Nerve injury-induced calcium channel alpha-2-delta-1 protein dysregulation leads to increased pre-synaptic excitatory input into deep dorsal horn neurons and neuropathic allodynia
about
Current Status of the New Antiepileptic Drugs in Chronic PainCentral Mechanisms Mediating Thrombospondin-4-induced Pain States.Injury-induced maladaptation and dysregulation of calcium channel α2 δ subunit proteins and its contribution to neuropathic pain development.The Anti-Allodynic Gabapentinoids: Myths, Paradoxes, and Acute Effects.Thrombospondin-4 acts on pre-synaptic calcium channel alpha-2-delta-1 subunit to induce synaptogenesis between sensory and spinal cord neurons through a T-type calcium channel dependent pathway that is blocked by early, low-dose gabapentin treatmentChronic pregabalin treatment decreases excitability of dentate gyrus and accelerates maturation of adult-born granule cells.Peripheral nerve injury increases contribution of L-type calcium channels to synaptic transmission in spinal lamina II: Role of α2δ-1 subunits.
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Nerve injury-induced calcium channel alpha-2-delta-1 protein dysregulation leads to increased pre-synaptic excitatory input into deep dorsal horn neurons and neuropathic allodynia
description
2015 nî lūn-bûn
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2015年の論文
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2015年論文
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2015年論文
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2015年論文
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2015年論文
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2015年論文
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2015年论文
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2015年论文
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2015年论文
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name
Nerve injury-induced calcium c ...... rons and neuropathic allodynia
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Nerve injury-induced calcium c ...... rons and neuropathic allodynia
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type
label
Nerve injury-induced calcium c ...... rons and neuropathic allodynia
@ast
Nerve injury-induced calcium c ...... rons and neuropathic allodynia
@en
prefLabel
Nerve injury-induced calcium c ...... rons and neuropathic allodynia
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Nerve injury-induced calcium c ...... rons and neuropathic allodynia
@en
P2860
P356
P1476
Nerve injury-induced calcium c ...... rons and neuropathic allodynia
@en
P2860
P304
P356
10.1002/EJP.656
P577
2015-02-17T00:00:00Z