Role of the TGF-beta/Alk5 signaling pathway in monocrotaline-induced pulmonary hypertension. - Test2 - elhamkhani - TriplyDB

Role of the TGF-beta/Alk5 signaling pathway in monocrotaline-induced pulmonary hypertension.

Role of the TGF-beta/Alk5 signaling pathway in monocrotaline-induced pulmonary hypertension.

http://www.wikidata.org/entity/Q36539476

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Immune and inflammatory mechanisms in pulmonary arterial hypertension A comprehensive review: the evolution of animal models in pulmonary hypertension research; are we there yet?Longitudinal in vivo SPECT/CT imaging reveals morphological changes and cardiopulmonary apoptosis in a rodent model of pulmonary arterial hypertension Calpain mediates pulmonary vascular remodeling in rodent models of pulmonary hypertension, and its inhibition attenuates pathologic features of disease TGF beta and IL13 in Schistosomiasis mansoni associated pulmonary arterial hypertension; a descriptive study with comparative groups.TGF-β activation by bone marrow-derived thrombospondin-1 causes Schistosoma- and hypoxia-induced pulmonary hypertension.Heritable forms of pulmonary arterial hypertension.Schistosomiasis-induced experimental pulmonary hypertension: role of interleukin-13 signaling.Targeting the vasoprotective axis of the renin-angiotensin system: a novel strategic approach to pulmonary hypertensive therapy.Transforming growth factor-β inhibition attenuates pulmonary arterial hypertension in rats.Gene delivery of cytochrome p450 epoxygenase ameliorates monocrotaline-induced pulmonary artery hypertension in rats Schistosomiasis and the pulmonary vasculature (2013 Grover Conference series)Monocrotaline Induces Endothelial Injury and Pulmonary Hypertension by Targeting the Extracellular Calcium-Sensing Receptor.Transforming growth factor-β inhibition and endothelin receptor blockade in rats with monocrotaline-induced pulmonary hypertension.Obesity-induced tissue free radical generation: an in vivo immuno-spin trapping study.Transforming growth factor-beta1 protects against pulmonary artery endothelial cell apoptosis via ALK5.Diminazene attenuates pulmonary hypertension and improves angiogenic progenitor cell functions in experimental models.Genetics of pulmonary arterial hypertension.Calpain-2 activates Akt via TGF-β1-mTORC2 pathway in pulmonary artery smooth muscle cells Hypoxia-induced mitogenic factor (HIMF/FIZZ1/RELMalpha) induces the vascular and hemodynamic changes of pulmonary hypertension VEGF ameliorates pulmonary hypertension through inhibition of endothelial apoptosis in experimental lung fibrosis in rats.Transforming growth factor-beta1 causes pulmonary microvascular endothelial cell apoptosis via ALK5.Cyclohexanone contamination from extracorporeal circuits impairs cardiovascular function Transforming growth factor-β(1) represses bone morphogenetic protein-mediated Smad signaling in pulmonary artery smooth muscle cells via Smad3 Relevant issues in the pathology and pathobiology of pulmonary hypertension.TGFβ signaling and cardiovascular diseases.The Role of Astrocytes in Metabolism and Neurotoxicity of the Pyrrolizidine Alkaloid Monocrotaline, the Main Toxin of Crotalaria retusa.The transforming growth factor-β-bone morphogenetic protein type signalling pathway in pulmonary vascular homeostasis and disease.Molecular switches under TGFβ signalling during progression from cardiac hypertrophy to heart failure.Regulatory T cells limit vascular endothelial injury and prevent pulmonary hypertension.Matricellular protein thrombospondin-1 in pulmonary hypertension: multiple pathways to disease.Pulmonary artery smooth muscle hypertrophy: roles of glycogen synthase kinase-3beta and p70 ribosomal S6 kinase.Therapeutic potential of adipose stem cell-derived conditioned medium against pulmonary hypertension and lung fibrosis.Genetics of pulmonary arterial hypertension: do the molecular findings have translational value?Transforming growth factor-β signaling promotes pulmonary hypertension caused by Schistosoma mansoni.BMP type II receptor deficiency confers resistance to growth inhibition by TGF-β in pulmonary artery smooth muscle cells: role of proinflammatory cytokines A translational preclinical model of interstitial pulmonary fibrosis and pulmonary hypertension: mechanistic pathways driving disease pathophysiology Involvement of BMPR2 in the protective effect of fluoxetine against monocrotaline-induced endothelial apoptosis in rats.A Selective Transforming Growth Factor-β Ligand Trap Attenuates Pulmonary Hypertension.Activin-like kinase 5 (ALK5) mediates abnormal proliferation of vascular smooth muscle cells from patients with familial pulmonary arterial hypertension and is involved in the progression of experimental pulmonary arterial hypertension induced by mo

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Role of the TGF-beta/Alk5 signaling pathway in monocrotaline-induced pulmonary hypertension.

http://www.wikidata.org/entity/Q36539476

description

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2008年论文

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Role of the TGF-beta/Alk5 sign ...... nduced pulmonary hypertension.

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Role of the TGF-beta/Alk5 sign ...... nduced pulmonary hypertension.

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Role of the TGF-beta/Alk5 sign ...... nduced pulmonary hypertension.

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Role of the TGF-beta/Alk5 sign ...... nduced pulmonary hypertension.

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Role of the TGF-beta/Alk5 sign ...... nduced pulmonary hypertension.

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Role of the TGF-beta/Alk5 sign ...... nduced pulmonary hypertension.

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RCCM.200707-1083OC

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American Journal of Respiratory and Critical Care Medicine

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Role of the TGF-beta/Alk5 sign ...... nduced pulmonary hypertension.

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Alison Murphy

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Andrew Protter

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Ari L Zaiman

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Enid Neptune

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Fang Xu

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Hunter C Champion

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Kimberley Gordy

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Larissa A Shimoda

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Lijie Zhen

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Linda Higgins

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Altered growth responses of pulmonary artery smooth muscle cells from patients with primary pulmonary hypertension to transforming growth factor-beta(1) and bone morphogenetic proteins

Activin receptor-like kinase (ALK)1 is an antagonistic mediator of lateral TGFbeta/ALK5 signaling

Molecular and functional analysis identifies ALK-1 as the predominant cause of pulmonary hypertension related to hereditary haemorrhagic telangiectasia

Cell surface-localized matrix metalloproteinase-9 proteolytically activates TGF-beta and promotes tumor invasion and angiogenesis

Mechanisms of TGF-beta signaling from cell membrane to the nucleus

Smad-dependent and Smad-independent pathways in TGF-beta family signalling

A syndrome of altered cardiovascular, craniofacial, neurocognitive and skeletal development caused by mutations in TGFBR1 or TGFBR2

Aneurysm syndromes caused by mutations in the TGF-beta receptor

Smads: transcriptional activators of TGF-beta responses

BMPR2 haploinsufficiency as the inherited molecular mechanism for primary pulmonary hypertension.

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5649895

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