Identification of infected B-cell populations by using a recombinant murine gammaherpesvirus 68 expressing a fluorescent protein
about
Shared mechanisms in stemness and carcinogenesis: lessons from oncogenic virusesDefining immune engagement thresholds for in vivo control of virus-driven lymphoproliferationPhosphoproteomic analyses reveal signaling pathways that facilitate lytic gammaherpesvirus replicationInterplay of Murine Gammaherpesvirus 68 with NF-kappaB Signaling of the HostA gammaherpesvirus Bcl-2 ortholog blocks B cell receptor-mediated apoptosis and promotes the survival of developing B cells in vivoCharacterization of omental immune aggregates during establishment of a latent gammaherpesvirus infectionTracking murine gammaherpesvirus 68 infection of germinal center B cells in vivoVariable episomal silencing of a recombinant herpesvirus renders its encoded GFP an unreliable marker of infection in primary cells.Gammaherpesvirus-driven plasma cell differentiation regulates virus reactivation from latently infected B lymphocytes.Expansion of murine gammaherpesvirus latently infected B cells requires T follicular help.Identification of alternative transcripts encoding the essential murine gammaherpesvirus lytic transactivator RTASpecific humoral immunity versus polyclonal B cell activation in Trypanosoma cruzi infection of susceptible and resistant mice.Murine gammaherpesvirus 68 LANA is essential for virus reactivation from splenocytes but not long-term carriage of viral genome.Global mRNA degradation during lytic gammaherpesvirus infection contributes to establishment of viral latency.The murine gammaherpesvirus immediate-early Rta synergizes with IRF4, targeting expression of the viral M1 superantigen to plasma cellsMurine gamma-herpesvirus immortalization of fetal liver-derived B cells requires both the viral cyclin D homolog and latency-associated nuclear antigenTyrosine 129 of the murine gammaherpesvirus M2 protein is critical for M2 function in vivoUnbiased mutagenesis of MHV68 LANA reveals a DNA-binding domain required for LANA function in vitro and in vivo.Stabilization of Myc through heterotypic poly-ubiquitination by mLANA is critical for γ-herpesvirus lymphoproliferation.Establishment of murine gammaherpesvirus latency in B cells is not a stochastic event.Interleukin 21 signaling in B cells is required for efficient establishment of murine gammaherpesvirus latencyMurine Gammaherpesvirus 68 Pathogenesis Is Independent of Caspase-1 and Caspase-11 in Mice and Impairs Interleukin-1β Production upon Extrinsic Stimulation in Culture.Tiled microarray identification of novel viral transcript structures and distinct transcriptional profiles during two modes of productive murine gammaherpesvirus 68 infection.ATM facilitates mouse gammaherpesvirus reactivation from myeloid cells during chronic infection.Retention of anergy and inhibition of antibody responses during acute γ herpesvirus 68 infectionSubcapsular sinus macrophages limit acute gammaherpesvirus disseminationThe absence of M1 leads to increased establishment of murine gammaherpesvirus 68 latency in IgD-negative B cells.Systemic and local infection routes govern different cellular dissemination pathways during gammaherpesvirus infection in vivoAblation of STAT3 in the B Cell Compartment Restricts Gammaherpesvirus Latency In Vivo.MicroRNA miR-155 Is Necessary for Efficient Gammaherpesvirus Reactivation from Latency, but Not for Establishment of LatencyGLUT1-mediated glucose uptake plays a crucial role during Plasmodium hepatic infection.Murid Gammaherpesvirus Latency-Associated Protein M2 Promotes the Formation of Conjugates between Transformed B Lymphoma Cells and T Helper Cells.Murine gammaherpesvirus 68 reactivation from B cells requires IRF4 but not XBP-1.Tumor Suppressor Interferon-Regulatory Factor 1 Counteracts the Germinal Center Reaction Driven by a Cancer-Associated Gammaherpesvirus.Use of a virus-encoded enzymatic marker reveals that a stable fraction of memory B cells expresses latency-associated nuclear antigen throughout chronic gammaherpesvirus infection.The de novo methyltransferases DNMT3a and DNMT3b target the murine gammaherpesvirus immediate-early gene 50 promoter during establishment of latency.Germinal center B cells latently infected with Epstein-Barr virus proliferate extensively but do not increase in number.Blimp-1-dependent plasma cell differentiation is required for efficient maintenance of murine gammaherpesvirus latency and antiviral antibody responses.Cross-species conservation of episome maintenance provides a basis for in vivo investigation of Kaposi's sarcoma herpesvirus LANA.Murine gammaherpesvirus M2 antigen modulates splenic B cell activation and terminal differentiation in vivo.
P2860
Q26863339-47BFD033-95AB-4F8B-8CF8-0A3A01059653Q27324746-DB153A82-293B-4659-9522-49469DE3C122Q27333727-059B8349-2B1B-4099-863D-1DD771B58F78Q28080198-864BF441-C450-48F9-8645-C058DC910F77Q28539635-76560FC4-F924-49BC-AC8D-034E9370A19CQ28728172-CFDA1643-729F-48D9-BF9F-7AB7595508ADQ28731326-1F283DC1-9523-4025-B152-B5FB7042A9B3Q30301295-F55CC1FB-0AFF-4949-B2E6-4E2808ED140DQ30943994-971253BF-2625-4A8E-AD43-3B9B94B9FF6AQ33553789-D8E8938F-455B-429B-91BB-37A09C63E1A1Q33602737-8C4647A1-96B8-4DD2-B437-C0D50D3FD17DQ33631293-F211C54A-A93E-4F74-B7D4-B4CD46B1F738Q33966678-71C4F235-06A5-4478-88AD-D878AD7E6566Q33979518-A5CF37B7-DE92-4524-A032-0895D174B314Q34015827-BE707B63-7E9A-49E8-ADD1-D53457F93F01Q34022999-49A68350-61BB-4FBC-ADF3-65460044F506Q34047454-7124ABAA-8D68-415F-8406-85D640413BFBQ34411851-3FB848D5-5521-4B0F-9278-FA9D22E6569AQ34945490-12BD7762-C76F-45D2-93F0-E6EA4479AB1AQ35216453-0F2BC73C-C981-44C0-8E48-DAFF1CD85557Q35392834-750EC7F3-41EC-4EB6-B9EE-9BBB13836B6FQ35745447-DE547542-E753-4BBF-9EB5-8D19C0FA4859Q35867993-6F4481FF-9584-4125-B2D1-77424C8B595CQ35893600-B2E0304D-9F07-48DA-AC00-E3DF16CE68EFQ36218594-968FA5C3-98BA-4734-8EEB-7B4548301555Q36376628-31C78297-2D6C-4A27-BFAE-EDF771D2CB2CQ36667866-D0191ED4-34B3-4A55-82EC-BBD1B9349565Q36760054-795FC967-CDF1-40B1-B3C8-BE10111688B1Q37168674-808447F9-8D74-404C-ADEB-8A5E3FDA6EB7Q37182630-90B96B56-C16A-453F-88BB-A7E57EAABA4CQ37631769-D463D712-4E5E-4B1A-9C75-A6F7AF1A6284Q38821524-0308CE26-FB17-4DE2-80C2-BA3E53CEE206Q38970026-7D2883E1-81DE-4700-82C7-902F5B20BA1AQ39602832-A2707E28-94B0-42CD-ADB3-B6FCC6989B78Q39702331-605269B0-5621-4436-BA9F-7F07690BEEF9Q39732626-EB6CC4B5-5163-4823-A772-1C75B2FE2DDCQ39778902-4354CBD7-CC63-4A28-AB5B-2EAFEB053F2FQ39778958-E0E1FD2D-C301-4CB8-A730-52833F7E3CD6Q40041231-91291677-5FEB-494E-BE83-0AF0057CBC59Q40098067-BF3024BE-C7F5-4E50-9B1A-96862FCB9431
P2860
Identification of infected B-cell populations by using a recombinant murine gammaherpesvirus 68 expressing a fluorescent protein
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 22 April 2009
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Identification of infected B-c ...... pressing a fluorescent protein
@en
Identification of infected B-c ...... ressing a fluorescent protein.
@nl
type
label
Identification of infected B-c ...... pressing a fluorescent protein
@en
Identification of infected B-c ...... ressing a fluorescent protein.
@nl
prefLabel
Identification of infected B-c ...... pressing a fluorescent protein
@en
Identification of infected B-c ...... ressing a fluorescent protein.
@nl
P2093
P2860
P356
P1433
P1476
Identification of infected B-c ...... pressing a fluorescent protein
@en
P2093
Christopher M Collins
Jeremy M Boss
Samuel H Speck
P2860
P304
P356
10.1128/JVI.00297-09
P407
P577
2009-04-22T00:00:00Z