MET is required for the recruitment of anti-tumoural neutrophils - Test2 - elhamkhani - TriplyDB

MET is required for the recruitment of anti-tumoural neutrophils

MET is required for the recruitment of anti-tumoural neutrophils

http://www.wikidata.org/entity/Q36125652

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Trial Watch-Immunostimulation with cytokines in cancer therapy TNF is a key cytokine mediating neutrophil cytotoxic activity in breast cancer patients.Constitutive activation of MET signaling impairs myogenic differentiation of rhabdomyosarcoma and promotes its development and progression.Tumor-induced inflammation alters neutrophil phenotype and disease progression Neutrophils support lung colonization of metastasis-initiating breast cancer cells Neutrophil Granulocytes in Ovarian Cancer - Induction of Epithelial-To-Mesenchymal-Transition and Tumor Cell Migration.Cytokine Profiles during Invasive Nontyphoidal Salmonella Disease Predict Outcome in African Children.Neutrophils: Critical components in experimental animal models of cancer.Reprogramming the immunological microenvironment through radiation and targeting Axl.Tumor microenvironment: driving forces and potential therapeutic targets for breast cancer metastasis Pharmacologic or Genetic Targeting of Glutamine Synthetase Skews Macrophages toward an M1-like Phenotype and Inhibits Tumor Metastasis.Prolyl hydroxylase 2 inactivation enhances glycogen storage and promotes excessive neutrophilic responses.MET-targeted therapy for gastric cancer: the importance of a biomarker-based strategy.Neutrophils in the Tumor Microenvironment.Regulation of the MET oncogene: molecular mechanisms.Molecular Pathways: Oncologic Pathways and Their Role in T-cell Exclusion and Immune Evasion-A New Role for the AXL Receptor Tyrosine Kinase.Preclinical and clinical evaluation of MET functions in cancer cells and in the tumor stroma.Targeting c-MET in gastrointestinal tumours: rationale, opportunities and challenges.Neutrophils in cancer: neutral no more.The role of myeloid cells in cancer therapies.Cabozantinib Eradicates Advanced Murine Prostate Cancer by Activating Antitumor Innate Immunity.Neutrophils in cancer.The Diverse Biological Functions of Neutrophils, Beyond the Defense Against Infections.One microenvironment does not fit all: heterogeneity beyond cancer cells.Recent advances in understanding neutrophils.Epithelial Hypoxia-Inducible Factor 2α Facilitates the Progression of Colon Tumors through Recruiting Neutrophils.Peritumoral stromal neutrophils are essential for c-Met-elicited metastasis in human hepatocellular carcinoma.Mouse versus Human Neutrophils in Cancer: A Major Knowledge Gap.Pathogen response-like recruitment and activation of neutrophils by sterile immunogenic dying cells drives neutrophil-mediated residual cell killing.Occurrence and significance of tumor-associated neutrophils in patients with colorectal cancer.Neutrophils in cancer: prognostic role and therapeutic strategies.Hypoxia-inducible factors: a central link between inflammation and cancer.Neutrophils fan cancer's flames.Ly6Clo monocytes drive immunosuppression and confer resistance to anti-VEGFR2 cancer therapy.Loss of HIF-1α in natural killer cells inhibits tumour growth by stimulating non-productive angiogenesis.Double-stranded RNA promotes CTL-independent tumor cytolysis mediated by CD11b+Ly6G+ intratumor myeloid cells through the TICAM-1 signaling pathway.Inhibition of KPNA4 attenuates prostate cancer metastasis.Role of LFA-1 and ICAM-1 in Cancer.Biological roles of hepatocyte growth factor-Met signaling from genetically modified animals.CD177+ neutrophils suppress epithelial cell tumourigenesis in colitis-associated cancer and predict good prognosis in colorectal cancer.

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P2860

MET is required for the recruitment of anti-tumoural neutrophils

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2015年论文

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name

MET is required for the recruitment of anti-tumoural neutrophils

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MET is required for the recruitment of anti-tumoural neutrophils

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MET is required for the recruitment of anti-tumoural neutrophils

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MET is required for the recruitment of anti-tumoural neutrophils

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MET is required for the recruitment of anti-tumoural neutrophils

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MET is required for the recruitment of anti-tumoural neutrophils

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MET is required for the recruitment of anti-tumoural neutrophils

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Andrea Casazza

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Els Wauters

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Giusy Di Conza

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Jens Serneels

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Mario Di Matteo

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Veronica Finisguerra

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Zvi Granot

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P2860

Cloning and expression of two human genes encoding calcium-binding proteins that are regulated during myeloid differentiation

Phase II and biomarker study of the dual MET/VEGFR2 inhibitor foretinib in patients with papillary renal cell carcinoma

Hypoxia promotes invasive growth by transcriptional activation of the met protooncogene

A novel method for isolation of neutrophils from murine blood using negative immunomagnetic separation

Neutrophil-specific deletion of Syk kinase results in reduced host defense to bacterial infection

HIF-1alpha is essential for myeloid cell-mediated inflammation

Rapid chemotherapy-induced acute endothelial progenitor cell mobilization: implications for antiangiogenic drugs as chemosensitizing agents.

An inducible mouse model of colon carcinogenesis for the analysis of sporadic and inflammation-driven tumor progression.

Drug development of MET inhibitors: targeting oncogene addiction and expedience.

Deletion of Syk in neutrophils prevents immune complex arthritis

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10.1038/NATURE14407

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7556

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522

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Massimiliano Mazzone

Sarah R Walmsley

A A Roger Thompson

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2015-05-18T00:00:00Z

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1030668074

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