GLUT4 is retained by an intracellular cycle of vesicle formation and fusion with endosomes
about
The clathrin heavy chain isoform CHC22 functions in a novel endosomal sorting stepGapex-5, a Rab31 guanine nucleotide exchange factor that regulates Glut4 trafficking in adipocytes.Insulin increases cell surface GLUT4 levels by dose dependently discharging GLUT4 into a cell surface recycling pathwayThe glucose transporter 4-regulating protein TUG is essential for highly insulin-responsive glucose uptake in 3T3-L1 adipocytesPIKfyve: Partners, significance, debates and paradoxesInsulin regulates Glut4 confinement in plasma membrane clusters in adipose cellsSites of glucose transporter-4 vesicle fusion with the plasma membrane correlate spatially with microtubulesRole of EHD1 and EHBP1 in perinuclear sorting and insulin-regulated GLUT4 recycling in 3T3-L1 adipocytesInsulin responsiveness of glucose transporter 4 in 3T3-L1 cells depends on the presence of sortilinRab8A and Rab13 are activated by insulin and regulate GLUT4 translocation in muscle cellsMapping of R-SNARE function at distinct intracellular GLUT4 trafficking steps in adipocytesDisruption of Adipose Rab10-Dependent Insulin Signaling Causes Hepatic Insulin Resistance.Insulin-regulated aminopeptidase is a key regulator of GLUT4 trafficking by controlling the sorting of GLUT4 from endosomes to specialized insulin-regulated vesiclesGLUT4 is sorted to vesicles whose accumulation beneath and insertion into the plasma membrane are differentially regulated by insulin and selectively affected by insulin resistanceSelf-assembly of Glut4 storage vesicles during differentiation of 3T3-L1 adipocytesIdentification of amino acid residues within the C terminus of the Glut4 glucose transporter that are essential for insulin-stimulated redistribution to the plasma membrane.Insulin signaling diverges into Akt-dependent and -independent signals to regulate the recruitment/docking and the fusion of GLUT4 vesicles to the plasma membraneInsulin stimulates membrane fusion and GLUT4 accumulation in clathrin coats on adipocyte plasma membranes.Unconventional myosin Myo1c promotes membrane fusion in a regulated exocytic pathway.Dynamic tracking and mobility analysis of single GLUT4 storage vesicle in live 3T3-L1 cells.Kinetic evidence for unique regulation of GLUT4 trafficking by insulin and AMP-activated protein kinase activators in L6 myotubesThe GLUT4 codeA novel choline cotransporter sequestration compartment in cholinergic neurons revealed by selective endosomal ablation.High basal cell surface levels of fish GLUT4 are related to reduced sensitivity of insulin-induced translocation toward GGA and AS160 inhibition in adipocytes.Insulin-regulated Glut4 translocation: membrane protein trafficking with six distinctive steps.Reciprocal regulation of endocytosis and metabolismThe C-terminus of GLUT4 targets the transporter to the perinuclear compartment but not to the insulin-responsive vesiclesIdentification of three distinct functional sites of insulin-mediated GLUT4 trafficking in adipocytes using quantitative single molecule imaging.A naturally occurring GIP receptor variant undergoes enhanced agonist-induced desensitization, which impairs GIP control of adipose insulin sensitivityGLUT4 traffic through an ESCRT-III-dependent sorting compartment in adipocytes.Glucose uptake in rat extraocular muscles: effect of insulin and contractile activity.Kinetic evidence that Glut4 follows different endocytic pathways than the receptors for transferrin and alpha2-macroglobulin.Single point mutations result in the miss-sorting of Glut4 to a novel membrane compartment associated with stress granule proteins.Hyperinsulinemia leads to uncoupled insulin regulation of the GLUT4 glucose transporter and the FoxO1 transcription factorReversible click reactions with boronic acids to build supramolecular architectures in water.Golgin-160 is required for the Golgi membrane sorting of the insulin-responsive glucose transporter GLUT4 in adipocytes.Gastric inhibitory peptide controls adipose insulin sensitivity via activation of cAMP-response element-binding protein and p110β isoform of phosphatidylinositol 3-kinaseGLUT4 exocytosis.The first luminal loop confers insulin responsiveness to glucose transporter 4.Development of a new model system to dissect isoform specific Akt signalling in adipocytes.
P2860
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P2860
GLUT4 is retained by an intracellular cycle of vesicle formation and fusion with endosomes
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
2003年论文
@zh
2003年论文
@zh-cn
name
GLUT4 is retained by an intrac ...... tion and fusion with endosomes
@ast
GLUT4 is retained by an intrac ...... tion and fusion with endosomes
@en
type
label
GLUT4 is retained by an intrac ...... tion and fusion with endosomes
@ast
GLUT4 is retained by an intrac ...... tion and fusion with endosomes
@en
prefLabel
GLUT4 is retained by an intrac ...... tion and fusion with endosomes
@ast
GLUT4 is retained by an intrac ...... tion and fusion with endosomes
@en
P2093
P2860
P356
P1476
GLUT4 is retained by an intrac ...... tion and fusion with endosomes
@en
P2093
Alona Cohen
Ola Karylowski
Timothy E McGraw
P2860
P304
P356
10.1091/MBC.E03-07-0517
P577
2003-10-31T00:00:00Z