Induction of cellular senescence by insulin-like growth factor binding protein-5 through a p53-dependent mechanism. - Test2 - elhamkhani - TriplyDB

Induction of cellular senescence by insulin-like growth factor binding protein-5 through a p53-dependent mechanism.

Induction of cellular senescence by insulin-like growth factor binding protein-5 through a p53-dependent mechanism.

http://www.wikidata.org/entity/Q36095787

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The Ccr4a (CNOT6) and Ccr4b (CNOT6L) deadenylase subunits of the human Ccr4-Not complex contribute to the prevention of cell death and senescence Cellular senescence controls fibrosis in wound healing Functional roles and clinical values of insulin-like growth factor-binding protein-5 in different types of cancers Redox control of senescence and age-related disease Insulin-like growth factor-binding protein-5-induced laminin gamma1 transcription requires filamin A Interruption of progerin-lamin A/C binding ameliorates Hutchinson-Gilford progeria syndrome phenotype Suppression of the DHX9 helicase induces premature senescence in human diploid fibroblasts in a p53-dependent manner.The subcellular localization of IGFBP5 affects its cell growth and migration functions in breast cancer.A mutant allele of the Swi/Snf member BAF250a determines the pool size of fetal liver hemopoietic stem cell populations Critical role of insulin‑like growth factor binding protein‑5 in methamphetamine‑induced apoptosis in cardiomyocytes.MicroRNA-mediated regulation of extracellular matrix formation modulates somatic cell reprogramming Important miRs of pathways in different tumor types.Bilateral administration of autologous CD133+ cells in ambulatory patients with refractory critical limb ischemia: lessons learned from a pilot randomized, double-blind, placebo-controlled trial.Functional role of cellular senescence in biliary injury Amniotic fluid RNA gene expression profiling provides insights into the phenotype of Turner syndrome.Insulin-like growth factor binding protein-6 delays replicative senescence of human fibroblasts.Regulation of cellular senescence by the essential caveolar component PTRF/Cavin-1.Defining the disulfide bonds of insulin-like growth factor-binding protein-5 by tandem mass spectrometry with electron transfer dissociation and collision-induced dissociation.Cellular Senescence in Type 2 Diabetes: A Therapeutic Opportunity.Conserved Senescence Associated Genes and Pathways in Primary Human Fibroblasts Detected by RNA-Seq.Alteration of Aging-Dependent MicroRNAs in Idiopathic Pulmonary Fibrosis.LDL biochemical modifications: a link between atherosclerosis and aging.Identification of ageing-associated naturally occurring peptides in human urine Senescence sensitivity of breast cancer cells is defined by positive feedback loop between CIP2A and E2F1.Age-related changes in miR-143-3p:Igfbp5 interactions affect muscle regeneration.Insulin Like Growth Factor Binding Protein-5 Regulates Excessive Vascular Smooth Muscle Cell Proliferation in Spontaneously Hypertensive Rats via ERK 1/2 Phosphorylation Induction of cellular senescence by secretory phospholipase A2 in human dermal fibroblasts through an ROS-mediated p53 pathway.Conserved genes and pathways in primary human fibroblast strains undergoing replicative and radiation induced senescence.Critical pathways in cellular senescence and immortalization revealed by gene expression profiling.IL-6-STAT3 signaling and premature senescence.Alterations in microRNA expression in stress-induced cellular senescence.Cellular senescence: unravelling complexity.Insulin-like growth factor binding proteins-3 and -5: central mediators of fibrosis and promising new therapeutic targets.Involvement of IGF binding protein 5 in prostaglandin E(2)-induced cellular senescence in human fibroblasts.Cellular senescence: a hitchhiker's guide.Talin-1 interaction network promotes hepatocellular carcinoma progression.Distinct Receptor Tyrosine Kinase Subsets Mediate Anti-HER2 Drug Resistance in Breast Cancer.Meeting Report: International Symposium on the Genetics of Aging and Life History II.Mitotic centromere-associated kinase (MCAK/Kif2C) regulates cellular senescence in human primary cells through a p53-dependent pathway.IGFBP-rP1 induces p21 expression through a p53-independent pathway, leading to cellular senescence of MCF-7 breast cancer cells.

P2860

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P2860

Induction of cellular senescence by insulin-like growth factor binding protein-5 through a p53-dependent mechanism.

http://www.wikidata.org/entity/Q36095787

description

2007 nî lūn-bûn

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2007年の論文

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name

Induction of cellular senescen ...... ugh a p53-dependent mechanism.

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Induction of cellular senescen ...... ugh a p53-dependent mechanism.

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Induction of cellular senescen ...... ugh a p53-dependent mechanism.

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Induction of cellular senescen ...... ugh a p53-dependent mechanism.

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Induction of cellular senescen ...... ugh a p53-dependent mechanism.

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Induction of cellular senescen ...... ugh a p53-dependent mechanism.

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Molecular Biology of the Cell

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Induction of cellular senescen ...... ugh a p53-dependent mechanism.

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Jae-Ryong Kim

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Jung Hye Kim

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Keuk Jun Kim

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Kwang Seok Kim

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Mi Jin Kim

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Suk-Hwan Baek

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Young Bae Seu

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P2860

Oncogenic ras provokes premature cell senescence associated with accumulation of p53 and p16INK4a

ING2 regulates the onset of replicative senescence by induction of p300-dependent p53 acetylation

DNA damage signaling and p53-dependent senescence after prolonged beta-interferon stimulation

A biomarker that identifies senescent human cells in culture and in aging skin in vivo

Insulin-like growth factor (IGF)-binding protein-3 (IGFBP-3) binds to fibronectin (FN): demonstration of IGF-I/IGFBP-3/fn ternary complexes in human plasma

IGF-binding protein-5: flexible player in the IGF system and effector on its own

Cellular actions of the insulin-like growth factor binding proteins

Insulin-like growth factor binding protein-5 binds to plasminogen activator inhibitor-I

Characterization of the chromosomal gene and promoter for human insulin-like growth factor binding protein-5

The human insulin-like growth factor (IGF) binding protein-3 inhibits the growth of fibroblasts with a targeted disruption of the IGF-I receptor gene

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