fibroblasts with a proinflammatory epigenetically altered phenotype in severe hypoxic pulmonary hypertension

Emergence of fibroblasts with a proinflammatory epigenetically altered phenotype in severe hypoxic pulmonary hypertension

http://www.wikidata.org/entity/Q35175427

about

Dysregulation of histone acetyltransferases and deacetylases in cardiovascular diseases Molecular pathogenesis of pulmonary arterial hypertension Fibroblasts in fibrosis: novel roles and mediators The adventitia: essential regulator of vascular wall structure and function NF-kappaB Signaling in Chronic Inflammatory Airway Disease Beyond the genome: epigenetic mechanisms in lung remodeling Frontiers in pulmonary hypertension in infants and children with bronchopulmonary dysplasia Emerging roles for histone deacetylases in pulmonary hypertension and right ventricular remodeling (2013 Grover Conference series)Acetyl-lysine erasers and readers in the control of pulmonary hypertension and right ventricular hypertrophy Megakaryocytic leukemia 1 (MKL1) regulates hypoxia induced pulmonary hypertension in rats Role of epigenetics in pulmonary hypertension Hypoxia-induced mitogenic factor (FIZZ1/RELMα) induces endothelial cell apoptosis and subsequent interleukin-4-dependent pulmonary hypertension TGF-β activation by bone marrow-derived thrombospondin-1 causes Schistosoma- and hypoxia-induced pulmonary hypertension.Inflammation and immunity in the pathogenesis of pulmonary arterial hypertension Adventitial fibroblasts induce a distinct proinflammatory/profibrotic macrophage phenotype in pulmonary hypertension.MicroRNA-124 controls the proliferative, migratory, and inflammatory phenotype of pulmonary vascular fibroblasts Obesity-related pulmonary arterial hypertension in rats correlates with increased circulating inflammatory cytokines and lipids and with oxidant damage in the arterial wall but not with hypoxia Restoration of impaired endothelial myocyte enhancer factor 2 function rescues pulmonary arterial hypertension.Suppressive effects of induced pluripotent stem cell-conditioned medium on in vitro hypertrophic scarring fibroblast activation.The role of inflammation in hypoxic pulmonary hypertension: from cellular mechanisms to clinical phenotypes.BMPR2 preserves mitochondrial function and DNA during reoxygenation to promote endothelial cell survival and reverse pulmonary hypertension Inflammation, Innate Immunity, and the Intestinal Stromal Cell Niche: Opportunities and Challenges.Identification of functional progenitor cells in the pulmonary vasculature.The reversal of pulmonary vascular remodeling through inhibition of p38 MAPK-alpha: a potential novel anti-inflammatory strategy in pulmonary hypertension.Evaluation of a mouse model of necrotic granuloma formation using C3HeB/FeJ mice for testing of drugs against Mycobacterium tuberculosis.Eosinophils and IL-33 Perpetuate Chronic Inflammation and Fibrosis in a Pediatric Population with Stricturing Crohn's Ileitis.Hypoxia induces unique proliferative response in adventitial fibroblasts by activating PDGFβ receptor-JNK1 signalling.Osteopontin is an endogenous modulator of the constitutively activated phenotype of pulmonary adventitial fibroblasts in hypoxic pulmonary hypertension.Attenuated Pik3r1 expression prevents insulin resistance and adipose tissue macrophage accumulation in diet-induced obese mice.Runx1 repression by histone deacetylation is critical for Setbp1-induced mouse myeloid leukemia development Pulmonary artery smooth muscle cell proliferation and migration in fetal lambs acclimatized to high-altitude long-term hypoxia: role of histone acetylation MAP kinase kinase kinase-2 (MEKK2) regulates hypertrophic remodeling of the right ventricle in hypoxia-induced pulmonary hypertension.Metabolic reprogramming and inflammation act in concert to control vascular remodeling in hypoxic pulmonary hypertension.Mechanics and mechanisms of pulmonary hypertension-Conference summary and translational perspectives.Selective class I histone deacetylase inhibition suppresses hypoxia-induced cardiopulmonary remodeling through an antiproliferative mechanism.Constitutive Reprogramming of Fibroblast Mitochondrial Metabolism in Pulmonary Hypertension.Schistosomiasis causes remodeling of pulmonary vessels in the lung in a heterogeneous localized manner: Detailed study.Transcription factors, transcriptional coregulators, and epigenetic modulation in the control of pulmonary vascular cell phenotype: therapeutic implications for pulmonary hypertension (2015 Grover Conference series).Reduced BMPR2 expression induces GM-CSF translation and macrophage recruitment in humans and mice to exacerbate pulmonary hypertension.Relevant issues in the pathology and pathobiology of pulmonary hypertension.

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P2860

Emergence of fibroblasts with a proinflammatory epigenetically altered phenotype in severe hypoxic pulmonary hypertension

http://www.wikidata.org/entity/Q35175427

description

2011 nî lūn-bûn

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2011 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի օգոստոսին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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Emergence of fibroblasts with ...... hypoxic pulmonary hypertension

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Emergence of fibroblasts with ...... hypoxic pulmonary hypertension

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Adil Anwar

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Amanda R Flockton

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B Alexandre McKeon

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Barbara Meyrick

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Carlos Barajas

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Derek Strassheim

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Douglas D Lemon

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Karim C El Kasmi

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Kurt R Stenmark

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Maria G Frid

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P2860

Novel histone deacetylase inhibitors in clinical trials as anti-cancer agents

HDAC inhibitors as anti-inflammatory agents

Fibroblast biology. Role of synovial fibroblasts in the pathogenesis of rheumatoid arthritis

Primer3 on the WWW for general users and for biologist programmers

Analyzing real-time PCR data by the comparative C(T) method

Epigenetics in human disease and prospects for epigenetic therapy

Arginase-1-expressing macrophages suppress Th2 cytokine-driven inflammation and fibrosis

Identification of markers that distinguish monocyte-derived fibrocytes from monocytes, macrophages, and fibroblasts

Antifibrotic activity of an inhibitor of histone deacetylases in DOCA-salt hypertensive rats

Neointimal macrophages colocalize with extracellular matrix gene expression in human atherosclerotic pulmonary arteries.

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