Reactive astrogliosis causes the development of spontaneous seizures. - Test2 - elhamkhani - TriplyDB

Reactive astrogliosis causes the development of spontaneous seizures.

Reactive astrogliosis causes the development of spontaneous seizures.

http://www.wikidata.org/entity/Q35117565

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Glia as drivers of abnormal neuronal activity Elusive roles for reactive astrocytes in neurodegenerative diseases Critical Evaluation of P2X7 Receptor Antagonists in Selected Seizure Models The Indispensable Roles of Microglia and Astrocytes during Brain Development Genetic variation in the adenosine regulatory cycle is associated with posttraumatic epilepsy development Astrocyte roles in traumatic brain injury.From Molecular Circuit Dysfunction to Disease: Case Studies in Epilepsy, Traumatic Brain Injury, and Alzheimer's Disease.Biomarkers of Epileptogenesis: The Focus on Glia and Cognitive Dysfunctions.2014 Epilepsy Benchmarks Area II: Prevent Epilepsy and Its Progression.Remarkable alterations of Nav1.6 in reactive astrogliosis during epileptogenesis Postnatal activation of TLR4 in astrocytes promotes excitatory synaptogenesis in hippocampal neurons.Spatiotemporal progression of ubiquitin-proteasome system inhibition after status epilepticus suggests protective adaptation against hippocampal injury.Acute Increases in Protein O-GlcNAcylation Dampen Epileptiform Activity in Hippocampus.Circadian clustering of spontaneous epileptic seizures emerges after pilocarpine-induced status epilepticus."Targeting astrocytes in CNS injury and disease: A translational research approach".Astroglial Scarring and Seizures: A Cell Biological Perspective on Epilepsy.Review: Roles for astrocytes in epilepsy: insights from malformations of cortical development.A potential role for glia-derived extracellular matrix remodeling in postinjury epilepsy.Functional remodeling of subtype-specific markers surrounding implanted neuroprostheses.Wnt/β-catenin signaling mediates the seizure-facilitating effect of postischemic reactive astrocytes after pentylenetetrazole-kindling.The ATP-Gated P2X7 Receptor As a Target for the Treatment of Drug-Resistant Epilepsy Mechanisms of Excessive Extracellular Glutamate Accumulation in Temporal Lobe Epilepsy.Ammonia toxicity: from head to toe?Astroglia as a cellular target for neuroprotection and treatment of neuro-psychiatric disorders.Altered gene expression profile in a mouse model of SCN8A encephalopathy.The Role of Astrocytes in Neuroprotection after Brain Stroke: Potential in Cell Therapy.Turning down the volume: Astrocyte volume change in the generation and termination of epileptic seizures.Deletion of mTOR in Reactive Astrocytes Suppresses Chronic Seizures in a Mouse Model of Temporal Lobe Epilepsy.Regulation of astrocyte glutamate transporter-1 (GLT1) and aquaporin-4 (AQP4) expression in a model of epilepsy.Sonic hedgehog signalling mediates astrocyte crosstalk with neurons to confer neuroprotection.Effects of anti-epileptic drugs on spreading depolarization-induced epileptiform activity in mouse hippocampal slices.Cyclooxygenase-1 as a Potential Therapeutic Target for Seizure Suppression: Evidences from Zebrafish Pentylenetetrazole-Seizure Model.Losing Touch With Your Astrocytes Can Cause Epilepsy.Repeated low-dose kainate administration in C57BL/6J mice produces temporal lobe epilepsy pathology but infrequent spontaneous seizures.Pharmacologic inhibition of Hsp90 to prevent GLT-1 degradation as an effective therapy for epilepsy.Astrocytic TLR4 at the crossroads of inflammation and seizure susceptibility Xenotransplantation of pediatric low grade gliomas confirms the enrichment of BRAF V600E mutation and preservation of CDKN2A deletion in a novel orthotopic xenograft mouse model of progressive pleomorphic xanthoastrocytoma.Seizure frequency correlates with loss of dentate gyrus GABAergic neurons in a mouse model of temporal lobe epilepsy.A role for ErbB signaling in the induction of reactive astrogliosis.Neonatal mouse cortical but not isogenic human astrocyte feeder layers enhance the functional maturation of induced pluripotent stem cell-derived neurons in culture.

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Reactive astrogliosis causes the development of spontaneous seizures.

http://www.wikidata.org/entity/Q35117565

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2015 nî lūn-bûn

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2015 թուականի Փետրուարին հրատարակուած գիտական յօդուած

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2015 թվականի փետրվարին հրատարակված գիտական հոդված

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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name

Reactive astrogliosis causes the development of spontaneous seizures.

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Reactive astrogliosis causes the development of spontaneous seizures.

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Reactive astrogliosis causes the development of spontaneous seizures.

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Reactive astrogliosis causes the development of spontaneous seizures.

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Reactive astrogliosis causes the development of spontaneous seizures.

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Reactive astrogliosis causes the development of spontaneous seizures.

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Reactive astrogliosis causes the development of spontaneous seizures.

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Bernd Sutor

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Stefanie Robel

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Susan L Campbell

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Therese Riedemann

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P2860

NMDA receptor activity downregulates KCC2 resulting in depolarizing GABAA receptor-mediated currents

Selective induction of astrocytic gliosis generates deficits in neuronal inhibition

Molecular dissection of reactive astrogliosis and glial scar formation

Blood-brain barrier dysfunction, TGFβ signaling, and astrocyte dysfunction in epilepsy

Modulation of neuronal activity by phosphorylation of the K-Cl cotransporter KCC2

AMPA receptors as a molecular target in epilepsy therapy

GABAergic disinhibition and impaired KCC2 cotransporter activity underlie tumor-associated epilepsy.

Astrocytes: biology and pathology

Differential expression of two glial glutamate transporters in the rat brain: quantitative and immunocytochemical observations

Conditional knock-out of Kir4.1 leads to glial membrane depolarization, inhibition of potassium and glutamate uptake, and enhanced short-term synaptic potentiation

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Niels Christian Danbolt

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Integrin beta 1 (fibronectin receptor beta)

Solute carrier family 1 (glial high affinity glutamate transporter), member 2

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