Transgenic models of Alzheimer's disease: better utilization of existing models through viral transgenesis. - Test2 - elhamkhani - TriplyDB

Transgenic models of Alzheimer's disease: better utilization of existing models through viral transgenesis.

Transgenic models of Alzheimer's disease: better utilization of existing models through viral transgenesis.

http://www.wikidata.org/entity/Q36950042

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Extracellular Vesicle-Associated Aβ Mediates Trans-Neuronal Bioenergetic and Ca2+-Handling Deficits in Alzheimer's Disease Models.Using mice to model Alzheimer's dementia: an overview of the clinical disease and the preclinical behavioral changes in 10 mouse models Obesity, diabetes, and leptin resistance promote tau pathology in a mouse model of disease.Abnormal tau induces cognitive impairment through two different mechanisms: synaptic dysfunction and neuronal loss The neuritic plaque facilitates pathological conversion of tau in an Alzheimer's disease mouse model.cAMP-PKA phosphorylation of tau confers risk for degeneration in aging association cortex.In vivo tracking of tau pathology using positron emission tomography (PET) molecular imaging in small animals.The role of cholesterol metabolism in Alzheimer's disease.Inspired by lipids: the Morton Lecture Award Presentation.Dietary DHA supplementation causes selective changes in phospholipids from different brain regions in both wild type mice and the Tg2576 mouse model of Alzheimer's disease.Amylin in Alzheimer's disease: Pathological peptide or potential treatment?

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P2860

Transgenic models of Alzheimer's disease: better utilization of existing models through viral transgenesis.

http://www.wikidata.org/entity/Q36950042

description

2013 nî lūn-bûn

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Transgenic models of Alzheimer ...... ls through viral transgenesis.

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Transgenic models of Alzheimer ...... ls through viral transgenesis.

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Transgenic models of Alzheimer ...... ls through viral transgenesis.

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J.BBADIS.2013.04.017

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Biochimica et Biophysica Acta

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Transgenic models of Alzheimer ...... ls through viral transgenesis.

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M Paul Murphy

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Thomas L Platt

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Valerie L Reeves

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P2860

Inhibition of mTOR by rapamycin abolishes cognitive deficits and reduces amyloid-beta levels in a mouse model of Alzheimer's disease

A protein factor essential for microtubule assembly

Correlative Memory Deficits, Abeta Elevation, and Amyloid Plaques in Transgenic Mice

Alzheimer's Disease

Alzheimer-type neuropathology in transgenic mice overexpressing V717F β-amyloid precursor protein

Tau pathology in Alzheimer disease and other tauopathies

Clinical features of Alzheimer's disease

Amyloid fibrils from the viewpoint of protein folding

Interaction of tau with the neural membrane cortex is regulated by phosphorylation at sites that are modified in paired helical filaments

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1437-1448

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10.1016/J.BBADIS.2013.04.017

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Alzheimer's disease

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