Repression of CDKN2C caused by PML/RARα binding promotes the proliferation and differentiation block in acute promyelocytic leukemia.

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Repression of CDKN2C caused by PML/RARα binding promotes the proliferation and differentiation block in acute promyelocytic leukemia.

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http://www.wikidata.org/entity/Q38728895

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2016 nî lūn-bûn

@nan

2016年の論文

@ja

2016年論文

@yue

2016年論文

@zh-hant

2016年論文

@zh-hk

2016年論文

@zh-mo

2016年論文

@zh-tw

2016年论文

@wuu

2016年论文

@zh

2016年论文

@zh-cn

name

Repression of CDKN2C caused by ...... acute promyelocytic leukemia.

@en

type

Item

label

Repression of CDKN2C caused by ...... acute promyelocytic leukemia.

@en

prefLabel

Repression of CDKN2C caused by ...... acute promyelocytic leukemia.

@en

P1476

Repression of CDKN2C caused by ...... acute promyelocytic leukemia.

@en

P2093

Jingming Li

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Kankan Wang

http://www.w3.org/2001/XMLSchema#string

Wen Jin

http://www.w3.org/2001/XMLSchema#string

Xiaoling Wang

http://www.w3.org/2001/XMLSchema#string

Yizhen Li

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Yun Tan

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P2860

PML/RARalpha targets promoter regions containing PU.1 consensus and RARE half sites in acute promyelocytic leukemia

High throughput digital quantification of mRNA abundance in primary human acute myeloid leukemia samples

Distinct versus redundant properties among members of the INK4 family of cyclin-dependent kinase inhibitors

Cdks, cyclins and CKIs: roles beyond cell cycle regulation

Functional collaboration between different cyclin-dependent kinase inhibitors suppresses tumor growth with distinct tissue specificity.

Control of spermatogenesis in mice by the cyclin D-dependent kinase inhibitors p18(Ink4c) and p19(Ink4d)

Coupled transcriptional and translational control of cyclin-dependent kinase inhibitor p18INK4c expression during myogenesis.

PML-RARalpha/RXR Alters the Epigenetic Landscape in Acute Promyelocytic Leukemia.

The regulation of INK4/ARF in cancer and aging.

Limited overlapping roles of P15(INK4b) and P18(INK4c) cell cycle inhibitors in proliferation and tumorigenesis

P2888

s11684-016-0478-3

P304

420-429

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P31

scholarly article

P356

10.1007/S11684-016-0478-3

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P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P577

2016-11-23T00:00:00Z

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P6179

1032574030

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P698

27888400

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