Wallerian degeneration: an emerging axon death pathway linking injury and disease.
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The PHR proteins: intracellular signaling hubs in neuronal development and axon degenerationTraumatic Axonal Injury: Mechanisms and Translational OpportunitiesSome reminiscences on studies of age-dependent and activity-dependent degeneration of sensory and motor endings in mammalian skeletal muscleAxon injury induced endoplasmic reticulum stress and neurodegenerationTwo for the Price of One: A Neuroprotective Chaperone Kit within NAD Synthase Protein NMNAT2Developmental Function of the PHR Protein RPM-1 Is Required for Learning in Caenorhabditis elegans.A rise in NAD precursor nicotinamide mononucleotide (NMN) after injury promotes axon degenerationCalpain-mediated cleavage of collapsin response mediator protein-2 drives acute axonal degenerationSystems Biology, Neuroimaging, Neuropsychology, Neuroconnectivity and Traumatic Brain Injury.Specific phospholipid scramblases are involved in exposure of phosphatidylserine, an "eat-me" signal for phagocytes, on degenerating axonsRIPK1 mediates axonal degeneration by promoting inflammation and necroptosis in ALS.NMNAT: It's an NAD+ synthase… It's a chaperone… It's a neuroprotector.Preserve and protect: maintaining axons within functional circuits.Mitochondrial alarmins released by degenerating motor axon terminals activate perisynaptic Schwann cellsAxonal transport declines with age in two distinct phases separated by a period of relative stability.Components of myelin damage and repair in the progression of white matter pathology after mild traumatic brain injuryActivity-dependent degeneration of axotomized neuromuscular synapses in Wld S mice.Absence of SARM1 rescues development and survival of NMNAT2-deficient axons.Hope on the (fruit) fly: the Drosophila wing paradigm of axon injury.The method of isolation of the crayfish abdominal stretch receptor maintaining a connection of the sensory neuron to the ventral nerve cord ganglion.Dexamethasone as adjuvant to bupivacaine prolongs the duration of thermal antinociception and prevents bupivacaine-induced rebound hyperalgesia via regional mechanism in a mouse sciatic nerve block model.A microchannel device tailored to laser axotomy and long-term microelectrode array electrophysiology of functional regeneration.Proteomic Profiling in the Brain of CLN1 Disease Model Reveals Affected Functional Modules.SARM1 activation triggers axon degeneration locally via NAD⁺ destructionGlutamate Stimulates Local Protein Synthesis in the Axons of Rat Cortical Neurons by Activating α-Amino-3-hydroxy-5-methyl-4-isoxazolepropionic Acid (AMPA) Receptors and Metabotropic Glutamate Receptors.Longitudinal Changes in White Matter Tract Integrity across the Adult Lifespan and Its Relation to Cortical Thinning.NMNAT2:HSP90 Complex Mediates Proteostasis in Proteinopathies.Generation of a Stable Transgenic Swine Model Expressing a Porcine Histone 2B-eGFP Fusion Protein for Cell Tracking and Chromosome Dynamics Studies.Human Umbilical Tissue-Derived Cells Promote Synapse Formation and Neurite Outgrowth via Thrombospondin Family ProteinsAlternative splicing of Drosophila Nmnat functions as a switch to enhance neuroprotection under stress.Blocking Smad2 signalling with loganin attenuates SW10 cell cycle arrest induced by TNF-α.Sympathetic Reinnervation Is Required for Mammalian Cardiac Regeneration.ROS produced by NOX2 control in vitro development of cerebellar granule neurons development.Rescue of Glaucomatous Neurodegeneration by Differentially Modulating Neuronal Endoplasmic Reticulum Stress Molecules.Antibodies to the RNA-binding protein hnRNP A1 contribute to neurodegeneration in a model of central nervous system autoimmune inflammatory disease.Attenuated traumatic axonal injury and improved functional outcome after traumatic brain injury in mice lacking Sarm1.Delayed glial clearance of degenerating axons in aged Drosophila is due to reduced PI3K/Draper activityNMNAT1 inhibits axon degeneration via blockade of SARM1-mediated NAD+ depletion.Distorted Coarse Axon Targeting and Reduced Dendrite Connectivity Underlie Dysosmia after Olfactory Axon Injury.MAPK signaling promotes axonal degeneration by speeding the turnover of the axonal maintenance factor NMNAT2.
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Wallerian degeneration: an emerging axon death pathway linking injury and disease.
description
article científic
@ca
article scientifique
@fr
articol științific
@ro
articolo scientifico
@it
artigo científico
@gl
artigo científico
@pt
artigo científico
@pt-br
artikel ilmiah
@id
artikull shkencor
@sq
artículo científico
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name
Wallerian degeneration: an emerging axon death pathway linking injury and disease.
@en
type
label
Wallerian degeneration: an emerging axon death pathway linking injury and disease.
@en
prefLabel
Wallerian degeneration: an emerging axon death pathway linking injury and disease.
@en
P2093
P2860
P356
P1476
Wallerian degeneration: an emerging axon death pathway linking injury and disease.
@en
P2093
Jonathan Gilley
Laura Conforti
Michael P Coleman
P2860
P2888
P304
P356
10.1038/NRN3680
P407
P577
2014-06-01T00:00:00Z
P5875
P6179
1038122987