about
Sequence and expression of human GABAA receptor alpha 1 and beta 1 subunitsGlia as drivers of abnormal neuronal activitySLC7A11 expression is associated with seizures and predicts poor survival in patients with malignant gliomaGABAergic disinhibition and impaired KCC2 cotransporter activity underlie tumor-associated epilepsy.Current transients associated with BK channels in human glioma cellsChlorotoxin inhibits glioma cell invasion via matrix metalloproteinase-2Functional chloride channels by mammalian cell expression of rat glycine receptor subunitBradykinin promotes the chemotactic invasion of primary brain tumorsAutocrine regulation of glioma cell proliferation via pHe-sensitive K(+) channelsRole of glutamate transporters in redox homeostasis of the brain.Spinal cord injury causes a wide-spread, persistent loss of Kir4.1 and glutamate transporter 1: benefit of 17 beta-oestradiol treatmentMolecular interaction and functional regulation of ClC-3 by Ca2+/calmodulin-dependent protein kinase II (CaMKII) in human malignant glioma.Inhibition of the Sodium-Potassium-Chloride Cotransporter Isoform-1 reduces glioma invasion.Disruption of astrocyte-vascular coupling and the blood-brain barrier by invading glioma cells.KCa3.1 modulates neuroblast migration along the rostral migratory stream (RMS) in vivo.Hypoxia increases the dependence of glioma cells on glutathioneSulfasalazine inhibits the growth of primary brain tumors independent of nuclear factor-kappaB.Reactive astrogliosis causes the development of spontaneous seizures.Ion channels and transporters [corrected] in cancer. 2. Ion channels and the control of cancer cell migrationGlutamate release by primary brain tumors induces epileptic activityMalignant gliomas: perverting glutamate and ion homeostasis for selective advantage.Hydrodynamic cellular volume changes enable glioma cell invasionIon channels and amino acid transporters support the growth and invasion of primary brain tumors.Kinase activation of ClC-3 accelerates cytoplasmic condensation during mitotic cell roundingAutocrine glutamate signaling promotes glioma cell invasion.Differential role of IK and BK potassium channels as mediators of intrinsic and extrinsic apoptotic cell death.BK channels are linked to inositol 1,4,5-triphosphate receptors via lipid rafts: a novel mechanism for coupling [Ca(2+)](i) to ion channel activation.Anion channels in astrocytes: biophysics, pharmacology, and function.Unique biology of gliomas: challenges and opportunities.Neuregulin-1 enhances survival of human astrocytic glioma cells.Biophysical and pharmacological characterization of hypotonically activated chloride currents in cortical astrocytesRelative contribution of chloride channels and transporters to regulatory volume decrease in human glioma cellsA role for glutamate in growth and invasion of primary brain tumorsAn unexpected role for ion channels in brain tumor metastasisFunctional implications for Kir4.1 channels in glial biology: from K+ buffering to cell differentiationClC3 is a critical regulator of the cell cycle in normal and malignant glial cellsBradykinin-induced chemotaxis of human gliomas requires the activation of KCa3.1 and ClC-3Chloride accumulation drives volume dynamics underlying cell proliferation and migration.Vascular amyloidosis impairs the gliovascular unit in a mouse model of Alzheimer's disease.Transient receptor potential canonical channels are essential for chemotactic migration of human malignant gliomas.
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description
hulumtues
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researcher
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ricercatore
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wetenschapper
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հետազոտող
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name
Harald Sontheimer
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Harald Sontheimer
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Harald Sontheimer
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Harald Sontheimer
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Harald Sontheimer
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type
label
Harald Sontheimer
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Harald Sontheimer
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Harald Sontheimer
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Harald Sontheimer
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Harald Sontheimer
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H.W. Sontheimer
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prefLabel
Harald Sontheimer
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Harald Sontheimer
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Harald Sontheimer
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Harald Sontheimer
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Harald Sontheimer
@sl
P108
P214
P244
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7102210801
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P214
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n2015187267
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P496
0000-0002-5843-9871
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lccn-n2015187267