Type I interferon sensitizes lymphocytes to apoptosis and reduces resistance to Listeria infection. - Test2 - elhamkhani - TriplyDB

Type I interferon sensitizes lymphocytes to apoptosis and reduces resistance to Listeria infection.

Type I interferon sensitizes lymphocytes to apoptosis and reduces resistance to Listeria infection.

http://www.wikidata.org/entity/Q36399581

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IFN-γ Priming Effects on the Maintenance of Effector Memory CD4(+) T Cells and on Phagocyte Function: Evidences from Infectious Diseases Immunomodulatory functions of type I interferons Differential effects of type I and II interferons on myeloid cells and resistance to intracellular bacterial infections Multifaceted activity of listeriolysin O, the cholesterol-dependent cytolysin of Listeria monocytogenes Irf3 polymorphism alters induction of interferon beta in response to Listeria monocytogenes infection Influenza vaccination accelerates recovery of ferrets from lymphopenia Peripheral Leukocyte Migration in Ferrets in Response to Infection with Seasonal Influenza Virus Opposing roles for interferon regulatory factor-3 (IRF-3) and type I interferon signaling during plague Conditional Stat1 ablation reveals the importance of interferon signaling for immunity to Listeria monocytogenes infection Disease-promoting effects of type I interferons in viral, bacterial, and coinfections Legionella pneumophila induces IFNbeta in lung epithelial cells via IPS-1 and IRF3, which also control bacterial replication Type I interferon: friend or foe?Twist mediates suppression of inflammation by type I IFNs and Axl Cell-Based Screen Identifies Human Interferon-Stimulated Regulators of Listeria monocytogenes Infection Interplay between CD8α+ dendritic cells and monocytes in response to Listeria monocytogenes infection attenuates T cell responses Innate and adaptive immunologic functions of complement in the host response to Listeria monocytogenes infection.Salmonella Suppresses the TRIF-Dependent Type I Interferon Response in Macrophages.Identification of host cytosolic sensors and bacterial factors regulating the type I interferon response to Legionella pneumophila Bacterial ligands generated in a phagosome are targets of the cytosolic innate immune system.Distinct TLR- and NLR-mediated transcriptional responses to an intracellular pathogen.Tentative T cells: memory cells are quick to respond, but slow to divide Forced IFIT-2 expression represses LPS induced TNF-alpha expression at posttranscriptional levels.Type I interferon is a therapeutic target for virus-induced lethal vascular damage.Characterization of the interferon-producing cell in mice infected with Listeria monocytogenes The bacterial pathogen Listeria monocytogenes and the interferon family: type I, type II and type III interferons Type I Interferon Induction by Neisseria gonorrhoeae: Dual Requirement of Cyclic GMP-AMP Synthase and Toll-like Receptor 4.Attenuated Listeria monocytogenes: a powerful and versatile vector for the future of tumor immunotherapy.Getting "Inside" Type I IFNs: Type I IFNs in Intracellular Bacterial Infections Induction of IFN-alphabeta enables Listeria monocytogenes to suppress macrophage activation by IFN-gamma.Type I IFN signaling in CD8- DCs impairs Th1-dependent malaria immunity.Influenza virus-induced glucocorticoids compromise innate host defense against a secondary bacterial infection.Listeria monocytogenes induces an interferon-enhanced activation of the integrated stress response that is detrimental for resolution of infection in mice STAT1β is not dominant negative and is capable of contributing to gamma interferon-dependent innate immunity.FAS-associated factor-1 positively regulates type I interferon response to RNA virus infection by targeting NLRX1.Down regulation of macrophage IFNGR1 exacerbates systemic L. monocytogenes infection The role of the activated macrophage in clearing Listeria monocytogenes infection A fluorescence reporter model defines "Tip-DCs" as the cellular source of interferon β in murine listeriosis Induction of type I and type III interferons by Borrelia burgdorferi correlates with pathogenesis and requires linear plasmid 36.Intranasal Poly-IC treatment exacerbates tuberculosis in mice through the pulmonary recruitment of a pathogen-permissive monocyte/macrophage population.Production of IFN-β during Listeria monocytogenes infection is restricted to monocyte/macrophage lineage

P2860

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P2860

Type I interferon sensitizes lymphocytes to apoptosis and reduces resistance to Listeria infection.

http://www.wikidata.org/entity/Q36399581

description

2004 nî lūn-bûn

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2004年の論文

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Type I interferon sensitizes l ...... istance to Listeria infection.

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Type I interferon sensitizes l ...... istance to Listeria infection.

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Journal of Experimental Medicine

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Type I interferon sensitizes l ...... sistance to Listeria infection

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P2093

Javier A Carrero

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P2860

Functional role of type I and type II interferons in antiviral defense

Multiple defects of immune cell function in mice with disrupted interferon-gamma genes

Macrophages that have ingested apoptotic cells in vitro inhibit proinflammatory cytokine production through autocrine/paracrine mechanisms involving TGF-beta, PGE2, and PAF

Integration of interferon-alpha/beta signalling to p53 responses in tumour suppression and antiviral defence.

Immunity to Listeria infection.

Innate recognition of bacteria by a macrophage cytosolic surveillance pathway

A blast from the past: clearance of apoptotic cells regulates immune responses.

Lymphocyte apoptosis during early phase of Listeria infection in mice

Two roads diverged: interferon alpha/beta- and interleukin 12-mediated pathways in promoting T cell interferon gamma responses during viral infection

Crucial role of interferon consensus sequence binding protein, but neither of interferon regulatory factor 1 nor of nitric oxide synthesis for protection against murine listeriosis

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535-540

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8408063

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15302900

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apoptotic process

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2211931

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