Amyloid accumulation is a late event in sporadic Alzheimer's disease-like pathology in nontransgenic rats.
about
Precocious Alterations of Brain Oscillatory Activity in Alzheimer's Disease: A Window of Opportunity for Early Diagnosis and TreatmentOn cognitive ecology and the environmental factors that promote Alzheimer disease: lessons from Octodon degus (Rodentia: Octodontidae)Genes of susceptibility to early neurodegenerative changes in the rat retina and brain: analysis by means of congenic strainsAmyloid β Protein and Alzheimer's Disease: When Computer Simulations Complement Experimental Studies.A comprehensive multiomics approach toward understanding the relationship between aging and dementia.Neuroprotective effects of salidroside through PI3K/Akt pathway activation in Alzheimer's disease modelsWhite matter degeneration in subjective cognitive decline: a diffusion tensor imaging study.Early pathogenic event of Alzheimer's disease documented in iPSCs from patients with PSEN1 mutations.Ion channel regulation by β-secretase BACE1 - enzymatic and non-enzymatic effects beyond Alzheimer's disease.Mitophagy and Alzheimer's Disease: Cellular and Molecular Mechanisms.An antioxidant specifically targeting mitochondria delays progression of Alzheimer's disease-like pathology.Mitochondria-targeted antioxidant SkQ1 reduces age-related alterations in the ultrastructure of the lacrimal gland.Melatonin Attenuates Memory Impairment, Amyloid-β Accumulation, and Neurodegeneration in a Rat Model of Sporadic Alzheimer's Disease.Melatonin attenuates impairments of structural hippocampal neuroplasticity in OXYS rats during active progression of Alzheimer's disease-like pathology.Association of cerebrovascular dysfunction with the development of Alzheimer's disease-like pathology in OXYS rats.Modulation of the expression of genes related to the system of amyloid-beta metabolism in the brain as a novel mechanism of ceftriaxone neuroprotective properties.
P2860
Q26770751-668946A8-EF67-4EE6-9A22-ADB9C10F9153Q28070356-8A6A110F-EBF2-4CB0-ADF1-FE75C800F928Q28559519-33D7081D-B092-4117-94C2-45F5C90298ABQ33893017-21B7E730-60DE-4DCC-8FD0-3490E91DF134Q34501570-0583681C-27D8-4CC1-8336-636D005FED33Q36787765-29F26643-A543-475F-8809-D366E9CB62DEQ37688073-AEACE8F7-1681-4032-A58A-0C96742281D5Q37702630-62FD944D-ADCD-499A-A30F-81EB00470DC0Q38851332-18C1100B-47C9-4237-A51D-B254A4FF4297Q39132336-9817E6C5-1082-45B5-84B2-93EE0D8E4E16Q39281122-AF2C9BEA-3D4E-4A44-9FBC-3E793F3E8941Q41861443-00BAEA34-D085-4E0F-BC9E-3E6086187889Q47612057-AB18E7FE-3BBB-4464-B406-A17EE6B21A14Q48169931-5D54A1F1-0946-4634-9004-C029775F637EQ50417222-A4806842-0CB5-4061-BCA6-AA4BFD03D44CQ55346138-96BADBD8-9830-4079-B756-3CBF2938EE51
P2860
Amyloid accumulation is a late event in sporadic Alzheimer's disease-like pathology in nontransgenic rats.
description
2015 nî lūn-bûn
@nan
2015 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2015 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
name
Amyloid accumulation is a late ...... thology in nontransgenic rats.
@ast
Amyloid accumulation is a late ...... thology in nontransgenic rats.
@en
type
label
Amyloid accumulation is a late ...... thology in nontransgenic rats.
@ast
Amyloid accumulation is a late ...... thology in nontransgenic rats.
@en
prefLabel
Amyloid accumulation is a late ...... thology in nontransgenic rats.
@ast
Amyloid accumulation is a late ...... thology in nontransgenic rats.
@en
P2093
P2860
P50
P356
P1433
P1476
Amyloid accumulation is a late ...... athology in nontransgenic rats
@en
P2093
Elena E Korbolina
Kseniya Yi Maksimova
Natalia A Stefanova
P2860
P304
P356
10.18632/ONCOTARGET.2751
P407
P577
2015-01-01T00:00:00Z