Activation of RAGE induces elevated O2- generation by mononuclear phagocytes in diabetes
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Diabetes mellitus related bone metabolism and periodontal diseaseDiabetes-induced oxidative stress is mediated by Ca2+-independent phospholipase A2 in neutrophils.Ursolic acid protects diabetic mice against monocyte dysfunction and accelerated atherosclerosis.Annato extract and β-carotene modulate the production of reactive oxygen species/nitric oxide in neutrophils from diabetic rats.Oral inflammatory diseases and systemic inflammation: role of the macrophage.Burkholderia pseudomallei triggers altered inflammatory profiles in a whole-blood model of type 2 diabetes-melioidosis comorbidityPhosphorylation of pleckstrin increases proinflammatory cytokine secretion by mononuclear phagocytes in diabetes mellitus.Markers of Antioxidant Defense in Patients with Type 2 DiabetesTuning three-dimensional collagen matrix stiffness independently of collagen concentration modulates endothelial cell behaviorReduced Mycobacterium tuberculosis association with monocytes from diabetes patients that have poor glucose controlIPLA2 mRNA expression by human neutrophils in type 2 diabetes and chronic periodontitisNa+/H+ exchanger 1 inhibition reverses manifestation of peripheral diabetic neuropathy in type 1 diabetic rats.Potential of the angiotensin receptor blockers (ARBs) telmisartan, irbesartan, and candesartan for inhibiting the HMGB1/RAGE axis in prevention and acute treatment of stroke.Endogenous damage-associated molecular pattern molecules at the crossroads of inflammation and cancer.Controlling the mechanical properties of three-dimensional matrices via non-enzymatic collagen glycationPriming of neutrophil oxidative burst in diabetes requires preassembly of the NADPH oxidase.Ulinastatin preconditioning attenuates inflammatory reaction of hepatic ischemia reperfusion injury in rats via high mobility group box 1(HMGB1) inhibitionQuintessential risk factors: their role in promoting cognitive dysfunction and Alzheimer's disease.Mechanisms of distal axonal degeneration in peripheral neuropathies.2016 ATVB Plenary Lecture: Receptor for Advanced Glycation Endproducts and Implications for the Pathogenesis an Treatment of Cardiometabolic Disorders: Spotlight on the Macrophage.Beneficial effects of a novel RAGE inhibitor on early diabetic retinopathy and tactile allodynia.Overexpression of Bcl-2 in vascular endothelium inhibits the microvascular lesions of diabetic retinopathy.Erythropoietin attenuates advanced glycation endproducts-induced toxicity of Schwann cells in vitro.Scavenging of reactive oxygen species by astaxanthin inhibits epithelial-mesenchymal transition in high glucose-stimulated mesothelial cells.STR/ort mice, a model for spontaneous osteoarthritis, exhibit elevated levels of both local and systemic inflammatory markers.Abnormal cell responses and role of TNF-α in impaired diabetic wound healing.RAGE mediates the inactivation of nAChRs in sympathetic neurons under high glucose conditions.Linking senile dementia to type 2 diabetes: role of oxidative stress markers, C-reactive protein and tumor necrosis factor-α.
P2860
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P2860
Activation of RAGE induces elevated O2- generation by mononuclear phagocytes in diabetes
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
2006年论文
@zh
2006年论文
@zh-cn
name
Activation of RAGE induces elevated O2- generation by mononuclear phagocytes in diabetes
@en
type
label
Activation of RAGE induces elevated O2- generation by mononuclear phagocytes in diabetes
@en
prefLabel
Activation of RAGE induces elevated O2- generation by mononuclear phagocytes in diabetes
@en
P2093
P2860
P356
P1476
Activation of RAGE induces elevated O2- generation by mononuclear phagocytes in diabetes
@en
P2093
Alan Malabanan
Alpdogan Kantarci
Hatice Hasturk
Thomas E Van Dyke
P2860
P304
P356
10.1189/JLB.0406262
P577
2006-11-09T00:00:00Z