Using mice to model Alzheimer's dementia: an overview of the clinical disease and the preclinical behavioral changes in 10 mouse models - Test2 - elhamkhani - TriplyDB

Using mice to model Alzheimer's dementia: an overview of the clinical disease and the preclinical behavioral changes in 10 mouse models

Using mice to model Alzheimer's dementia: an overview of the clinical disease and the preclinical behavioral changes in 10 mouse models

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Neurotrophic factor small-molecule mimetics mediated neuroregeneration and synaptic repair: emerging therapeutic modality for Alzheimer's disease Organotypic Cultures as a Model to Study Adult Neurogenesis in CNS Disorders Induced pluripotent stem cells in Alzheimer's disease: applications for disease modeling and cell-replacement therapy Emerging Link between Alzheimer's Disease and Homeostatic Synaptic Plasticity Toward more predictive genetic mouse models of Alzheimer's disease Dissecting Alzheimer disease in Down syndrome using mouse models Translational neurophysiology in sheep: measuring sleep and neurological dysfunction in CLN5 Batten disease affected sheep.Transnasal delivery of human A-beta peptides elicits impaired learning and memory performance in wild type mice.Cross-species translation of the Morris maze for Alzheimer's disease.Suggesting a Way to Understand the Actual Potential of Anti-Alzheimer's Disease Drugs That Show Promise in Transgenic Mouse Models Cognitive Deficits, Changes in Synaptic Function, and Brain Pathology in a Mouse Model of Normal Aging(1,2,3).A semi-automated motion-tracking analysis of locomotion speed in the C. elegans transgenics overexpressing beta-amyloid in neurons.Longitudinal analysis of the behavioral phenotype in a novel transgenic rat model of early stages of Alzheimer's disease.Trial-unique, delayed nonmatching-to-location (TUNL) touchscreen testing for mice: sensitivity to dorsal hippocampal dysfunction.Elevated emotional contagion in a mouse model of Alzheimer's disease is associated with increased synchronization in the insula and amygdala Age- and Brain Region-Specific Changes of Glucose Metabolic Disorder, Learning, and Memory Dysfunction in Early Alzheimer's Disease Assessed in APP/PS1 Transgenic Mice Using 18F-FDG-PET.The Effect of Apolipoprotein E ε4 (APOE ε4) on Visuospatial Working Memory in Healthy Elderly and Amnestic Mild Cognitive Impairment Patients: An Event-Related Potentials Study.Analysis of RNA from Alzheimer's Disease Post-mortem Brain Tissues.Brain and Brown Adipose Tissue Metabolism in Transgenic Tg2576 Mice Models of Alzheimer Disease Assessed Using 18F-FDG PET Imaging.Alzheimer's patient feedback to complement research using model systems for cognitive aging and dementia.Insulin resistance in Alzheimer's disease.APOE4 enhances age-dependent decline in cognitive function by down-regulating an NMDA receptor pathway in EFAD-Tg mice.Intraneuronal APP and extracellular Aβ independently cause dendritic spine pathology in transgenic mouse models of Alzheimer's disease.Nxf7 deficiency impairs social exploration and spatio-cognitive abilities as well as hippocampal synaptic plasticity in mice.Long-Term Treatment with Liraglutide, a Glucagon-Like Peptide-1 (GLP-1) Receptor Agonist, Has No Effect on β-Amyloid Plaque Load in Two Transgenic APP/PS1 Mouse Models of Alzheimer's Disease.Lessons from a Rare Familial Dementia: Amyloid and Beyond.Forebrain microglia from wild-type but not adult 5xFAD mice prevent amyloid-β plaque formation in organotypic hippocampal slice cultures.Motor Cortex Theta and Gamma Architecture in Young Adult APPswePS1dE9 Alzheimer Mice.Impaired Cholinergic Excitation of Prefrontal Attention Circuitry in the TgCRND8 Model of Alzheimer's Disease Metabolic and Non-Cognitive Manifestations of Alzheimer's Disease: The Hypothalamus as Both Culprit and Target of Pathology Amelioration of cognitive impairments in APPswe/PS1dE9 mice is associated with metabolites alteration induced by total salvianolic acid.L-Stepholidine rescues memory deficit and synaptic plasticity in models of Alzheimer's disease via activating dopamine D1 receptor/PKA signaling pathway.Chronic Treatment with Naltrexone Prevents Memory Retention Deficits in Rats Poisoned with the Sarin Analog Diisopropylfluorophosphate (DFP) and Treated with Atropine and Pralidoxime Hippocampal Injections of Oligomeric Amyloid β-peptide (1-42) Induce Selective Working Memory Deficits and Long-lasting Alterations of ERK Signaling Pathway.Wet or dry: translatable "water mazes" for mice and humans Abnormal tau induces cognitive impairment through two different mechanisms: synaptic dysfunction and neuronal loss Vitamin D interacts with Esr1 and Igf1 to regulate molecular pathways relevant to Alzheimer's disease Absence of Prenatal Forebrain Defects in the Dp(16)1Yey/+ Mouse Model of Down Syndrome.Deficits in hippocampal-dependent transfer generalization learning accompany synaptic dysfunction in a mouse model of amyloidosis Hippocampal neurogenesis: Learning to remember

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Using mice to model Alzheimer's dementia: an overview of the clinical disease and the preclinical behavioral changes in 10 mouse models

http://www.wikidata.org/entity/Q34418781

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2014 nî lūn-bûn

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2014 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2014 թվականի ապրիլին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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Using mice to model Alzheimer' ...... ral changes in 10 mouse models

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Using mice to model Alzheimer' ...... ral changes in 10 mouse models

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Using mice to model Alzheimer' ...... ral changes in 10 mouse models

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Using mice to model Alzheimer' ...... ral changes in 10 mouse models

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Using mice to model Alzheimer' ...... ral changes in 10 mouse models

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Using mice to model Alzheimer' ...... ral changes in 10 mouse models

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FGENE.2014.00088

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Frontiers in Genetics

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Using mice to model Alzheimer' ...... ral changes in 10 mouse models

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Frederick A Schmitt

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Linda J Van Eldik

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Peter T Nelson

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Scott J Webster

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P2860

DHA improves cognition and prevents dysfunction of entorhinal cortex neurons in 3xTg-AD mice

The importance of neuritic plaques and tangles to the development and evolution of AD

Correlative Memory Deficits, Abeta Elevation, and Amyloid Plaques in Transgenic Mice

Alzheimer-type neuropathology in transgenic mice overexpressing V717F β-amyloid precursor protein

ApoE-directed therapeutics rapidly clear β-amyloid and reverse deficits in AD mouse models

Passive immunotherapy against Abeta in aged APP-transgenic mice reverses cognitive deficits and depletes parenchymal amyloid deposits in spite of increased vascular amyloid and microhemorrhage

“Mini-mental state”

Early phenotypic changes in transgenic mice that overexpress different mutants of amyloid precursor protein in brain

Memory dysfunction

Early-onset Alzheimer's disease caused by mutations at codon 717 of the beta-amyloid precursor protein gene

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10.3389/FGENE.2014.00088

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5

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Adam D Bachstetter

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