MLK3 is part of a feedback mechanism that regulates different cellular responses to reactive oxygen species.
about
A systems-biological study on the identification of safe and effective molecular targets for the reduction of ultraviolet B-induced skin pigmentationContext-independent essential regulatory interactions for apoptosis and hypertrophy in the cardiac signaling networkMechanisms of acetaminophen hepatotoxicity and their translation to the human pathophysiology.Differential Epigenetic Effects of Atmospheric Cold Plasma on MCF-7 and MDA-MB-231 Breast Cancer Cells.Model-Driven Understanding of Palmitoylation Dynamics: Regulated Acylation of the Endoplasmic Reticulum Chaperone CalnexinElucidating the Mechanism of Weissella-dependent Lifespan Extension in Caenorhabditis elegansUnravelling druggable signalling networks that control F508del-CFTR proteostasisNOX5-L can stimulate proliferation and apoptosis depending on its levels and cellular context, determining cancer cell susceptibility to cisplatinSynthetic mixed-signal computation in living cellsSystems biological approaches to the cardiac signaling network.Heme oxygenase-1 protects spinal cord neurons from hydrogen peroxide-induced apoptosis via suppression of Cdc42/MLK3/MKK7/JNK3 signaling.A novel interaction perturbation analysis reveals a comprehensive regulatory principle underlying various biochemical oscillators.MLK3 phosphorylation by ERK1/2 is required for oxidative stress-induced invasion of colorectal cancer cells.A novel lnc-PCF promotes the proliferation of TGF-β1-activated epithelial cells by targeting miR-344a-5p to regulate map3k11 in pulmonary fibrosis.Dissecting Cell-Fate Determination Through Integrated Mathematical Modeling of the ERK/MAPK Signaling Pathway.
P2860
Q28262027-A894D26A-D457-4D01-B89A-B2CF1BBD7C30Q33671530-764A3EE8-F1BC-411B-88A9-30A6538212DBQ33848167-19D0DD54-74C0-4D88-A8AB-DFB7CAC4A511Q35652452-5C0FF6EE-E717-4CBB-B011-F6760D45BCE6Q35930512-CDF294B9-D601-4139-9DD8-2A6AF718732AQ36316598-919A66BB-808A-4816-BED4-4D12D1EE38DAQ36568529-1875640B-75DD-4C53-9D43-66C65ECE9406Q36629569-84E6E0EE-1C6E-4A4A-9606-268A5944C0A3Q36975786-DCB35298-F34B-4CC8-AF1F-019CBB99345EQ38537087-5ECA77F0-441A-4E2C-B65E-AD514BE75FB7Q40467191-52EC0999-F293-42AE-AF57-ECF36AACB46AQ42374465-3C1FA49E-097C-4C5E-A5B1-7AE797FCA647Q46272315-DFCD38AD-7D16-4360-87AA-8328BA10DAD9Q47149080-B88B7A83-9EE2-4FCF-8A5B-C9885087B019Q50538898-822B4020-072B-4BBF-A08D-014B8D1AE744
P2860
MLK3 is part of a feedback mechanism that regulates different cellular responses to reactive oxygen species.
description
2014 nî lūn-bûn
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2014年の論文
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2014年学术文章
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name
MLK3 is part of a feedback mec ...... es to reactive oxygen species.
@en
type
label
MLK3 is part of a feedback mec ...... es to reactive oxygen species.
@en
prefLabel
MLK3 is part of a feedback mec ...... es to reactive oxygen species.
@en
P2093
P2860
P1433
P1476
MLK3 is part of a feedback mec ...... es to reactive oxygen species.
@en
P2093
Chae Young Hwang
Ho-Sung Lee
Ki-Sun Kwon
Sung-Young Shin
P2860
P356
10.1126/SCISIGNAL.2005260
P577
2014-06-03T00:00:00Z