Plac8-dependent and inducible NO synthase-dependent mechanisms clear Chlamydia muridarum infections from the genital tract - Test2 - elhamkhani - TriplyDB

Plac8-dependent and inducible NO synthase-dependent mechanisms clear Chlamydia muridarum infections from the genital tract

Plac8-dependent and inducible NO synthase-dependent mechanisms clear Chlamydia muridarum infections from the genital tract

http://www.wikidata.org/entity/Q35829883

about

Tissue-Resident T Cells as the Central Paradigm of Chlamydia Immunity A Molecular Host Response Assay to Discriminate Between Sepsis and Infection-Negative Systemic Inflammation in Critically Ill Patients: Discovery and Validation in Independent Cohorts The effect of infectious dose on humoral and cellular immune responses in Chlamydophila caviae primary ocular infection Chlamydia trachomatis control requires a vaccine.Evaluation of a multisubunit recombinant polymorphic membrane protein and major outer membrane protein T cell vaccine against Chlamydia muridarum genital infection in three strains of mice.Perforin is detrimental to controlling [corrected] C. muridarum replication in vitro, but not in vivo.Lack of a Functioning P2X7 Receptor Leads to Increased Susceptibility to Toxoplasmic Ileitis.Identification of stage-specific genes associated with lupus nephritis and response to remission induction in (NZB × NZW)F1 and NZM2410 mice.Modeling the transcriptome of genital tract epithelial cells and macrophages in healthy mucosa versus mucosa inflamed by Chlamydia muridarum infection.IFNγ is Required for Optimal Antibody-Mediated Immunity against Genital Chlamydia Infection.Borrelia burgdorferi elongation factor EF-Tu is an immunogenic protein during Lyme borreliosis.Chlamydial Type III Secretion System Needle Protein Induces Protective Immunity against Chlamydia muridarum Intravaginal Infection.Reply to Vicetti Miguel et al., "Setting Sights on Chlamydia Immunity's Central Paradigm: Can We Hit a Moving Target?"Subunit vaccines for the prevention of mucosal infection with Chlamydia trachomatis.An atypical CD8 T-cell response to Chlamydia muridarum genital tract infections includes T cells that produce interleukin-13.Differentially expressed genes in PPARγ-deficient MSCs.Overexpression of placenta specific 8 is associated with malignant progression and poor prognosis of clear cell renal cell carcinoma.A systems approach to renal inflammation in SLE.The many ways tissue phagocytes respond to dying cells.A Chlamydia-Specific TCR-Transgenic Mouse Demonstrates Th1 Polyfunctionality with Enhanced Effector Function.Beyond an AFLP genome scan towards the identification of immune genes involved in plague resistance in Rattus rattus from Madagascar.Peptide array-based screening reveals a large number of proteins interacting with the ankyrin-repeat domain of the zDHHC17 S-acyltransferase.Perspective: my 37 year journey through Chlamydia research: Chlamydia antigen analysis using monoclonal antibodies and major histocompatibility complex molecules.B cell presentation of Chlamydia antigen selects out protective CD4γ13 T cells; implications for genital tract tissue resident memory lymphocyte clusters.Toll-like receptor 3 (TLR3) promotes the resolution of Chlamydia muridarum genital tract infection in congenic C57BL/6N mice.The novel KLF4/PLAC8 signaling pathway regulates lung cancer growth.

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Plac8-dependent and inducible NO synthase-dependent mechanisms clear Chlamydia muridarum infections from the genital tract

http://www.wikidata.org/entity/Q35829883

description

2012 nî lūn-bûn

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2012年论文

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2012年论文

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2012年论文

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Plac8-dependent and inducible ...... ections from the genital tract

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Plac8-dependent and inducible ...... ections from the genital tract

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Plac8-dependent and inducible ...... ections from the genital tract

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JIMMUNOL.1102764

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Journal of Immunology

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Plac8-dependent and inducible ...... ections from the genital tract

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James E Slaven

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Micah S Kerr

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Raymond M Johnson

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P2860

IFN-gamma-inducible Irga6 mediates host resistance against Chlamydia trachomatis via autophagy

Impaired host defense in mice lacking ONZIN

The negative c-Myc target onzin affects proliferation and apoptosis via its obligate interaction with phospholipid scramblase 1

Nitric oxide and macrophage function

Inducible nitric oxide synthase does not affect resolution of murine chlamydial genital tract infections or eradication of chlamydiae in primary murine cell culture.

Chlamydial infection in inducible nitric oxide synthase knockout mice.

Chlamydia muridarum T-cell antigens formulated with the adjuvant DDA/TDB induce immunity against infection that correlates with a high frequency of gamma interferon (IFN-gamma)/tumor necrosis factor alpha and IFN-gamma/interleukin-17 double-positive

Chlamydial IFN-gamma immune evasion is linked to host infection tropism.

Clearance of Chlamydia trachomatis from the murine genital mucosa does not require perforin-mediated cytolysis or Fas-mediated apoptosis.

Resolution of secondary Chlamydia trachomatis genital tract infection in immune mice with depletion of both CD4+ and CD8+ T cells.

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1896-1904

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10.4049/JIMMUNOL.1102764

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4

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2012-01-11T00:00:00Z

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221933804

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22238459

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3303601

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