Krüppel-like factor 5 promotes breast cell proliferation partially through upregulating the transcription of fibroblast growth factor binding protein 1.
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Mammalian Krüppel-like factors in health and diseasesEvasion of anti-growth signaling: A key step in tumorigenesis and potential target for treatment and prophylaxis by natural compounds.FBW7-mediated ubiquitination and degradation of KLF5The double life of KLF5: Opposing roles in regulation of gene-expression, cellular function, and transformationFBXW7 mutations typically found in human cancers are distinct from null alleles and disrupt lung developmentMetformin suppresses triple-negative breast cancer stem cells by targeting KLF5 for degradation.miR-217 inhibits triple-negative breast cancer cell growth, migration, and invasion through targeting KLF5Klf5 Mediates Odontoblastic Differentiation through Regulating Dentin-Specific Extracellular Matrix Gene Expression during Mouse Tooth DevelopmentThe induction of KLF5 transcription factor by progesterone contributes to progesterone-induced breast cancer cell proliferation and dedifferentiation.The Fbw7/human CDC4 tumor suppressor targets proproliferative factor KLF5 for ubiquitination and degradation through multiple phosphodegron motifs.WWOX suppresses KLF5 expression and breast cancer cell growthInterruption of KLF5 acetylation converts its function from tumor suppressor to tumor promoter in prostate cancer cells.Klf5 deletion promotes Pten deletion-initiated luminal-type mouse prostate tumors through multiple oncogenic signaling pathwaysHelicobacter pylori promotes the expression of Krüppel-like factor 5, a mediator of carcinogenesis, in vitro and in vivo.Regulation of anti-apoptotic signaling by Kruppel-like factors 4 and 5 mediates lapatinib resistance in breast cancer.Ataxin-3 like (ATXN3L), a member of the Josephin family of deubiquitinating enzymes, promotes breast cancer proliferation by deubiquitinating Krüppel-like factor 5 (KLF5).Krüppel-like factors 4 and 5 expression and their involvement in differentiation of oral carcinomas.Anti-tumor effects of fibroblast growth factor-binding protein (FGF-BP) knockdown in colon carcinoma.BAP1 promotes breast cancer cell proliferation and metastasis by deubiquitinating KLF5.Tumor suppressor functions of FBW7 in cancer development and progression.The ubiquitin E3 ligase WWP1 decreases CXCL12-mediated MDA231 breast cancer cell migration and bone metastasis.The interplay between TEAD4 and KLF5 promotes breast cancer partially through inhibiting the transcription of p27Kip1.Mifepristone Suppresses Basal Triple-Negative Breast Cancer Stem Cells by Down-regulating KLF5 Expression.Beyond proliferation: KLF5 promotes angiogenesis of bladder cancer through directly regulating VEGFA transcription.Oestrogen causes degradation of KLF5 by inducing the E3 ubiquitin ligase EFP in ER-positive breast cancer cells.Kruppel-like factor 5 transcription factor promotes microsomal prostaglandin E2 synthase 1 gene transcription in breast cancerEvidence for independent evolution of functional progesterone withdrawal in primates and guinea pigs.KLF5 activates microRNA 200 transcription to maintain epithelial characteristics and prevent induced epithelial-mesenchymal transition in epithelial cells.Muscle Fibers Secrete FGFBP1 to Slow Degeneration of Neuromuscular Synapses during Aging and Progression of ALS.Dexamethasone induces docetaxel and cisplatin resistance partially through up-regulating Krüppel-like factor 5 in triple-negative breast cancer.KLF5 promotes breast cancer proliferation, migration and invasion in part by upregulating the transcription of TNFAIP2.Functional and clinical evidence that TAZ is a candidate oncogene in hepatocellular carcinoma.Krüppel-like factor 5: a novel biomarker for lymph node metastasis and recurrence in supraglottic squamous cell laryngeal carcinoma.TAZ antagonizes the WWP1-mediated KLF5 degradation and promotes breast cell proliferation and tumorigenesis.YD277 Suppresses Triple-Negative Breast Cancer Partially Through Activating the Endoplasmic Reticulum Stress Pathway.KLF5 promotes cervical cancer proliferation, migration and invasion in a manner partly dependent on TNFRSF11a expression.Krüppel-like factor 5 as potential molecular marker in cervical cancer and the KLF family profile expression.Mithramycin A suppresses basal triple-negative breast cancer cell survival partially via down-regulating Krüppel-like factor 5 transcription by Sp1.
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P2860
Krüppel-like factor 5 promotes breast cell proliferation partially through upregulating the transcription of fibroblast growth factor binding protein 1.
description
2009 nî lūn-bûn
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2009年の論文
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2009年学术文章
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2009年学术文章
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2009年学术文章
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2009年学术文章
@zh-my
2009年学术文章
@zh-sg
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name
Krüppel-like factor 5 promotes ...... owth factor binding protein 1.
@en
Krüppel-like factor 5 promotes ...... owth factor binding protein 1.
@nl
type
label
Krüppel-like factor 5 promotes ...... owth factor binding protein 1.
@en
Krüppel-like factor 5 promotes ...... owth factor binding protein 1.
@nl
prefLabel
Krüppel-like factor 5 promotes ...... owth factor binding protein 1.
@en
Krüppel-like factor 5 promotes ...... owth factor binding protein 1.
@nl
P2093
P2860
P356
P1433
P1476
Krüppel-like factor 5 promotes ...... rowth factor binding protein 1
@en
P2093
P2860
P2888
P304
P356
10.1038/ONC.2009.235
P407
P577
2009-08-10T00:00:00Z