Targeting trisomic treatments: optimizing Dyrk1a inhibition to improve Down syndrome deficits.

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Targeting trisomic treatments: optimizing Dyrk1a inhibition to improve Down syndrome deficits.

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2017 nî lūn-bûn

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2017年の論文

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2017年論文

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2017年論文

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2017年論文

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2017年論文

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2017年論文

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2017年论文

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Targeting trisomic treatments: ...... mprove Down syndrome deficits.

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Targeting trisomic treatments: ...... mprove Down syndrome deficits.

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Targeting trisomic treatments: ...... mprove Down syndrome deficits.

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Targeting trisomic treatments: ...... improve Down syndrome deficits

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P2093

Charles R Goodlett

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Megan Stringer

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P2860

DYRK1A, a novel determinant of the methionine-homocysteine cycle in different mouse models overexpressing this Down-syndrome-associated kinase

Updated national birth prevalence estimates for selected birth defects in the United States, 2004-2006

DYRK1A, a Dosage-Sensitive Gene Involved in Neurodevelopmental Disorders, Is a Target for Drug Development in Down Syndrome

Timing of therapies for Down syndrome: the sooner, the better

Mouse models of Down syndrome as a tool to unravel the causes of mental disabilities

Principal Component Analysis of the Effects of Environmental Enrichment and (-)-epigallocatechin-3-gallate on Age-Associated Learning Deficits in a Mouse Model of Down Syndrome

Development of a novel selective inhibitor of the Down syndrome-related kinase Dyrk1A

Global up-regulation of chromosome 21 gene expression in the developing Down syndrome brain

Dyrk1A expression pattern supports specific roles of this kinase in the adult central nervous system

Olig1 and Olig2 triplication causes developmental brain defects in Down syndrome

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451-465

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scholarly article

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10.1002/MGG3.334

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English

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Randall J. Roper

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2017-09-20T00:00:00Z

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28944229

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P921

Down syndrome

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5606891

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