DCIR2+ cDC2 DCs and Zbtb32 Restore CD4+ T-Cell Tolerance and Inhibit Diabetes - Test2 - elhamkhani - TriplyDB

DCIR2+ cDC2 DCs and Zbtb32 Restore CD4+ T-Cell Tolerance and Inhibit Diabetes

DCIR2+ cDC2 DCs and Zbtb32 Restore CD4+ T-Cell Tolerance and Inhibit Diabetes

http://www.wikidata.org/entity/Q36104619

about

Dendritic cell-activated cytokine-induced killer cell-mediated immunotherapy is safe and effective for cancer patients >65ï¿½years old The Role of Dendritic Cell Subsets and Innate Immunity in the Pathogenesis of Type 1 Diabetes and Other Autoimmune Diseases.Integrated transcriptomic and proteomic analysis of the molecular cargo of extracellular vesicles derived from porcine adipose tissue-derived mesenchymal stem cells Type 1 diabetes genetic susceptibility and dendritic cell function: potential targets for treatment.Flow cytometric gating for spleen monocyte and DC subsets: differences in autoimmune NOD mice and with acute inflammation Advancing drug delivery systems for the treatment of multiple sclerosis.The Importance of Dendritic Cells in Maintaining Immune Tolerance.Transient expression of ZBTB32 in anti-viral CD8+ T cells limits the magnitude of the effector response and the generation of memory.Profiling MHC II immunopeptidome of blood-stage malaria reveals that cDC1 control the functionality of parasite-specific CD4 T cells.Low CD25 on autoreactive Tregs impairs tolerance via low dose IL-2 and antigen delivery.Erratum to: Advancing drug delivery systems for the treatment of multiple sclerosis.Loss of Zbtb32 in NOD mice does not significantly alter T cell responses.

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DCIR2+ cDC2 DCs and Zbtb32 Restore CD4+ T-Cell Tolerance and Inhibit Diabetes

http://www.wikidata.org/entity/Q36104619

description

2015 nî lūn-bûn

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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2015年论文

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2015年论文

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name

DCIR2+ cDC2 DCs and Zbtb32 Restore CD4+ T-Cell Tolerance and Inhibit Diabetes

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DCIR2+ cDC2 DCs and Zbtb32 Restore CD4+ T-Cell Tolerance and Inhibit Diabetes

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type

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DCIR2+ cDC2 DCs and Zbtb32 Restore CD4+ T-Cell Tolerance and Inhibit Diabetes

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DCIR2+ cDC2 DCs and Zbtb32 Restore CD4+ T-Cell Tolerance and Inhibit Diabetes

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DCIR2+ cDC2 DCs and Zbtb32 Restore CD4+ T-Cell Tolerance and Inhibit Diabetes

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DCIR2+ cDC2 DCs and Zbtb32 Restore CD4+ T-Cell Tolerance and Inhibit Diabetes

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DCIR2+ cDC2 DCs and Zbtb32 Restore CD4+ T-Cell Tolerance and Inhibit Diabetes

@en

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Chie Hotta-Iwamura

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Jeffrey D Price

http://www.w3.org/2001/XMLSchema#string

Nicole M Beauchamp

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Yongge Zhao

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P2860

Chromogranin A is an autoantigen in type 1 diabetes

Central role of defective interleukin-2 production in the triggering of islet autoimmune destruction

Immune tolerance after delivery of dying cells to dendritic cells in situ

Dendritic cell subsets in type 1 diabetes: friend or foe?

Batf3 Deficiency Reveals a Critical Role for CD8 + Dendritic Cells in Cytotoxic T Cell Immunity

Inhibitory Fc receptors modulate in vivo cytotoxicity against tumor targets

Genetics, pathogenesis and clinical interventions in type 1 diabetes

Extrathymic Aire-expressing cells are a distinct bone marrow-derived population that induce functional inactivation of CD4⁺ T cells

Differential antigen processing by dendritic cell subsets in vivo

Dendritic cells induce peripheral T cell unresponsiveness under steady state conditions in vivo

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3521-3531

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scholarly article

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10.2337/DB14-1880

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10

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64

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Kristin V Tarbell

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2015-06-12T00:00:00Z

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278159968

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26070317

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4587633

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