DNA base excision repair: a mechanism of trinucleotide repeat expansion.
about
DNA Polymerases λ and β: The Double-Edged Swords of DNA RepairInstability of CTG repeats is governed by the position of a DNA base lesion through base excision repairEmergence and evolution of Zfp36l3Microsatellite-encoded domain in rodent Sry functions as a genetic capacitor to enable the rapid evolution of biological noveltyModulation of trinucleotide repeat instability by DNA polymerase β polymorphic variant R137Q.Schizosaccharomyces pombe MutSα and MutLα Maintain Stability of Tetra-Nucleotide Repeats and Msh3 of Hepta-Nucleotide Repeats.Base excision repair: a critical player in many gamesTrinucleotide expansion in disease: why is there a length threshold?A 5', 8-cyclo-2'-deoxypurine lesion induces trinucleotide repeat deletion via a unique lesion bypass by DNA polymerase βInvolvement of DNA polymerase β overexpression in the malignant transformation induced by benzo[a]pyrene.Recognition and repair of chemically heterogeneous structures at DNA endsNovel method for site-specific induction of oxidative DNA damage reveals differences in recruitment of repair proteins to heterochromatin and euchromatinBase excision repair of chemotherapeutically-induced alkylated DNA damage predominantly causes contractions of expanded GAA repeats associated with Friedreich's ataxia.Repeat instability during DNA repair: Insights from model systems14-3-3σ confers cisplatin resistance in esophageal squamous cell carcinoma cells via regulating DNA repair molecules.AP endonuclease 1 prevents trinucleotide repeat expansion via a novel mechanism during base excision repair.The Repeat Expansion Diseases: The dark side of DNA repair.Bypass of a 5',8-cyclopurine-2'-deoxynucleoside by DNA polymerase β during DNA replication and base excision repair leads to nucleotide misinsertions and DNA strand breaks.Rate-determining Step of Flap Endonuclease 1 (FEN1) Reflects a Kinetic Bias against Long Flaps and Trinucleotide Repeat Sequences.Is There an Association of Physical Activity with Brain Volume, Behavior, and Day-to-day Functioning? A Cross Sectional Design in Prodromal and Early Huntington Disease.Msh2-Msh3 interferes with Okazaki fragment processing to promote trinucleotide repeat expansions.Polyglutamine (polyQ) disorders: the chromatin connection.Evidence for abasic site sugar phosphate-mediated cytotoxicity in alkylating agent treated Saccharomyces cerevisiaeDeregulated expression of DNA polymerase β is involved in the progression of genomic instabilityBase excision repair in physiology and pathology of the central nervous systemTrinucleotide repeat deletion via a unique hairpin bypass by DNA polymerase β and alternate flap cleavage by flap endonuclease 1.CAG/CTG repeats alter the affinity for the histone core and the positioning of DNA in the nucleosome.Parkinson's disease: a complex interplay of mitochondrial DNA alterations and oxidative stress.Flap endonuclease 1.Oxidized dNTPs and the OGG1 and MUTYH DNA glycosylases combine to induce CAG/CTG repeat instability.Bidirectional transcription of trinucleotide repeats: roles for excision repair.Inhibition of DNA synthesis facilitates expansion of low-complexity repeats: is strand slippage stimulated by transient local depletion of specific dNTPs?Oxidative damage and mutagenesis in Saccharomyces cerevisiae: genetic studies of pathways affecting replication fidelity of 8-oxoguanineCrosstalk between MSH2-MSH3 and polβ promotes trinucleotide repeat expansion during base excision repair.Dynamics of enzymatic interactions during short flap human Okazaki fragment processing by two forms of human DNA polymerase δ.Abnormal base excision repair at trinucleotide repeats associated with diseases: a tissue-selective mechanism.Proliferating cell nuclear antigen prevents trinucleotide repeat expansions by promoting repeat deletion and hairpin removal.Base excision repair of oxidative DNA damage coupled with removal of a CAG repeat hairpin attenuates trinucleotide repeat expansion.AGG interruptions within the maternal FMR1 gene reduce the risk of offspring with fragile X syndrome.Integration of DNA damage and repair with murine double-minute 2 (Mdm2) in tumorigenesis.
P2860
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P2860
DNA base excision repair: a mechanism of trinucleotide repeat expansion.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
DNA base excision repair: a mechanism of trinucleotide repeat expansion.
@ast
DNA base excision repair: a mechanism of trinucleotide repeat expansion.
@en
type
label
DNA base excision repair: a mechanism of trinucleotide repeat expansion.
@ast
DNA base excision repair: a mechanism of trinucleotide repeat expansion.
@en
prefLabel
DNA base excision repair: a mechanism of trinucleotide repeat expansion.
@ast
DNA base excision repair: a mechanism of trinucleotide repeat expansion.
@en
P2860
P1476
DNA base excision repair: a mechanism of trinucleotide repeat expansion
@en
P2093
P2860
P304
P356
10.1016/J.TIBS.2011.12.002
P577
2012-01-27T00:00:00Z