CNS peroxiredoxins and their regulation in health and disease.
about
The active site architecture in peroxiredoxins: a case study on Mycobacterium tuberculosis AhpEAdaptive regulation of the brain's antioxidant defences by neurons and astrocytesNeuronal development is promoted by weakened intrinsic antioxidant defences due to epigenetic repression of Nrf2The extraordinary catalytic ability of peroxiredoxins: a combined experimental and QM/MM study on the fast thiol oxidation stepNeurons and neuronal activity control gene expression in astrocytes to regulate their development and metabolism.The neuroprotective effect of Klotho is mediated via regulation of members of the redox system.Sestrin2 decreases renal oxidative stress, lowers blood pressure, and mediates dopamine D2 receptor-induced inhibition of reactive oxygen species production.Astroglial PGC-1alpha increases mitochondrial antioxidant capacity and suppresses inflammation: implications for multiple sclerosisOxidative stress in neurodegenerative diseases: mechanisms and therapeutic perspectivesQuantitative proteomic and genetic analyses of the schizophrenia susceptibility factor dysbindin identify novel roles of the biogenesis of lysosome-related organelles complex 1.Expression of mRNA Encoding Mcu and Other Mitochondrial Calcium Regulatory Genes Depends on Cell Type, Neuronal Subtype, and Ca2+ SignalingSelective dendritic susceptibility to bioenergetic, excitotoxic and redox perturbations in cortical neurons.Sequential Upregulation of Superoxide Dismutase 2 and Heme Oxygenase 1 by tert-Butylhydroquinone Protects Mitochondria during Oxidative Stress.Human umbilical cord blood cells induce neuroprotective change in gene expression profile in neurons after ischemia through activation of Akt pathway.Adaptive changes in the neuronal proteome: mitochondrial energy production, endoplasmic reticulum stress, and ribosomal dysfunction in the cellular response to metabolic stress.Human stem cell-derived astrocytes and their application to studying Nrf2-mediated neuroprotective pathways and therapeutics in neurodegenerationActivation of Nrf2-regulated glutathione pathway genes by ischemic preconditioning.Synaptic NMDA receptor activity is coupled to the transcriptional control of the glutathione system.Nogo/RTN4 isoforms and RTN3 expression protect SH-SY5Y cells against multiple death insults.Nrf2-dependent persistent oxidative stress results in stress-induced vulnerability to depression.Pro-death NMDA receptor signaling is promoted by the GluN2B C-terminus independently of Dapk1.Aerobic exercise combined with antioxidative treatment does not counteract moderate- or mid-stage Alzheimer-like pathophysiology of APP/PS1 mice.Transcriptional regulators of redox balance and other homeostatic processes with the potential to alter neurodegenerative disease trajectory.Neuroprotective dobutamine treatment upregulates superoxide dismutase 3, anti-oxidant and survival genes and attenuates genes mediating inflammation.Neuroprotectants in the Era of Reperfusion Therapy.
P2860
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P2860
CNS peroxiredoxins and their regulation in health and disease.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 25 February 2011
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
CNS peroxiredoxins and their regulation in health and disease.
@en
CNS peroxiredoxins and their regulation in health and disease.
@nl
type
label
CNS peroxiredoxins and their regulation in health and disease.
@en
CNS peroxiredoxins and their regulation in health and disease.
@nl
prefLabel
CNS peroxiredoxins and their regulation in health and disease.
@en
CNS peroxiredoxins and their regulation in health and disease.
@nl
P356
P1476
CNS peroxiredoxins and their regulation in health and disease.
@en
P2093
Karen F S Bell
P304
P356
10.1089/ARS.2010.3567
P577
2011-02-25T00:00:00Z