Aldosterone impairs vascular reactivity by decreasing glucose-6-phosphate dehydrogenase activity. - Test2 - elhamkhani - TriplyDB

Aldosterone impairs vascular reactivity by decreasing glucose-6-phosphate dehydrogenase activity.

Aldosterone impairs vascular reactivity by decreasing glucose-6-phosphate dehydrogenase activity.

http://www.wikidata.org/entity/Q36829138

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Regulation of G6PD acetylation by SIRT2 and KAT9 modulates NADPH homeostasis and cell survival during oxidative stress Mineralocorticoid Receptors in the Pathophysiology of Vascular Inflammation and Atherosclerosis S-nitrosothiols and the S-nitrosoproteome of the cardiovascular system Pharmacology of Aldosterone and the Effects of Mineralocorticoid Receptor Blockade on Cardiovascular Systems The role of the renin-angiotensin-aldosterone system in the pathobiology of pulmonary arterial hypertension (2013 Grover Conference series)Regulation of bone-renal mineral and energy metabolism: the PHEX, FGF23, DMP1, MEPE ASARM pathway Update on RAAS Modulation for the Treatment of Diabetic Cardiovascular Disease Emerging Concepts in the Molecular Basis of Pulmonary Arterial Hypertension: Part II: Neurohormonal Signaling Contributes to the Pulmonary Vascular and Right Ventricular Pathophenotype of Pulmonary Arterial Hypertension SIRT2 activates G6PD to enhance NADPH production and promote leukaemia cell proliferation Glutathione (GSH) and the GSH synthesis gene Gclm modulate plasma redox and vascular responses to acute diesel exhaust inhalation in mice Mineralocorticoid receptor antagonists and endothelial function Aldosterone's rapid, nongenomic effects are mediated by striatin: a modulator of aldosterone's effect on estrogen action.Oxidative risk for atherothrombotic cardiovascular disease.Matrix extracellular phosphoglycoprotein (MEPE) is a new bone renal hormone and vascularization modulator.Aldosterone receptor antagonists: effective but often forgotten.Aldosterone and inflammation.Upregulation of steroidogenic acute regulatory protein by hypoxia stimulates aldosterone synthesis in pulmonary artery endothelial cells to promote pulmonary vascular fibrosis.Glutathione peroxidase-1 modulates lipopolysaccharide-induced adhesion molecule expression in endothelial cells by altering CD14 expression.Regulation of the pentose phosphate pathway in cancer Deletion of the angiotensin II type 1 receptor-associated protein enhances renal sodium reabsorption and exacerbates angiotensin II-mediated hypertension Chronic aldosterone administration causes Nox2-mediated increases in reactive oxygen species production and endothelial dysfunction in the cerebral circulation.A functional RANKL polymorphism associated with younger age at onset of rheumatoid arthritis.pO(2)-dependent NO production determines OPPC activity in macrophages.Mineralocorticoid receptor antagonists and the metabolic syndrome Spironolactone lowers portal hypertension by inhibiting liver fibrosis, ROCK-2 activity and activating NO/PKG pathway in the bile-duct-ligated rat.Redox Pioneer: Professor Joseph Loscalzo.Endothelial cell metabolism: parallels and divergences with cancer cell metabolism Human alpha B-crystallin mutation causes oxido-reductive stress and protein aggregation cardiomyopathy in mice Aldosterone inhibits the fetal program and increases hypertrophy in the heart of hypertensive mice Systems analysis of oxidant stress in the vasculature Vascular endothelial function and hypertension: insights and directions Impact of glucose-6-phosphate dehydrogenase deficiency on the pathophysiology of cardiovascular disease.Resveratrol induces premature senescence in lung cancer cells via ROS-mediated DNA damage.Mineralocorticoid receptor blockade improves coronary microvascular function in individuals with type 2 diabetes.Upregulation of CREM/ICER suppresses wound endothelial CRE-HIF-1α-VEGF-dependent signaling and impairs angiogenesis in type 2 diabetes.Impairment of endothelial progenitor cell function and vascularization capacity by aldosterone in mice and humans.Aldosterone: a forgotten mediator of the relationship between psychological stress and heart disease Hypoxia-induced glucose-6-phosphate dehydrogenase overexpression and -activation in pulmonary artery smooth muscle cells: implication in pulmonary hypertension Healing the orphaned heart: heart failure in a patient with glucose-6-phosphate dehydrogenase deficiency.Glutathione peroxidase-1 deficiency augments proinflammatory cytokine-induced redox signaling and human endothelial cell activation.

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P2860

Aldosterone impairs vascular reactivity by decreasing glucose-6-phosphate dehydrogenase activity.

http://www.wikidata.org/entity/Q36829138

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2007 nî lūn-bûn

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Aldosterone impairs vascular r ...... sphate dehydrogenase activity.

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Aldosterone impairs vascular r ...... sphate dehydrogenase activity.

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Aamir Dam

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Anne W Scribner

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Bradley A Maron

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Robert C Stanton

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Ronglih Liao

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P2860

Expression of 5-lipoxygenase in pulmonary artery endothelial cells

Glucose-6 phosphate dehydrogenase deficiency decreases the vascular response to angiotensin II

Endothelial dysfunction in a murine model of mild hyperhomocyst(e)inemia

Endothelial nitric oxide synthase dysfunction in diabetic mice: importance of tetrahydrobiopterin in eNOS dimerisation.

Characterization of heme-deficient neuronal nitric-oxide synthase reveals a role for heme in subunit dimerization and binding of the amino acid substrate and tetrahydrobiopterin.

Glucose-6-phosphate dehydrogenase deficiency promotes endothelial oxidant stress and decreases endothelial nitric oxide bioavailability.

Glucose-6-phosphate dehydrogenase overexpression decreases endothelial cell oxidant stress and increases bioavailable nitric oxide.

Glucose-6-phosphate dehydrogenase modulates vascular endothelial growth factor-mediated angiogenesis.

Cyclic strain modulates resistance to oxidant stress by increasing G6PDH expression in smooth muscle cells.

Functional analysis of transcription factors CREB and CREM.

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2

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13

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6531800

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3648863

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