Cross-talk between RORγt+ innate lymphoid cells and intestinal macrophages induces mucosal IL-22 production in Crohn's disease.
about
Interleukin-22: immunobiology and pathology.A breakthrough in probiotics: Clostridium butyricum regulates gut homeostasis and anti-inflammatory response in inflammatory bowel disease.Immunopathogenesis of IBD: current state of the art.Intestinal macrophages arising from CCR2(+) monocytes control pathogen infection by activating innate lymphoid cells.The unusual suspects--innate lymphoid cells as novel therapeutic targets in IBD.Enrichment of IL-17A+ IFN-γ+ and IL-22+ IFN-γ+ T cell subsets is associated with reduction of NKp44+ ILC3s in the terminal ileum of Crohn's disease patients.Biological and pathological activities of interleukin-22.Lessons Learned From Trials Targeting Cytokine Pathways in Patients With Inflammatory Bowel Diseases.Indole compounds may be promising medicines for ulcerative colitis.Recent advances in gut immunology.The orphan nuclear receptor RORα and group 3 innate lymphoid cells drive fibrosis in a mouse model of Crohn's disease.Aryl hydrocarbon receptor signaling involves in the human intestinal ILC3/ILC1 conversion in the inflamed terminal ileum of Crohn's disease patients.IL-22 Increases Permeability of Intestinal Epithelial Tight Junctions by Enhancing Claudin-2 Expression.Urinary interleukin 22 binding protein as a marker of lupus nephritis in Egyptian children with juvenile systemic lupus erythematosus.Barrier-tissue macrophages: functional adaptation to environmental challenges.Regulation of Innate Lymphoid Cells by Aryl Hydrocarbon Receptor.Immunological pathogenesis of inflammatory bowel disease.The Innate Immune Response in Myocardial Infarction, Repair, and Regeneration.Functional macrophages and gastrointestinal disorders.A Noninvasive Method to Assess Mucosal Healing in Patients* With Crohn’s Disease.IL22 Inhibits Epithelial Stem Cell Expansion in an Ileal Organoid ModelAnti-IL-22 Antibody Attenuates Acute Graft-versus-Host Disease via Increasing Foxp3 T Cell through Modulation of CD11b Cell Function
P2860
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P2860
Cross-talk between RORγt+ innate lymphoid cells and intestinal macrophages induces mucosal IL-22 production in Crohn's disease.
description
2014 nî lūn-bûn
@nan
2014年の論文
@ja
2014年学术文章
@wuu
2014年学术文章
@zh-cn
2014年学术文章
@zh-hans
2014年学术文章
@zh-my
2014年学术文章
@zh-sg
2014年學術文章
@yue
2014年學術文章
@zh
2014年學術文章
@zh-hant
name
Cross-talk between RORγt+ inna ...... production in Crohn's disease.
@en
Cross-talk between RORγt+ inna ...... production in Crohn's disease.
@nl
type
label
Cross-talk between RORγt+ inna ...... production in Crohn's disease.
@en
Cross-talk between RORγt+ inna ...... production in Crohn's disease.
@nl
prefLabel
Cross-talk between RORγt+ inna ...... production in Crohn's disease.
@en
Cross-talk between RORγt+ inna ...... production in Crohn's disease.
@nl
P2093
P2860
P50
P1476
Cross-talk between RORγt+ inna ...... production in Crohn's disease.
@en
P2093
Akira Sugita
Atsushi Hayashi
Katsuyoshi Matsuoka
Kazutaka Koganei
Mami Matano
Masakazu Takazoe
Rikisaburo Sahara
Shinta Mizuno
Tadakazu Hisamatsu
Tango Handa
P2860
P304
P356
10.1097/MIB.0000000000000105
P577
2014-08-01T00:00:00Z