Strand invasion by HLTF as a mechanism for template switch in fork rescue.
about
Human HLTF mediates postreplication repair by its HIRAN domain-dependent replication fork remodelling.Structure of a Novel DNA-binding Domain of Helicase-like Transcription Factor (HLTF) and Its Functional Implication in DNA Damage ToleranceHelicase-like transcription factor: a new marker of well-differentiated thyroid cancers.DNA-dependent protease activity of human Spartan facilitates replication of DNA-protein crosslink-containing DNA.Chronic treatment with cisplatin induces replication-dependent sister chromatid recombination to confer cisplatin-resistant phenotype in nasopharyngeal carcinoma.Visualization of recombination-mediated damage bypass by template switching.Rad51-mediated replication fork reversal is a global response to genotoxic treatments in human cells.Protein degradation pathways regulate the functions of helicases in the DNA damage response and maintenance of genomic stabilityDNA damage tolerance pathway involving DNA polymerase ι and the tumor suppressor p53 regulates DNA replication fork progression.SMARCAL1 maintains telomere integrity during DNA replication.HIV-1 Vpr degrades the HLTF DNA translocase in T cells and macrophages.Heterozygous PALB2 c.1592delT mutation channels DNA double-strand break repair into error-prone pathways in breast cancer patients.Rad51 and Rad54 promote noncrossover recombination between centromere repeats on the same chromatid to prevent isochromosome formation.Risks at the DNA Replication Fork: Effects upon Carcinogenesis and Tumor HeterogeneityMechanisms of Post-Replication DNA RepairReplication fork reversal in eukaryotes: from dead end to dynamic response.Chromosomal Rearrangements in Cancer: Detection and potential causal mechanisms.The helicase-like transcription factor (HLTF) in cancer: loss of function or oncomorphic conversion of a tumor suppressor?Homologous recombination maintenance of genome integrity during DNA damage tolerance.SMARCAL1 and telomeres: Replicating the troublesome endsEssential domains of Schizosaccharomyces pombe Rad8 required for DNA damage responseThe identification of FANCD2 DNA binding domains reveals nuclear localization sequences.Functions of SMARCAL1, ZRANB3, and HLTF in maintaining genome stability.DNA Damage Tolerance Pathway Choice Through Uls1 Modulation of Srs2 SUMOylation in Saccharomyces cerevisiae.AtRAD5A is a DNA translocase harboring a HIRAN domain which confers binding to branched DNA structures and is required for DNA repair in vivo.The HIRAN domain of helicase-like transcription factor positions the DNA translocase motor to drive efficient DNA fork regressionUbiquitylation at the Fork: Making and Breaking Chains to Complete DNA Replication
P2860
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P2860
Strand invasion by HLTF as a mechanism for template switch in fork rescue.
description
2013 nî lūn-bûn
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2013年の論文
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2013年学术文章
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2013年学术文章
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2013年学术文章
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2013年学术文章
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2013年学术文章
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2013年學術文章
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2013年學術文章
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2013年學術文章
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name
Strand invasion by HLTF as a mechanism for template switch in fork rescue.
@en
Strand invasion by HLTF as a mechanism for template switch in fork rescue.
@nl
type
label
Strand invasion by HLTF as a mechanism for template switch in fork rescue.
@en
Strand invasion by HLTF as a mechanism for template switch in fork rescue.
@nl
prefLabel
Strand invasion by HLTF as a mechanism for template switch in fork rescue.
@en
Strand invasion by HLTF as a mechanism for template switch in fork rescue.
@nl
P2093
P2860
P356
P1476
Strand invasion by HLTF as a mechanism for template switch in fork rescue.
@en
P2093
David Balogh
Lajos Haracska
Peter Burkovics
P2860
P304
P356
10.1093/NAR/GKT1040
P407
P577
2013-11-05T00:00:00Z