SOD1 (copper/zinc superoxide dismutase) deficiency drives amyloid β protein oligomerization and memory loss in mouse model of Alzheimer disease - Test2 - elhamkhani - TriplyDB

SOD1 (copper/zinc superoxide dismutase) deficiency drives amyloid β protein oligomerization and memory loss in mouse model of Alzheimer disease

SOD1 (copper/zinc superoxide dismutase) deficiency drives amyloid β protein oligomerization and memory loss in mouse model of Alzheimer disease

http://www.wikidata.org/entity/Q35639816

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The Therapeutic Potential of Rosemary (Rosmarinus officinalis) Diterpenes for Alzheimer's Disease The Role of Oxidative Stress in Neurodegenerative Diseases The Role of Oxidative Stress-Induced Epigenetic Alterations in Amyloid-β Production in Alzheimer's Disease A genetic cause of Alzheimer disease: mechanistic insights from Down syndrome Copper-zinc superoxide dismutase-deficient mice show increased susceptibility to experimental autoimmune encephalomyelitis induced with myelin oligodendrocyte glycoprotein 35-55 Paradoxical Roles of Antioxidant Enzymes: Basic Mechanisms and Health Implications Liuwei Dihuang (LWDH), a traditional Chinese medicinal formula, protects against β-amyloid toxicity in transgenic Caenorhabditis elegans Presenilin promotes dietary copper uptake Site-specific inhibitory mechanism for amyloid β42 aggregation by catechol-type flavonoids targeting the Lys residues.Methylene blue upregulates Nrf2/ARE genes and prevents tau-related neurotoxicity A comparison of neuroinflammation to implanted microelectrodes in rat and mouse models.Senescence marker protein-30/superoxide dismutase 1 double knockout mice exhibit increased oxidative stress and hepatic steatosis.Cerium oxide nanoparticles protect against Aβ-induced mitochondrial fragmentation and neuronal cell death.Superoxide dismutase 1 loss disturbs intracellular redox signaling, resulting in global age-related pathological changes.Palladium and platinum nanoparticles attenuate aging-like skin atrophy via antioxidant activity in mice Accelerating neuronal aging in in vitro model brain disorders: a focus on reactive oxygen species Sod1 deficiency reduces incubation time in mouse models of prion disease Systematic review of the relationship between amyloid-β levels and measures of transgenic mouse cognitive deficit in Alzheimer's disease.Oxidative Stress during the Progression of β-Amyloid Pathology in the Neocortex of the Tg2576 Mouse Model of Alzheimer's Disease.The Ambiguous Relationship of Oxidative Stress, Tau Hyperphosphorylation, and Autophagy Dysfunction in Alzheimer's Disease.New insights in the amyloid-Beta interaction with mitochondria Increased pro-inflammatory cytokines, glial activation and oxidative stress in the hippocampus after short-term bilateral adrenalectomy Toxicity in rat primary neurons through the cellular oxidative stress induced by the turn formation at positions 22 and 23 of Aβ42.Shared Molecular Mechanisms in Alzheimer's Disease and Amyotrophic Lateral Sclerosis: Neurofilament-Dependent Transport of sAPP, FUS, TDP-43 and SOD1, with Endoplasmic Reticulum-Like Tubules.Redox proteomics analysis of HNE-modified proteins in Down syndrome brain: clues for understanding the development of Alzheimer disease.Age-dependent roles of peroxisomes in the hippocampus of a transgenic mouse model of Alzheimer's disease.Sod1 loss induces intrinsic superoxide accumulation leading to p53-mediated growth arrest and apoptosis Oxidative stress and the pathogenesis of Alzheimer's disease.ACE inhibition with captopril retards the development of signs of neurodegeneration in an animal model of Alzheimer's disease SOD1 Lysine 123 Acetylation in the Adult Central Nervous System.Antioxidant gene therapy against neuronal cell death.Increased acetylation of Peroxiredoxin1 by HDAC6 inhibition leads to recovery of Aβ-induced impaired axonal transport.Oxidative stress in Alzheimer's disease: Primary villain or physiological by-product?Synthetic nucleic acids delivered by exosomes: a potential therapeutic for generelated metabolic brain diseases.Biological metals and metal-targeting compounds in major neurodegenerative diseases.Conformation-specific antibodies to target amyloid β oligomers and their application to immunotherapy for Alzheimer's disease.The Essential Role of Soluble Aβ Oligomers in Alzheimer's Disease.Redox Signaling in Neurotransmission and Cognition During Aging.Neurotropin® alleviates hippocampal neuron damage through a HIF-1α/MAPK pathway.Topical Application of Trisodium Ascorbyl 6-Palmitate 2-Phosphate Actively Supplies Ascorbate to Skin Cells in an Ascorbate Transporter-Independent Manner.

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SOD1 (copper/zinc superoxide dismutase) deficiency drives amyloid β protein oligomerization and memory loss in mouse model of Alzheimer disease

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JBC.M111.279208

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Journal of Biological Chemistry

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SOD1 (copper/zinc superoxide d ...... use model of Alzheimer disease

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Hiroyuki Hatsuta

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Kazuma Murakami

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Kevin J Barnham

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Nakaba Murata

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Noriaki Kinoshita

http://www.w3.org/2001/XMLSchema#string

Shigeo Murayama

http://www.w3.org/2001/XMLSchema#string

Shoichi Tahara

http://www.w3.org/2001/XMLSchema#string

Takahiko Shimizu

http://www.w3.org/2001/XMLSchema#string

Takao Kaneko

http://www.w3.org/2001/XMLSchema#string

Takuji Shirasawa

http://www.w3.org/2001/XMLSchema#string

P2860

Correlative Memory Deficits, Abeta Elevation, and Amyloid Plaques in Transgenic Mice

The potential role of amyloid beta in the pathogenesis of age-related macular degeneration.

Amyloid plaque core protein in Alzheimer disease and Down syndrome

Amyloid-beta protein dimers isolated directly from Alzheimer's brains impair synaptic plasticity and memory

Soluble protein oligomers in neurodegeneration: lessons from the Alzheimer's amyloid beta-peptide

Naturally secreted oligomers of amyloid beta protein potently inhibit hippocampal long-term potentiation in vivo

Deficiency of the copper chaperone for superoxide dismutase increases amyloid-β production

Apomorphine treatment in Alzheimer mice promoting amyloid-β degradation

Elevated oxidative stress in erythrocytes due to a SOD1 deficiency causes anaemia and triggers autoantibody production

Drusen, choroidal neovascularization, and retinal pigment epithelium dysfunction in SOD1-deficient mice: a model of age-related macular degeneration

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44557-44568

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10.1074/JBC.M111.279208

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52

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286

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2011-11-09T00:00:00Z

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51786179

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22072713

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Alzheimer's disease

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3247976

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