Slug Is Increased in Vascular Remodeling and Induces a Smooth Muscle Cell Proliferative Phenotype. - Test2 - elhamkhani - TriplyDB

Slug Is Increased in Vascular Remodeling and Induces a Smooth Muscle Cell Proliferative Phenotype.

Slug Is Increased in Vascular Remodeling and Induces a Smooth Muscle Cell Proliferative Phenotype.

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Correction: Slug Is Increased in Vascular Remodeling and Induces a Smooth Muscle Cell Proliferative Phenotype.

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Slug Is Increased in Vascular Remodeling and Induces a Smooth Muscle Cell Proliferative Phenotype.

http://www.wikidata.org/entity/Q37115214

description

article científic

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article scientifique

@fr

articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 21 July 2016

@en

vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Slug Is Increased in Vascular ...... Cell Proliferative Phenotype.

@en

Slug Is Increased in Vascular ...... Cell Proliferative Phenotype.

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type

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Slug Is Increased in Vascular ...... Cell Proliferative Phenotype.

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Slug Is Increased in Vascular ...... Cell Proliferative Phenotype.

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Slug Is Increased in Vascular ...... Cell Proliferative Phenotype.

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Slug Is Increased in Vascular ...... Cell Proliferative Phenotype.

@nl

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JOURNAL.PONE.0159460

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Slug Is Increased in Vascular ...... Cell Proliferative Phenotype.

@en

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Gunter Meister

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Isabel Blanco

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Jessica García-Lucio

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Joan Albert Barberà

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María V Pisano

http://www.w3.org/2001/XMLSchema#string

Maurits Evers

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Núria Coll-Bonfill

http://www.w3.org/2001/XMLSchema#string

Olga Tura-Ceide

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The zinc-finger protein slug causes desmosome dissociation, an initial and necessary step for growth factor-induced epithelial-mesenchymal transition

Evolving gene/transcript definitions significantly alter the interpretation of GeneChip data.

Myocardin is a master regulator of smooth muscle gene expression

Linear models and empirical bayes methods for assessing differential expression in microarray experiments

Summaries of Affymetrix GeneChip probe level data

The snail superfamily of zinc-finger transcription factors

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KLF4-dependent phenotypic modulation of smooth muscle cells has a key role in atherosclerotic plaque pathogenesis.

Smooth muscle alpha-actin gene requires two E-boxes for proper expression in vivo and is a target of class I basic helix-loop-helix proteins.

TNF-α induces phenotypic modulation in cerebral vascular smooth muscle cells: implications for cerebral aneurysm pathology.

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e0159460

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scholarly article

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10.1371/JOURNAL.PONE.0159460

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7

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11

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Marcelina Parrizas

Julia C Engelmann

Victor I Peinado

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2016-07-21T00:00:00Z

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27441378

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4956159

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