Transgenic mice recapitulate the phenotypic heterogeneity of genetic prion diseases without developing prion infectivity: Role of intracellular PrP retention in neurotoxicity
about
Misfolding leads the way to unraveling signaling pathways in the pathophysiology of prion diseases.Prion protein inhibits fast axonal transport through a mechanism involving casein kinase 2.A Chinese patient of P102L Gerstmann-Sträussler-Scheinker disease contains three other disease-associated mutations in SYNE1.
P2860
Transgenic mice recapitulate the phenotypic heterogeneity of genetic prion diseases without developing prion infectivity: Role of intracellular PrP retention in neurotoxicity
description
2016 nî lūn-bûn
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2016年の論文
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2016年学术文章
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2016年学术文章
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2016年学术文章
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2016年学术文章
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2016年学术文章
@zh-sg
2016年學術文章
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2016年學術文章
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2016年學術文章
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name
Transgenic mice recapitulate t ...... PrP retention in neurotoxicity
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type
label
Transgenic mice recapitulate t ...... PrP retention in neurotoxicity
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prefLabel
Transgenic mice recapitulate t ...... PrP retention in neurotoxicity
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P2860
P50
P1433
P1476
Transgenic mice recapitulate t ...... PrP retention in neurotoxicity
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P2093
Liliana Comerio
P2860
P304
P356
10.1080/19336896.2016.1139276
P577
2016-03-01T00:00:00Z