Helicobacter pylori requires TlpD-driven chemotaxis to proliferate in the antrum.
about
Metalloregulation of Helicobacter pylori physiology and pathogenesisChemodetection and Destruction of Host Urea Allows Helicobacter pylori to Locate the Epithelium.The host protein calprotectin modulates the Helicobacter pylori cag type IV secretion system via zinc sequestrationMotility and chemotaxis mediate the preferential colonization of gastric injury sites by Helicobacter pyloriAdhesion and Invasion of Gastric Mucosa Epithelial Cells by Helicobacter pyloriPathogenesis of Helicobacter pylori infection.Helicobacter pylori strains vary cell shape and flagellum number to maintain robust motility in viscous environmentsHelical and rod-shaped bacteria swim in helical trajectories with little additional propulsion from helical shape.Survival in hostile territory: the microbiota of the stomach.Multiple Acid Sensors Control Helicobacter pylori Colonization of the StomachLife in the human stomach: persistence strategies of the bacterial pathogen Helicobacter pylori.The Helicobacter pylori CZB Cytoplasmic Chemoreceptor TlpD Forms an Autonomous Polar Chemotaxis Signaling Complex That Mediates a Tactic Response to Oxidative Stress.Role of energy sensor TlpD of Helicobacter pylori in gerbil colonization and genome analyses after adaptation in the gerbil.Spatial and Temporal Shifts in Bacterial Biogeography and Gland Occupation during the Development of a Chronic Infection.Rapid identification of Helicobacter pylori and assessment of clarithromycin susceptibility from clinical specimens using FISH.Genetic determinants of swimming motility in the squid light-organ symbiont Vibrio fischeriInternal sense of direction: sensing and signaling from cytoplasmic chemoreceptors.How Helicobacter pylori senses, targets and interacts with the gastric epithelium.Recent Advances in Helicobacter pylori Replication: Possible Implications in Adaptation to a Pathogenic Lifestyle and Perspectives for Drug Design.Survival of Helicobacter pylori in gastric acidic territory.Role of Flagella in the Pathogenesis of Helicobacter pyloriLocalisation and protein-protein interactions of the Helicobacter pylori taxis sensor TlpD and their connection to metabolic functionsThe Norepinephrine Metabolite 3,4-Dihydroxymandelic Acid Is Produced by the Commensal Microbiota and Promotes Chemotaxis and Virulence Gene Expression in Enterohemorrhagic Escherichia coli.Colonization, localization, and inflammation: the roles of H. pylori chemotaxis in vivo.Sensory Repertoire of Bacterial Chemoreceptors.The effect of bacterial chemotaxis on host infection and pathogenicity.Chemotaxis allows bacteria to overcome host-generated reactive oxygen species that constrain gland colonization.
P2860
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P2860
Helicobacter pylori requires TlpD-driven chemotaxis to proliferate in the antrum.
description
2012 nî lūn-bûn
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2012年の論文
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年学术文章
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name
Helicobacter pylori requires TlpD-driven chemotaxis to proliferate in the antrum.
@ast
Helicobacter pylori requires TlpD-driven chemotaxis to proliferate in the antrum.
@en
type
label
Helicobacter pylori requires TlpD-driven chemotaxis to proliferate in the antrum.
@ast
Helicobacter pylori requires TlpD-driven chemotaxis to proliferate in the antrum.
@en
prefLabel
Helicobacter pylori requires TlpD-driven chemotaxis to proliferate in the antrum.
@ast
Helicobacter pylori requires TlpD-driven chemotaxis to proliferate in the antrum.
@en
P2093
P2860
P356
P1476
Helicobacter pylori requires TlpD-driven chemotaxis to proliferate in the antrum
@en
P2093
Annah S Rolig
J Elliot Carter
James Shanks
P2860
P304
P356
10.1128/IAI.00407-12
P407
P577
2012-07-16T00:00:00Z