DNA Double-Strand Break Resection Occurs during Non-homologous End Joining in G1 but Is Distinct from Resection during Homologous Recombination. - Test2 - elhamkhani - TriplyDB

DNA Double-Strand Break Resection Occurs during Non-homologous End Joining in G1 but Is Distinct from Resection during Homologous Recombination.

DNA Double-Strand Break Resection Occurs during Non-homologous End Joining in G1 but Is Distinct from Resection during Homologous Recombination.

http://www.wikidata.org/entity/Q42320742

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A Process of Resection-Dependent Nonhomologous End Joining Involving the Goddess Artemis.DNA end resection requires constitutive sumoylation of CtIP by CBX4.Chromatin modifiers and remodellers in DNA repair and signalling.BRCA2 antagonizes classical and alternative nonhomologous end-joining to prevent gross genomic instability.PARP2 controls double-strand break repair pathway choice by limiting 53BP1 accumulation at DNA damage sites and promoting end-resection.H2AX facilitates classical non-homologous end joining at the expense of limited nucleotide loss at repair junctions.DNA Damage as a Driver for Growth Delay: Chromosome Instability Syndromes with Intrauterine Growth Retardation.DNA non-homologous end-joining enters the resection arena.EXD2 governs germ stem cell homeostasis and lifespan by promoting mitoribosome integrity and translation.Main steps in DNA double-strand break repair: an introduction to homologous recombination and related processes.Drosha drives the formation of DNA:RNA hybrids around DNA break sites to facilitate DNA repair.How cells ensure correct repair of DNA double-strand breaks.Reading chromatin signatures after DNA double-strand breaks.Repair of DNA double-strand breaks by mammalian alternative end-joining pathways.Deoxyribonucleic Acid Damage and Repair: Capitalizing on Our Understanding of the Mechanisms of Maintaining Genomic Integrity for Therapeutic Purposes.The MRE11-RAD50-NBS1 Complex Conducts the Orchestration of Damage Signaling and Outcomes to Stress in DNA Replication and Repair.The mitochondrial outer-membrane location of the EXD2 exonuclease contradicts its direct role in nuclear DNA repair.Comprehensive Mapping of Histone Modifications at DNA Double-Strand Breaks Deciphers Repair Pathway Chromatin Signatures An OB-fold complex controls the repair pathways for DNA double-strand breaks Robust activation of microhomology-mediated end joining for precision gene editing applications The road less traveled: strategies to enhance the frequency of homology-directed repair (HDR) for increased efficiency of CRISPR/Cas-mediated transgenesis TDP1 suppresses mis-joining of radiomimetic DNA double-strand breaks and cooperates with Artemis to promote optimal nonhomologous end joining Using Persistent Homology as a New Approach for Super-Resolution Localization Microscopy Data Analysis and Classification of γH2AX Foci/Clusters

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DNA Double-Strand Break Resection Occurs during Non-homologous End Joining in G1 but Is Distinct from Resection during Homologous Recombination.

http://www.wikidata.org/entity/Q42320742

description

2017 nî lūn-bûn

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2017年の論文

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2017年論文

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2017年論文

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2017年論文

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2017年論文

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2017年論文

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2017年论文

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2017年论文

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2017年论文

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name

DNA Double-Strand Break Resect ...... ring Homologous Recombination.

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DNA Double-Strand Break Resect ...... ring Homologous Recombination.

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type

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DNA Double-Strand Break Resect ...... ring Homologous Recombination.

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DNA Double-Strand Break Resect ...... ring Homologous Recombination.

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prefLabel

DNA Double-Strand Break Resect ...... ring Homologous Recombination.

@en

DNA Double-Strand Break Resect ...... ring Homologous Recombination.

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J.MOLCEL.2016.12.016

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DNA Double-Strand Break Resect ...... ring Homologous Recombination.

@en

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Julia Künzel

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Markus Löbrich

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Monika Steinlage

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Olivia Barton

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Penny A Jeggo

http://www.w3.org/2001/XMLSchema#string

Ronja Biehs

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Szilvia Juhász

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P2860

The RAD2 domain of human exonuclease 1 exhibits 5' to 3' exonuclease and flap structure-specific endonuclease activities

Hairpin opening and overhang processing by an Artemis/DNA-dependent protein kinase complex in nonhomologous end joining and V(D)J recombination

DNA damage-induced cell cycle checkpoint control requires CtIP, a phosphorylation-dependent binding partner of BRCA1 C-terminal domains

The 3' to 5' exonuclease activity of Mre 11 facilitates repair of DNA double-strand breaks

DNA repair. PAXX, a paralog of XRCC4 and XLF, interacts with Ku to promote DNA double-strand break repair

The DNA-damage response in human biology and disease

Human CtIP promotes DNA end resection

ATM and Artemis promote homologous recombination of radiation-induced DNA double-strand breaks in G2

DNA Double-Strand Break Repair Pathway Choice Is Directed by Distinct MRE11 Nuclease Activities

A pathway of double-strand break rejoining dependent upon ATM, Artemis, and proteins locating to gamma-H2AX foci.

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671-684.e5

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scholarly article

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10.1016/J.MOLCEL.2016.12.016

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4

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65

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Atsushi Shibata

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2017-01-25T00:00:00Z

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28132842

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5316416

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