about
Control of pathogens and pathobionts by the gut microbiotaA novel apoptosis-inducing monoclonal antibody (anti-LHK) against a cell surface antigen on colon cancer cells.Imbalance in intestinal microflora constitution could be involved in the pathogenesis of inflammatory bowel disease.Tryptophan catabolism restricts IFN-γ-expressing neutrophils and Clostridium difficile immunopathologyRegulated virulence controls the ability of a pathogen to compete with the gut microbiota.Nonpathogenic Escherichia coli strain Nissle1917 prevents murine acute and chronic colitis.Interleukin-22 regulates the complement system to promote resistance against pathobionts after pathogen-induced intestinal damage.Macrophages and dendritic cells emerge in the liver during intestinal inflammation and predispose the liver to inflammation.TACI deficiency enhances antibody avidity and clearance of an intestinal pathogenDistinct Commensals Induce Interleukin-1β via NLRP3 Inflammasome in Inflammatory Monocytes to Promote Intestinal Inflammation in Response to InjuryBoth exogenous commensal and endogenous self antigens stimulate T cell proliferation under lymphopenic conditionsMicrobiota-induced IL-1β, but not IL-6, is critical for the development of steady-state TH17 cells in the intestineRegulation of virulence: the rise and fall of gastrointestinal pathogens.Intestinal macrophages arising from CCR2(+) monocytes control pathogen infection by activating innate lymphoid cells.Increased Expression of DUOX2 Is an Epithelial Response to Mucosal Dysbiosis Required for Immune Homeostasis in Mouse IntestineRole of the gut microbiota in the development and function of lymphoid cells.Th17 Cell Induction by Adhesion of Microbes to Intestinal Epithelial Cells.Unique CD14 intestinal macrophages contribute to the pathogenesis of Crohn disease via IL-23/IFN-gamma axis.TGR5 signalling inhibits the production of pro-inflammatory cytokines by in vitro differentiated inflammatory and intestinal macrophages in Crohn's diseaseNod2-mediated recognition of the microbiota is critical for mucosal adjuvant activity of cholera toxinDiet-dependent, microbiota-independent regulation of IL-10-producing lamina propria macrophages in the small intestine.Functional Characterization of Inflammatory Bowel Disease-Associated Gut Dysbiosis in Gnotobiotic MiceHomeostatic (IL-7) and effector (IL-17) cytokines as distinct but complementary target for an optimal therapeutic strategy in inflammatory bowel disease.Regulation of the immune system by the resident intestinal bacteria.Cross-talk between RORγt+ innate lymphoid cells and intestinal macrophages induces mucosal IL-22 production in Crohn's disease.Mesenchymal Cell-Specific MyD88 Signaling Promotes Systemic Dissemination of Salmonella Typhimurium via Inflammatory Monocytes.Humoral Immunity in the Gut Selectively Targets Phenotypically Virulent Attaching-and-Effacing Bacteria for Intraluminal Elimination.Protective role of commensals against Clostridium difficile infection via an IL-1β-mediated positive-feedback loopMultiple effects of dendritic cell depletion on murine norovirus infection.The tuning of the gut nervous system by commensal microbiota.Tetomilast suppressed production of proinflammatory cytokines from human monocytes and ameliorated chronic colitis in IL-10-deficient mice.IL23 differentially regulates the Th1/Th17 balance in ulcerative colitis and Crohn's disease.Activated hepatic stellate cells mediate the differentiation of macrophages.A single strain of Clostridium butyricum induces intestinal IL-10-producing macrophages to suppress acute experimental colitis in mice.Human CD14+ macrophages in intestinal lamina propria exhibit potent antigen-presenting ability.Retinoic acid contributes to the induction of IL-12-hypoproducing dendritic cells.Dietary histidine ameliorates murine colitis by inhibition of proinflammatory cytokine production from macrophages.Establishment of novel prediction system of intestinal absorption in humans using human intestinal tissues.TL1A produced by lamina propria macrophages induces Th1 and Th17 immune responses in cooperation with IL-23 in patients with Crohn's disease.Imbalance of NKp44(+)NKp46(-) and NKp44(-)NKp46(+) natural killer cells in the intestinal mucosa of patients with Crohn's disease.
P50
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P50
description
hulumtues
@sq
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Nobuhiko Kamada
@ast
Nobuhiko Kamada
@en
Nobuhiko Kamada
@es
Nobuhiko Kamada
@nl
Nobuhiko Kamada
@sl
type
label
Nobuhiko Kamada
@ast
Nobuhiko Kamada
@en
Nobuhiko Kamada
@es
Nobuhiko Kamada
@nl
Nobuhiko Kamada
@sl
prefLabel
Nobuhiko Kamada
@ast
Nobuhiko Kamada
@en
Nobuhiko Kamada
@es
Nobuhiko Kamada
@nl
Nobuhiko Kamada
@sl
P106
P1153
21134750600
P21
P31
P496
0000-0002-1980-4178
P569
2000-01-01T00:00:00Z