Epithelial-mesenchymal transition induces endoplasmic-reticulum-stress response in human colorectal tumor cells.
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Glucose deprivation elicits phenotypic plasticity via ZEB1-mediated expression of NNMT.New frontiers in the treatment of colorectal cancer: Autophagy and the unfolded protein response as promising targetsIntestinal epithelial HuR modulates distinct pathways of proliferation and apoptosis and attenuates small intestinal and colonic tumor developmentDriving Cancer Tumorigenesis and Metastasis Through UPR Signaling.
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Epithelial-mesenchymal transition induces endoplasmic-reticulum-stress response in human colorectal tumor cells.
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article científic
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article scientifique
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bilimsel makale
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scientific article published on 31 January 2014
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Epithelial-mesenchymal transit ...... human colorectal tumor cells.
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Epithelial-mesenchymal transit ...... human colorectal tumor cells.
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Epithelial-mesenchymal transit ...... human colorectal tumor cells.
@en
Epithelial-mesenchymal transit ...... human colorectal tumor cells.
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Epithelial-mesenchymal transit ...... human colorectal tumor cells.
@en
Epithelial-mesenchymal transit ...... human colorectal tumor cells.
@nl
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Epithelial-mesenchymal transit ...... human colorectal tumor cells.
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Andreas Jung
Evelyn Zeindl-Eberhart
Lydia Brandl
Sibylle Liebmann
Silvio K Scheel
Steffen Ormanns
Thomas Brabletz
Thomas Kirchner
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10.1371/JOURNAL.PONE.0087386
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P577
2014-01-31T00:00:00Z