Return to quiescence of mouse neural stem cells by degradation of a proactivation protein.
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Rapid trait evolution drives increased speed and variance in experimental range expansionsThe HECT domain ubiquitin ligase HUWE1 targets unassembled soluble proteins for degradation.REST regulation of gene networks in adult neural stem cells.E3 ubiquitin ligase Mule targets β-catenin under conditions of hyperactive Wnt signaling.The HECT Family Ubiquitin Ligase EEL-1 Regulates Neuronal Function and Development.Programming and reprogramming the brain: a meeting of minds in neural fate.RB: An essential player in adult neurogenesis.Forebrain neurogenesis: From embryo to adult.Cycling through developmental decisions: how cell cycle dynamics control pluripotency, differentiation and reprogramming.Ubiquitin-Dependent Regulation of Stem Cell Biology.The Potential of Targeting Brain Pathology with Ascl1/Mash1.Ubiquitylation at the crossroads of development and disease.Impaired oxidative stress response characterizes HUWE1-promoted X-linked intellectual disability.Keeping Neurons Young and Foxy: FoxOs Promote Neuronal Plasticity.Troy+ brain stem cells cycle through quiescence and regulate their number by sensing niche occupancy.A morphology independent approach for identifying dividing adult neural stem cells in the mouse hippocampus.Quiescent Oct4+ Neural Stem Cells (NSCs) Repopulate Ablated Glial Fibrillary Acidic Protein+ NSCs in the Adult Mouse Brain.Specific Phospholipids Regulate the Acquisition of Neuronal and Astroglial Identities in Post-Mitotic Cells.Review: adult neurogenesis contributes to hippocampal plasticity.Climate Degradation and Extreme Icing Events Constrain Life in Cold-Adapted Mammals.Spatial geometry of stem cell proliferation in the adult hippocampus.Subcellular localisation modulates ubiquitylation and degradation of Ascl1.Neural stem cell quiescence and stemness are molecularly distinct outputs of the Notch3 signaling cascade in the vertebrate adult brain.Radial glial cells in the adult dentate gyrus: what are they and where do they come from?A transcription factor collective defines the HSN serotonergic neuron regulatory landscape.The developmental origin of brain tumours: a cellular and molecular framework.Defective germline reprogramming rewires the spermatogonial transcriptomeE proteins sharpen neurogenesis by modulating proneural bHLH transcription factors' activity in an E-box-dependent manner
P2860
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P2860
Return to quiescence of mouse neural stem cells by degradation of a proactivation protein.
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年学术文章
@wuu
2016年学术文章
@zh-cn
2016年学术文章
@zh-hans
2016年学术文章
@zh-my
2016年学术文章
@zh-sg
2016年學術文章
@yue
2016年學術文章
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2016年學術文章
@zh-hant
name
Return to quiescence of mouse ...... on of a proactivation protein.
@en
type
label
Return to quiescence of mouse ...... on of a proactivation protein.
@en
prefLabel
Return to quiescence of mouse ...... on of a proactivation protein.
@en
P2093
P2860
P356
P1433
P1476
Return to quiescence of mouse neural stem cells by degradation of a proactivation protein
@en
P2093
Antoine Forget
Charles Hunt
François Guillemot
Jimena Andersen
Noelia Urbán
Olivier Ayrault
P2860
P304
P356
10.1126/SCIENCE.AAF4802
P407
P577
2016-07-01T00:00:00Z