KMT Set7/9 affects genotoxic stress response via the Mdm2 axis.
about
P53 functional abnormality in mesenchymal stem cells promotes osteosarcoma developmentOne-carbon metabolism and nucleotide biosynthesis as attractive targets for anticancer therapy.Ultraconserved long non-coding RNA uc.63 in breast cancerE3 ubiquitin ligase Pirh2 enhances tumorigenic properties of human non-small cell lung carcinoma cells.Human EHMT2/G9a activates p53 through methylation-independent mechanism.Opposite Effects of SET7/9 on Apoptosis of Human Acute Myeloid Leukemia Cells and Lung Cancer Cells.Lysine methylation of FEN1 by SET7 is essential for its cellular response to replicative stress.BTK blocks the inhibitory effects of MDM2 on p53 activity.SET7/9 inhibits oncogenic activities through regulation of Gli-1 expression in breast cancer.SET7/9 promotes hepatocellular carcinoma progression through regulation of E2F1Isatin-Schiff base-copper (II) complex induces cell death in p53-positive tumors
P2860
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P2860
KMT Set7/9 affects genotoxic stress response via the Mdm2 axis.
description
2015 nî lūn-bûn
@nan
2015年の論文
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2015年論文
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2015年論文
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2015年論文
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2015年論文
@zh-mo
2015年論文
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2015年论文
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2015年论文
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2015年论文
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name
KMT Set7/9 affects genotoxic stress response via the Mdm2 axis.
@en
type
label
KMT Set7/9 affects genotoxic stress response via the Mdm2 axis.
@en
prefLabel
KMT Set7/9 affects genotoxic stress response via the Mdm2 axis.
@en
P2093
P2860
P356
P1433
P1476
KMT Set7/9 affects genotoxic stress response via the Mdm2 axis.
@en
P2093
Alexey V Antonov
Daria Malikova
Gerry Melino
Larissa Lezina
Nikolai A Barlev
Oleg Shuvalov
Olga Fedorova
Vasilisa Aksenova
P2860
P304
25843-25855
P356
10.18632/ONCOTARGET.4584
P407
P577
2015-08-01T00:00:00Z