Mixed lineage leukemia 5 (MLL5) protein regulates cell cycle progression and E2F1-responsive gene expression via association with host cell factor-1 (HCF-1)
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mRNAs and miRNAs in whole blood associated with lung hyperplasia, fibrosis, and bronchiolo-alveolar adenoma and adenocarcinoma after multi-walled carbon nanotube inhalation exposure in miceMLL5 maintains spindle bipolarity by preventing aberrant cytosolic aggregation of PLK1O-GlcNAc signaling in cancer metabolism and epigeneticsThe making of a sweet modification: structure and function of O-GlcNAc transferase.Association of the histone-lysine N-methyltransferase MLL5 gene with coronary artery disease in Chinese Han people.Solution NMR structure and histone binding of the PHD domain of human MLL5.Mixed Lineage Leukemia 5 (MLL5) Protein Stability Is Cooperatively Regulated by O-GlcNac Transferase (OGT) and Ubiquitin Specific Protease 7 (USP7).Impact of MLL5 expression on decitabine efficacy and DNA methylation in acute myeloid leukemia.The Human Mixed Lineage Leukemia 5 (MLL5), a Sequentially and Structurally Divergent SET Domain-Containing Protein with No Intrinsic Catalytic Activity.Found: a cellular activating ligand for NKp44.Hemorrhagic transformation after ischemic stroke in animals and humansIdentification of Candidate Genes Related to Inflammatory Bowel Disease Using Minimum Redundancy Maximum Relevance, Incremental Feature Selection, and the Shortest-Path Approach.Inhibitors of Protein Methyltransferases and Demethylases.Nutrient regulation of gene expression by O-GlcNAcylation of chromatinMLL5 (KMT2E): structure, function, and clinical relevance.O-GlcNAcylation and chromatin remodeling in mammals: an up-to-date overview.Functional evidence implicating chromosome 7q22 haploinsufficiency in myelodysplastic syndrome pathogenesis.O-GlcNAcylation of MLL5β is essential for MLL5β-AP-1 transcription complex assembly at the HPV16/18-long control region.MLL5 expression as a biomarker for DNA hypermethylation and sensitivity to epigenetic therapy.Bivalent Epigenetic Control of Oncofetal Gene Expression in Cancer.Expression of mutant Asxl1 perturbs hematopoiesis and promotes susceptibility to leukemic transformation.ASXL1 as a critical regulator of epigenetic marks and therapeutic potential of mutated cells
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P248
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P2860
Mixed lineage leukemia 5 (MLL5) protein regulates cell cycle progression and E2F1-responsive gene expression via association with host cell factor-1 (HCF-1)
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
Mixed lineage leukemia 5 (MLL5 ...... ith host cell factor-1 (HCF-1)
@en
type
label
Mixed lineage leukemia 5 (MLL5 ...... ith host cell factor-1 (HCF-1)
@en
prefLabel
Mixed lineage leukemia 5 (MLL5 ...... ith host cell factor-1 (HCF-1)
@en
P2093
P2860
P921
P356
P1476
Mixed lineage leukemia 5 (MLL5 ...... ith host cell factor-1 (HCF-1)
@en
P2093
Chuangui Wang
Cuihong Zhou
Feng Zhang
Jinduo Yuan
Peipei Zhou
Sidong Xiong
Xiaodan Ding
Xiaoling Wan
P2860
P304
17532-17543
P356
10.1074/JBC.M112.439729
P407
P577
2013-04-29T00:00:00Z