Synergistic and feedback signaling mechanisms in the regulation of inflammation in respiratory infections. - Test2 - elhamkhani - TriplyDB

Synergistic and feedback signaling mechanisms in the regulation of inflammation in respiratory infections.

Synergistic and feedback signaling mechanisms in the regulation of inflammation in respiratory infections.

http://www.wikidata.org/entity/Q37728927

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Mechanisms of corticosteroid insensitivity in COPD alveolar macrophages exposed to NTHi Regulation of osteoclast homeostasis and inflammatory bone loss by MFG-E8.CYLD negatively regulates nontypeable Haemophilus influenzae-induced IL-8 expression via phosphatase MKP-1-dependent inhibition of ERK.MFG-E8, a novel homeostatic regulator of osteoclastogenesis.Modular Transcriptional Networks of the Host Pulmonary Response during Early and Late Pneumococcal Pneumonia.Association of sputum microbiota profiles with severity of community-acquired pneumonia in children.Local Epidermal Growth Factor Receptor Signaling Mediates the Systemic Pathogenic Effects of Staphylococcus aureus Toxic Shock Syndrome Downregulation of miR-362-5p inhibits proliferation, migration and invasion of human breast cancer MCF7 cells Inhibition of PDE4B suppresses inflammation by increasing expression of the deubiquitinase CYLD.Bacterial colonization dampens influenza-mediated acute lung injury via induction of M2 alveolar macrophages.Lower nasopharyngeal epithelial cell repair and diminished innate inflammation responses contribute to the onset of acute otitis media in otitis-prone children.Ephedrine hydrochloride inhibits PGN-induced inflammatory responses by promoting IL-10 production and decreasing proinflammatory cytokine secretion via the PI3K/Akt/GSK3β pathway.The Lung Immune Response to Nontypeable Haemophilus influenzae (Lung Immunity to NTHi).Genome Expression Profiling-Based Identification and Administration Efficacy of Host-Directed Antimicrobial Drugs against Respiratory Infection by Nontypeable Haemophilus influenzae.In the Shadow of Fibrosis: Innate Immune Suppression Mediated by Transforming Growth Factor-β.Clinical significance of CYLD downregulation in breast cancer.Serum Cytokine Responses over the Entire Clinical-Immunological Spectrum of Human Leishmania (L.) infantum chagasi Infection.Resveratrol therapeutics combines both antimicrobial and immunomodulatory properties against respiratory infection by nontypeable Haemophilus influenzae.miR‑197‑3p‑induced downregulation of lysine 63 deubiquitinase promotes cell proliferation and inhibits cell apoptosis in lung adenocarcinoma cell lines.A recombinant TGF-β1 vaccine ameliorates diabetic nephropathy in OLETF rats.

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Synergistic and feedback signaling mechanisms in the regulation of inflammation in respiratory infections.

http://www.wikidata.org/entity/Q37728927

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 06 February 2012

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Synergistic and feedback signa ...... ion in respiratory infections.

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Synergistic and feedback signa ...... ion in respiratory infections.

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type

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Synergistic and feedback signa ...... ion in respiratory infections.

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Synergistic and feedback signa ...... ion in respiratory infections.

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Synergistic and feedback signa ...... ion in respiratory infections.

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Synergistic and feedback signa ...... ion in respiratory infections.

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Cellular and Molecular Immunology

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Synergistic and feedback signa ...... ion in respiratory infections.

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Jae Hyang Lim

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Jian-Dong Li

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Wenzhuo Y Wang

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The E3 ligase Itch and deubiquitinase Cyld act together to regulate Tak1 and inflammation

De-ubiquitination and ubiquitin ligase domains of A20 downregulate NF-kappaB signalling

Failure to regulate TNF-induced NF-kappaB and cell death responses in A20-deficient mice

Synergistic effect of interleukin 1 alpha on nontypeable Haemophilus influenzae-induced up-regulation of human beta-defensin 2 in middle ear epithelial cells.

Identification of the familial cylindromatosis tumour-suppressor gene

CYLD is a deubiquitinating enzyme that negatively regulates NF-kappaB activation by TNFR family members

Loss of the cylindromatosis tumour suppressor inhibits apoptosis by activating NF-kappaB

The tumour suppressor CYLD negatively regulates NF-kappaB signalling by deubiquitination

New insights into TGF-beta-Smad signalling

NF-kappaB is essential for induction of CYLD, the negative regulator of NF-kappaB: evidence for a novel inducible autoregulatory feedback pathway

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131-135

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scholarly article

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10.1038/CMI.2011.65

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2

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9

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2012-02-06T00:00:00Z

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22307042

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